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carbonyl cyanide p-trifluoromethoxyphenylhydrazone and Myocardial Ischemia

carbonyl cyanide p-trifluoromethoxyphenylhydrazone has been researched along with Myocardial Ischemia in 6 studies

Carbonyl Cyanide p-Trifluoromethoxyphenylhydrazone: A proton ionophore that is commonly used as an uncoupling agent in biochemical studies.
carbonyl cyanide p-trifluoromethoxyphenylhydrazone : A hydrazone that is hydrazonomalononitrile in which one of the hydrazine hydrogens is substituted by a p-trifluoromethoxyphenyl group.

Myocardial Ischemia: A disorder of cardiac function caused by insufficient blood flow to the muscle tissue of the heart. The decreased blood flow may be due to narrowing of the coronary arteries (CORONARY ARTERY DISEASE), to obstruction by a thrombus (CORONARY THROMBOSIS), or less commonly, to diffuse narrowing of arterioles and other small vessels within the heart. Severe interruption of the blood supply to the myocardial tissue may result in necrosis of cardiac muscle (MYOCARDIAL INFARCTION).

Research

Studies (6)

TimeframeStudies, this research(%)All Research%
pre-19900 (0.00)18.7374
1990's1 (16.67)18.2507
2000's3 (50.00)29.6817
2010's2 (33.33)24.3611
2020's0 (0.00)2.80

Authors

AuthorsStudies
Kanaporis, G1
Treinys, R1
Fischmeister, R1
Jurevičius, J1
Smith, RM1
Velamakanni, SS1
Tolkacheva, EG1
Korge, P1
Honda, HM1
Weiss, JN1
Fukumoto, GH1
Lamp, ST1
Motter, C1
Bridge, JH1
Garfinkel, A1
Goldhaber, JI1
Brennan, JP1
Southworth, R1
Medina, RA1
Davidson, SM1
Duchen, MR1
Shattock, MJ1
Rouslin, W1
Broge, CW1

Other Studies

6 other studies available for carbonyl cyanide p-trifluoromethoxyphenylhydrazone and Myocardial Ischemia

ArticleYear
Metabolic inhibition reduces cardiac L-type Ca2+ channel current due to acidification caused by ATP hydrolysis.
    PloS one, 2017, Volume: 12, Issue:8

    Topics: Adenosine Triphosphate; Animals; Calcium Channels, L-Type; Carbonyl Cyanide p-Trifluoromethoxyphenyl

2017
Interventricular heterogeneity as a substrate for arrhythmogenesis of decoupled mitochondria during ischemia in the whole heart.
    American journal of physiology. Heart and circulatory physiology, 2012, Jul-15, Volume: 303, Issue:2

    Topics: Action Potentials; Animals; Carbonyl Cyanide p-Trifluoromethoxyphenylhydrazone; Glyburide; Heterocyc

2012
Effects of fatty acids in isolated mitochondria: implications for ischemic injury and cardioprotection.
    American journal of physiology. Heart and circulatory physiology, 2003, Volume: 285, Issue:1

    Topics: Adenosine Triphosphate; Animals; Calcium; Carbonyl Cyanide p-Trifluoromethoxyphenylhydrazone; Cell M

2003
Metabolic inhibition alters subcellular calcium release patterns in rat ventricular myocytes: implications for defective excitation-contraction coupling during cardiac ischemia and failure.
    Circulation research, 2005, Mar-18, Volume: 96, Issue:5

    Topics: Aerobiosis; Animals; Calcium; Calcium Channels, L-Type; Calcium Signaling; Carbonyl Cyanide p-Triflu

2005
Mitochondrial uncoupling, with low concentration FCCP, induces ROS-dependent cardioprotection independent of KATP channel activation.
    Cardiovascular research, 2006, Nov-01, Volume: 72, Issue:2

    Topics: Acetylcysteine; Adenosine Triphosphate; Animals; Anti-Arrhythmia Agents; Carbonyl Cyanide p-Trifluor

2006
IF1 function in situ in uncoupler-challenged ischemic rabbit, rat, and pigeon hearts.
    The Journal of biological chemistry, 1996, Sep-27, Volume: 271, Issue:39

    Topics: Adenosine Triphosphate; Animals; ATPase Inhibitory Protein; Carbonyl Cyanide p-Trifluoromethoxypheny

1996