Page last updated: 2024-08-21

carvacrol and Brain Neoplasms

carvacrol has been researched along with Brain Neoplasms in 3 studies

Research

Studies (3)

TimeframeStudies, this research(%)All Research%
pre-19900 (0.00)18.7374
1990's0 (0.00)18.2507
2000's0 (0.00)29.6817
2010's2 (66.67)24.3611
2020's1 (33.33)2.80

Authors

AuthorsStudies
Alanazi, R; Feng, ZP; Golbourn, B; Ji, D; Luo, Z; Nakatogawa, H; Rutka, JT; Sun, HS; Wang, H1
Baron, AK; Georgieff, M; Moepps, B; Mostafa, H; Pala, A; Sander, P; Schneider, JM; Schneider, M; Soboh, A; Wirtz, CR1
Barszczyk, A; Chen, WL; Deurloo, M; Feng, ZP; Liu, B; Rutka, JT; Sun, HS; Turlova, E; Yang, BB1

Other Studies

3 other study(ies) available for carvacrol and Brain Neoplasms

ArticleYear
Inhibition of TRPM7 with carvacrol suppresses glioblastoma functions in vivo.
    The European journal of neuroscience, 2022, Volume: 55, Issue:6

    Topics: Animals; Brain Neoplasms; Cell Line, Tumor; Cell Proliferation; Cymenes; Glioblastoma; Humans; Mice; Neoplasm Recurrence, Local; Protein Serine-Threonine Kinases; Temozolomide; TRPM Cation Channels

2022
Vacquinol-1 inducible cell death in glioblastoma multiforme is counter regulated by TRPM7 activity induced by exogenous ATP.
    Oncotarget, 2017, May-23, Volume: 8, Issue:21

    Topics: Adenosine Triphosphate; Brain Neoplasms; Caspase 3; Caspase 7; Cell Death; Cell Line, Tumor; Cymenes; Gene Expression Regulation, Neoplastic; Glioblastoma; Humans; Monoterpenes; Piperidines; Protein Serine-Threonine Kinases; Quinolines; TRPM Cation Channels

2017
Inhibition of TRPM7 by carvacrol suppresses glioblastoma cell proliferation, migration and invasion.
    Oncotarget, 2015, Jun-30, Volume: 6, Issue:18

    Topics: Actin Depolymerizing Factors; Antineoplastic Agents; Apoptosis; Brain Neoplasms; Caspase 3; Cell Line, Tumor; Cell Movement; Cell Proliferation; Cell Survival; Cymenes; Extracellular Signal-Regulated MAP Kinases; Glioblastoma; HEK293 Cells; Humans; Matrix Metalloproteinase 2; Monoterpenes; Neoplasm Invasiveness; Patch-Clamp Techniques; Phosphorylation; Protein Serine-Threonine Kinases; Proto-Oncogene Proteins c-akt; RNA Interference; RNA, Messenger; RNA, Small Interfering; Signal Transduction; TRPM Cation Channels

2015