carbon tetrachloride has been researched along with Obesity in 8 studies
Carbon Tetrachloride: A solvent for oils, fats, lacquers, varnishes, rubber waxes, and resins, and a starting material in the manufacturing of organic compounds. Poisoning by inhalation, ingestion or skin absorption is possible and may be fatal. (Merck Index, 11th ed)
tetrachloromethane : A chlorocarbon that is methane in which all the hydrogens have been replaced by chloro groups.
Obesity: A status with BODY WEIGHT that is grossly above the recommended standards, usually due to accumulation of excess FATS in the body. The standards may vary with age, sex, genetic or cultural background. In the BODY MASS INDEX, a BMI greater than 30.0 kg/m2 is considered obese, and a BMI greater than 40.0 kg/m2 is considered morbidly obese (MORBID OBESITY).
| Excerpt | Relevance | Reference |
|---|---|---|
| " C57BL/6N mice were fed a high-fat diet (HFD) to develop obesity and were either administered carbon tetrachloride (CCl4 ) eight times (0." | 3.79 | A high-fat diet and multiple administration of carbon tetrachloride induces liver injury and pathological features associated with non-alcoholic steatohepatitis in mice. ( Ishikawa, F; Kado, S; Kano, M; Kobayashi, T; Kubota, N; Masuoka, N; Miyazaki, K; Nagata, Y, 2013) |
| "Obesity is associated with hyperleptinemia and is also a risk factor for fibrosis and severity of fibrosis in several chronic liver diseases." | 3.71 | Leptin is essential for the hepatic fibrogenic response to chronic liver injury. ( Farrell, GC; Leclercq, IA; Robertson, GR; Schriemer, R, 2002) |
| "Non-alcoholic fatty liver disease (NAFLD) is the most common liver disease in the Western world, and it is closely associated to obesity, type 2 diabetes mellitus, and dyslipidemia." | 1.62 | Δ9-Tetrahydrocannabinolic Acid markedly alleviates liver fibrosis and inflammation in mice. ( Appendino, G; Carmona-Hidalgo, B; García-Martín, A; González-Mariscal, I; Muñoz, E; Prados, ME; Ruiz-Pino, F; Tena-Sempere, M, 2021) |
| Timeframe | Studies, this research(%) | All Research% |
|---|---|---|
| pre-1990 | 0 (0.00) | 18.7374 |
| 1990's | 0 (0.00) | 18.2507 |
| 2000's | 3 (37.50) | 29.6817 |
| 2010's | 2 (25.00) | 24.3611 |
| 2020's | 3 (37.50) | 2.80 |
| Authors | Studies |
|---|---|
| Xu, H | 1 |
| Zhao, Q | 1 |
| Song, N | 1 |
| Yan, Z | 1 |
| Lin, R | 1 |
| Wu, S | 1 |
| Jiang, L | 1 |
| Hong, S | 1 |
| Xie, J | 1 |
| Zhou, H | 1 |
| Wang, R | 1 |
| Jiang, X | 1 |
| Carmona-Hidalgo, B | 1 |
| González-Mariscal, I | 1 |
| García-Martín, A | 1 |
| Prados, ME | 1 |
| Ruiz-Pino, F | 1 |
| Appendino, G | 1 |
| Tena-Sempere, M | 1 |
| Muñoz, E | 1 |
| Price, NL | 1 |
| Zhang, X | 1 |
| Fernández-Tussy, P | 1 |
| Singh, AK | 1 |
| Burnap, SA | 1 |
| Rotllan, N | 1 |
| Goedeke, L | 1 |
| Sun, J | 1 |
| Canfrán-Duque, A | 1 |
| Aryal, B | 1 |
| Mayr, M | 1 |
| Suárez, Y | 1 |
| Fernández-Hernando, C | 1 |
| Kubota, N | 1 |
| Kado, S | 1 |
| Kano, M | 1 |
| Masuoka, N | 1 |
| Nagata, Y | 1 |
| Kobayashi, T | 1 |
| Miyazaki, K | 1 |
| Ishikawa, F | 1 |
| Chatterjee, S | 1 |
| Rana, R | 1 |
| Corbett, J | 1 |
| Kadiiska, MB | 1 |
| Goldstein, J | 1 |
| Mason, RP | 1 |
| Leclercq, IA | 1 |
| Farrell, GC | 1 |
| Schriemer, R | 1 |
| Robertson, GR | 1 |
| Potter, JJ | 1 |
| Rennie-Tankesley, L | 1 |
| Mezey, E | 1 |
| Martínez-Chantar, ML | 1 |
| Corrales, FJ | 1 |
| Martínez-Cruz, LA | 1 |
| García-Trevijano, ER | 1 |
| Huang, ZZ | 1 |
| Chen, L | 1 |
| Kanel, G | 1 |
| Avila, MA | 1 |
| Mato, JM | 1 |
| Lu, SC | 1 |
8 other studies available for carbon tetrachloride and Obesity
| Article | Year |
|---|---|
AdipoR1/AdipoR2 dual agonist recovers nonalcoholic steatohepatitis and related fibrosis via endoplasmic reticulum-mitochondria axis.
Topics: Adenylate Kinase; Alanine Transaminase; Animals; Carbon Tetrachloride; Diet, High-Fat; Disease Model | 2020 |
Δ9-Tetrahydrocannabinolic Acid markedly alleviates liver fibrosis and inflammation in mice.
Topics: Animals; Cannabis; Carbon Tetrachloride; Diet, High-Fat; Dronabinol; Gene Expression Regulation; Hep | 2021 |
Loss of hepatic miR-33 improves metabolic homeostasis and liver function without altering body weight or atherosclerosis.
Topics: Animals; Atherosclerosis; Biological Transport; Body Weight; Carbon Tetrachloride; Cholesterol; Diet | 2021 |
A high-fat diet and multiple administration of carbon tetrachloride induces liver injury and pathological features associated with non-alcoholic steatohepatitis in mice.
Topics: Alanine Transaminase; Animals; Apoptosis; Carbon Tetrachloride; Diet, High-Fat; Disease Models, Anim | 2013 |
P2X7 receptor-NADPH oxidase axis mediates protein radical formation and Kupffer cell activation in carbon tetrachloride-mediated steatohepatitis in obese mice.
Topics: Animals; Carbon Tetrachloride; Coculture Techniques; Fatty Liver; Kupffer Cells; Male; Mice; Mice, I | 2012 |
Leptin is essential for the hepatic fibrogenic response to chronic liver injury.
Topics: Animals; Carbon Tetrachloride; Chronic Disease; Disease Progression; Fatty Liver; Female; Leptin; Li | 2002 |
Influence of leptin in the development of hepatic fibrosis produced in mice by Schistosoma mansoni infection and by chronic carbon tetrachloride administration.
Topics: Animals; Carbon Tetrachloride; Cytokines; Drug Administration Schedule; Leptin; Liver; Liver Cirrhos | 2003 |
Spontaneous oxidative stress and liver tumors in mice lacking methionine adenosyltransferase 1A.
Topics: Animals; Carbon Tetrachloride; Chemical and Drug Induced Liver Injury; Cytochrome P-450 CYP2E1; Diab | 2002 |