cariporide has been researched along with Alloxan Diabetes in 5 studies
Timeframe | Studies, this research(%) | All Research% |
---|---|---|
pre-1990 | 0 (0.00) | 18.7374 |
1990's | 0 (0.00) | 18.2507 |
2000's | 4 (80.00) | 29.6817 |
2010's | 1 (20.00) | 24.3611 |
2020's | 0 (0.00) | 2.80 |
Authors | Studies |
---|---|
Lupachyk, S; Obrosov, A; Obrosova, IG; Shevalye, H; Vareniuk, I; Watcho, P; Yorek, MA | 1 |
Liu, L; Peng, Q; Wang, S; Zhang, J | 1 |
Couturier, K; Demaison, L; Dubouchaud, H; Lanson, M; Leverve, X; Vial, G | 1 |
Chattou, S; Coulombe, A; Diacono, J; Feuvray, D; John, G; Le Grand, B | 1 |
Allen, TJ; Cooper, ME; Farrelly, CA; Gilbert, RE; Hannan, KM; Jandeleit-Dahm, K; Little, PJ; Rumble, JR | 1 |
5 other study(ies) available for cariporide and Alloxan Diabetes
Article | Year |
---|---|
Na+/H+ exchanger 1 inhibition reverses manifestation of peripheral diabetic neuropathy in type 1 diabetic rats.
Topics: Aldehydes; Animals; Arterioles; Behavior, Animal; Blood Glucose; Blotting, Western; Body Weight; Diabetes Mellitus, Experimental; Diabetes Mellitus, Type 1; Diabetic Neuropathies; Glycation End Products, Advanced; Guanidines; Male; Nerve Fibers; Nitrates; Pain Measurement; Pyruvaldehyde; Rats; Rats, Wistar; Reduced Folate Carrier Protein; Sciatic Nerve; Skin; Sulfones; Superoxides; Tyrosine | 2013 |
Na+/H+ exchanger is required for hyperglycaemia-induced endothelial dysfunction via calcium-dependent calpain.
Topics: Animals; Calcium; Calpain; Cells, Cultured; Chelating Agents; Cysteine Proteinase Inhibitors; Diabetes Mellitus, Experimental; Dose-Response Relationship, Drug; Endothelium, Vascular; Glucose; Guanidines; HSP90 Heat-Shock Proteins; Humans; Hyperglycemia; Male; Nitric Oxide; Nitric Oxide Synthase Type II; Nitric Oxide Synthase Type III; Rats; Rats, Sprague-Dawley; RNA Interference; RNA, Small Interfering; Sodium-Hydrogen Exchangers; Sulfones; Time Factors; Vasodilation; Vasodilator Agents | 2008 |
Na+/H+ exchange inhibition with cariporide prevents alterations of coronary endothelial function in streptozotocin-induced diabetes.
Topics: 15-Hydroxy-11 alpha,9 alpha-(epoxymethano)prosta-5,13-dienoic Acid; Animals; Blood Glucose; Carbachol; Diabetes Mellitus, Experimental; Dilatation; Endothelium, Vascular; Glucose; Guanidines; Heart; Heart Function Tests; In Vitro Techniques; Lactic Acid; Male; Monocarboxylic Acid Transporters; Nitroprusside; Rats; Rats, Wistar; Sodium-Hydrogen Exchanger 1; Sodium-Hydrogen Exchangers; Streptozocin; Sulfones; Symporters | 2008 |
Slowly inactivating component of sodium current in ventricular myocytes is decreased by diabetes and partially inhibited by known Na(+)-H(+)Exchange blockers.
Topics: Amiloride; Animals; Anti-Arrhythmia Agents; Cells, Cultured; Diabetes Mellitus, Experimental; Dose-Response Relationship, Drug; Guanidines; Heart Ventricles; Lysophosphatidylcholines; Male; Myocardium; Patch-Clamp Techniques; Rats; Rats, Wistar; Sodium Channels; Sodium-Hydrogen Exchangers; Streptozocin; Sulfones; Tetrodotoxin; Veratrine | 2000 |
Diabetes-induced vascular hypertrophy is accompanied by activation of Na(+)-H(+) exchange and prevented by Na(+)-H(+) exchange inhibition.
Topics: Animals; Anti-Arrhythmia Agents; Blood Vessels; Diabetes Mellitus, Experimental; Disease Models, Animal; Guanidines; Hydrogen-Ion Concentration; Hypertrophy; Male; Mesenteric Arteries; Muscle, Smooth, Vascular; Rats; Rats, Sprague-Dawley; Sodium-Hydrogen Exchangers; Streptozocin; Sulfones | 2000 |