cathepsin-g and Dermatitis--Contact

cathepsin-g has been researched along with Dermatitis--Contact* in 1 studies

Other Studies

1 other study(ies) available for cathepsin-g and Dermatitis--Contact

Article	Year
Neutrophil Cathepsin G Regulates Dendritic Cell Production of IL-12 during Development of CD4 T Cell Responses to Antigens in the Skin. Journal of immunology (Baltimore, Md. : 1950), 2019, 02-15, Volume: 202, Issue:4 Contact hypersensitivity (CHS) is a CD8 T cell-mediated response to hapten skin sensitization and challenge. Sensitization of wild-type (WT) mice induces hapten-reactive effector CD8 T cells producing IFN-γ and IL-17- and IL-4-producing CD4 T cells that cannot mediate CHS. Although CXCR2-dependent Ly6G Topics: Animals; Cathepsin G; CD4-Positive T-Lymphocytes; Dendritic Cells; Dermatitis, Contact; Female; Haptens; Interleukin-12; Mice; Mice, Inbred BALB C; Mice, Inbred C57BL; Mice, Knockout; Neutrophils; Receptors, Interleukin-8B; Skin	2019

Article

Year

Neutrophil Cathepsin G Regulates Dendritic Cell Production of IL-12 during Development of CD4 T Cell Responses to Antigens in the Skin.

Journal of immunology (Baltimore, Md. : 1950), 2019, 02-15, Volume: 202, Issue:4

Contact hypersensitivity (CHS) is a CD8 T cell-mediated response to hapten skin sensitization and challenge. Sensitization of wild-type (WT) mice induces hapten-reactive effector CD8 T cells producing IFN-γ and IL-17- and IL-4-producing CD4 T cells that cannot mediate CHS. Although CXCR2-dependent Ly6G

Topics: Animals; Cathepsin G; CD4-Positive T-Lymphocytes; Dendritic Cells; Dermatitis, Contact; Female; Haptens; Interleukin-12; Mice; Mice, Inbred BALB C; Mice, Inbred C57BL; Mice, Knockout; Neutrophils; Receptors, Interleukin-8B; Skin

2019