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carbonyl cyanide m-chlorophenyl hydrazone and Reperfusion Injury

carbonyl cyanide m-chlorophenyl hydrazone has been researched along with Reperfusion Injury in 3 studies

Carbonyl Cyanide m-Chlorophenyl Hydrazone: A proton ionophore. It is commonly used as an uncoupling agent and inhibitor of photosynthesis because of its effects on mitochondrial and chloroplast membranes.
CCCP : A member of the class of monochlorobenzenes that is benzene substituted by 2-(1,3-dinitrilopropan-2-ylidene)hydrazinyl and chloro groups at positions 1 and 3, respectively. It is a mitochondrial depolarizing agent that induces reactive oxygen species mediated cell death.

Reperfusion Injury: Adverse functional, metabolic, or structural changes in tissues that result from the restoration of blood flow to the tissue (REPERFUSION) following ISCHEMIA.

Research Excerpts

ExcerptRelevanceReference
"Hypoxia/reperfusion injury is a major clinical problem."5.29Cyclosporine A protects mitochondria in an in vitro model of hypoxia/reperfusion injury. ( Gogvadze, V; Richter, C, 1993)
"Hypoxia/reperfusion injury is a major clinical problem."1.29Cyclosporine A protects mitochondria in an in vitro model of hypoxia/reperfusion injury. ( Gogvadze, V; Richter, C, 1993)

Research

Studies (3)

TimeframeStudies, this research(%)All Research%
pre-19900 (0.00)18.7374
1990's1 (33.33)18.2507
2000's1 (33.33)29.6817
2010's1 (33.33)24.3611
2020's0 (0.00)2.80

Authors

AuthorsStudies
Wang, S1
Kwon, SH1
Su, Y1
Dong, Z2
Wei, Q1
Alam, MM1
Wang, MH1
Yu, F1
Gogvadze, V1
Richter, C1

Other Studies

3 other studies available for carbonyl cyanide m-chlorophenyl hydrazone and Reperfusion Injury

ArticleYear
Stress granules are formed in renal proximal tubular cells during metabolic stress and ischemic injury for cell survival.
    American journal of physiology. Renal physiology, 2019, 07-01, Volume: 317, Issue:1

    Topics: Acute Kidney Injury; Animals; Carbonyl Cyanide m-Chlorophenyl Hydrazone; Cell Survival; Cells, Cultu

2019
Bid activation in kidney cells following ATP depletion in vitro and ischemia in vivo.
    American journal of physiology. Renal physiology, 2004, Volume: 286, Issue:4

    Topics: Acute Kidney Injury; Adenosine Triphosphate; Animals; Apoptosis; Azides; BH3 Interacting Domain Deat

2004
Cyclosporine A protects mitochondria in an in vitro model of hypoxia/reperfusion injury.
    FEBS letters, 1993, Nov-01, Volume: 333, Issue:3

    Topics: Adenosine Diphosphate; Animals; Calcium; Carbonyl Cyanide m-Chlorophenyl Hydrazone; Cyclosporine; Eg

1993