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carbonyl cyanide m-chlorophenyl hydrazone and Burkitt Lymphoma

carbonyl cyanide m-chlorophenyl hydrazone has been researched along with Burkitt Lymphoma in 2 studies

Carbonyl Cyanide m-Chlorophenyl Hydrazone: A proton ionophore. It is commonly used as an uncoupling agent and inhibitor of photosynthesis because of its effects on mitochondrial and chloroplast membranes.
CCCP : A member of the class of monochlorobenzenes that is benzene substituted by 2-(1,3-dinitrilopropan-2-ylidene)hydrazinyl and chloro groups at positions 1 and 3, respectively. It is a mitochondrial depolarizing agent that induces reactive oxygen species mediated cell death.

Burkitt Lymphoma: A form of undifferentiated malignant LYMPHOMA usually found in central Africa, but also reported in other parts of the world. It is commonly manifested as a large osteolytic lesion in the jaw or as an abdominal mass. B-cell antigens are expressed on the immature cells that make up the tumor in virtually all cases of Burkitt lymphoma. The Epstein-Barr virus (HERPESVIRUS 4, HUMAN) has been isolated from Burkitt lymphoma cases in Africa and it is implicated as the causative agent in these cases; however, most non-African cases are EBV-negative.

Research

Studies (2)

TimeframeStudies, this research(%)All Research%
pre-19900 (0.00)18.7374
1990's0 (0.00)18.2507
2000's1 (50.00)29.6817
2010's1 (50.00)24.3611
2020's0 (0.00)2.80

Authors

AuthorsStudies
Lombardo, T1
Folgar, MG1
Salaverry, L1
Rey-Roldán, E1
Alvarez, EM1
Carreras, MC1
Kornblihtt, L1
Blanco, GA1
O'Brien, KA1
Muscarella, DE1
Bloom, SE1

Other Studies

2 other studies available for carbonyl cyanide m-chlorophenyl hydrazone and Burkitt Lymphoma

ArticleYear
Regulated Cell Death of Lymphoma Cells after Graded Mitochondrial Damage is Differentially Affected by Drugs Targeting Cell Stress Responses.
    Basic & clinical pharmacology & toxicology, 2018, Volume: 122, Issue:5

    Topics: Autophagosomes; Autophagy; Burkitt Lymphoma; Carbonyl Cyanide m-Chlorophenyl Hydrazone; Cell Line, T

2018
Differential induction of apoptosis and MAP kinase signaling by mitochondrial toxicants in drug-sensitive compared to drug-resistant B-lineage lymphoid cell lines.
    Toxicology and applied pharmacology, 2001, Aug-01, Volume: 174, Issue:3

    Topics: Antimycin A; Antineoplastic Agents; Apoptosis; B-Lymphocytes; bcl-2-Associated X Protein; Burkitt Ly

2001