Page last updated: 2024-08-26

catalpol and Cardiovascular Stroke

catalpol has been researched along with Cardiovascular Stroke in 3 studies

Research

Studies (3)

TimeframeStudies, this research(%)All Research%
pre-19900 (0.00)18.7374
1990's0 (0.00)18.2507
2000's0 (0.00)29.6817
2010's3 (100.00)24.3611
2020's0 (0.00)2.80

Authors

AuthorsStudies
Bi, F; Sun, Q; Xu, Y1
Ge, B; Ju, X; Li, Z; Wang, T; Xue, D; Zhang, Y1
Chen, B; Cui, Y; Huang, C; Ji, L; Li, R; Ma, L; Xing, W; Yu, J; Zhang, H; Zhang, W; Zhou, H1

Other Studies

3 other study(ies) available for catalpol and Cardiovascular Stroke

ArticleYear
Catalpol pretreatment attenuates cardiac dysfunction following myocardial infarction in rats.
    Anatolian journal of cardiology, 2018, Volume: 19, Issue:5

    Topics: Animals; Cardiotonic Agents; Disease Models, Animal; Drugs, Chinese Herbal; Iridoid Glucosides; Isoproterenol; Male; Myocardial Infarction; Rats; Rats, Wistar; Ventricular Function, Left

2018
Catalpol Promotes the Survival and VEGF Secretion of Bone Marrow-Derived Stem Cells and Their Role in Myocardial Repair After Myocardial Infarction in Rats.
    Cardiovascular toxicology, 2018, Volume: 18, Issue:5

    Topics: Animals; Apoptosis; Apoptosis Regulatory Proteins; Bone Marrow Cells; Cell Hypoxia; Cells, Cultured; Disease Models, Animal; Glucose; Iridoid Glucosides; Male; Mesenchymal Stem Cell Transplantation; Mesenchymal Stem Cells; Myocardial Infarction; Myocardium; Rats, Wistar; Recovery of Function; Regeneration; Secretory Pathway; Signal Transduction; Stroke Volume; Vascular Endothelial Growth Factor A; Ventricular Function, Left; Ventricular Pressure

2018
Catalpol decreases peroxynitrite formation and consequently exerts cardioprotective effects against ischemia/reperfusion insult.
    Pharmaceutical biology, 2013, Volume: 51, Issue:4

    Topics: Animals; Antioxidants; Apoptosis; Cardiotonic Agents; Disease Models, Animal; Down-Regulation; Enzyme Activation; Free Radical Scavengers; Injections, Intraperitoneal; Iridoid Glucosides; Male; Myocardial Infarction; Myocardial Reperfusion Injury; Myocardium; Necrosis; Nitric Oxide; Nitric Oxide Synthase Type II; Nitric Oxide Synthase Type III; Oxidative Stress; Peroxynitrous Acid; Phosphatidylinositol 3-Kinase; Phosphoinositide-3 Kinase Inhibitors; Phosphorylation; Protein Kinase Inhibitors; Proto-Oncogene Proteins c-akt; Rats; Rats, Sprague-Dawley; Time Factors; Tyrosine

2013