carbon tetrachloride has been researched along with Genetic Predisposition in 13 studies
Carbon Tetrachloride: A solvent for oils, fats, lacquers, varnishes, rubber waxes, and resins, and a starting material in the manufacturing of organic compounds. Poisoning by inhalation, ingestion or skin absorption is possible and may be fatal. (Merck Index, 11th ed)
tetrachloromethane : A chlorocarbon that is methane in which all the hydrogens have been replaced by chloro groups.
| Excerpt | Relevance | Reference |
|---|---|---|
| " In this study, apolipoprotein E-deficient (ApoE-/-) mice were evaluated to define potential effects of hypercholesterolemia on the severity of carbon tetrachloride (CCl4)-induced liver injury." | 3.75 | Increased susceptibility to exacerbated liver injury in hypercholesterolemic ApoE-deficient mice: potential involvement of oxysterols. ( Arroyo, V; Bataller, R; Camps, J; Clària, J; Ferré, N; González-Périz, A; Guardiola, F; Horrillo, R; Joven, J; López-Parra, M; Martínez-Clemente, M; Planagumà, A; Tres, A, 2009) |
| "Liver fibrosis is a multifactorial trait that develops in response to chronic liver injury." | 1.62 | The Genetic Architecture of Carbon Tetrachloride-Induced Liver Fibrosis in Mice. ( Asaryan, A; Beaven, SW; Borawski, J; Cantor, RM; Carbone, W; Civelek, M; Clerkin, K; French, S; Fuqua, BK; Haroutunian, SG; Hui, ST; Knehr, J; Loureiro, J; Lusis, AJ; Magyar, C; Mehrabian, M; Pan, C; Parks, BW; Renaud, N; Roma, G; Tuominen, I; Ukomadu, C; Wroblewski, K, 2021) |
| "We tested in patients with suspected nonalcoholic steatohepatitis (NASH) the association of FNDC5 variants, hepatic expression, and circulating irisin with liver damage (F2 to F4 fibrosis as main outcome)." | 1.46 | Fibronectin Type III Domain-Containing Protein 5 rs3480 A>G Polymorphism, Irisin, and Liver Fibrosis in Patients With Nonalcoholic Fatty Liver Disease. ( Cabibi, D; Cammà, C; Craxì, A; Di Marco, V; Dongiovanni, P; Fargion, S; Ferri, N; Fracanzani, AL; Giordano, D; Grimaudo, S; Maggioni, M; Meroni, M; Petta, S; Pierantonelli, I; Pipitone, RM; Ruscica, M; Rychlicki, C; Svegliati-Baroni, G; Valenti, L, 2017) |
| Timeframe | Studies, this research(%) | All Research% |
|---|---|---|
| pre-1990 | 0 (0.00) | 18.7374 |
| 1990's | 0 (0.00) | 18.2507 |
| 2000's | 4 (30.77) | 29.6817 |
| 2010's | 8 (61.54) | 24.3611 |
| 2020's | 1 (7.69) | 2.80 |
| Authors | Studies |
|---|---|
| Xu, P | 1 |
| Yao, J | 1 |
| Ji, J | 1 |
| Shi, H | 2 |
| Jiao, Y | 1 |
| Hao, S | 1 |
| Chen, D | 1 |
| Tuominen, I | 1 |
| Fuqua, BK | 1 |
| Pan, C | 1 |
| Renaud, N | 1 |
| Wroblewski, K | 1 |
| Civelek, M | 1 |
| Clerkin, K | 1 |
| Asaryan, A | 1 |
| Haroutunian, SG | 1 |
| Loureiro, J | 1 |
| Borawski, J | 1 |
| Roma, G | 1 |
| Knehr, J | 1 |
| Carbone, W | 1 |
| French, S | 1 |
| Parks, BW | 1 |
| Hui, ST | 1 |
| Mehrabian, M | 1 |
| Magyar, C | 1 |
| Cantor, RM | 1 |
| Ukomadu, C | 1 |
| Lusis, AJ | 1 |
| Beaven, SW | 1 |
| Petta, S | 1 |
| Valenti, L | 1 |
| Svegliati-Baroni, G | 1 |
| Ruscica, M | 1 |
| Pipitone, RM | 1 |
| Dongiovanni, P | 1 |
| Rychlicki, C | 1 |
| Ferri, N | 1 |
| Cammà, C | 1 |
| Fracanzani, AL | 1 |
| Pierantonelli, I | 1 |
| Di Marco, V | 1 |
| Meroni, M | 1 |
| Giordano, D | 1 |
| Grimaudo, S | 1 |
| Maggioni, M | 1 |
| Cabibi, D | 1 |
| Fargion, S | 1 |
| Craxì, A | 1 |
| Liebe, R | 1 |
| Hall, RA | 2 |
| Williams, RW | 1 |
| Dooley, S | 1 |
| Lammert, F | 2 |
| Hillebrandt, S | 1 |
| Mu, X | 1 |
| Pradere, JP | 1 |
| Affò, S | 1 |
| Dapito, DH | 1 |
| Friedman, R | 1 |
| Lefkovitch, JH | 1 |
| Schwabe, RF | 1 |
| Ferré, N | 1 |
| Martínez-Clemente, M | 1 |
| López-Parra, M | 1 |
| González-Périz, A | 1 |
| Horrillo, R | 1 |
| Planagumà, A | 1 |
| Camps, J | 1 |
| Joven, J | 1 |
| Tres, A | 1 |
| Guardiola, F | 1 |
| Bataller, R | 1 |
| Arroyo, V | 1 |
| Clària, J | 1 |
| Yun, JW | 1 |
| Lee, TR | 1 |
| Kim, CW | 1 |
| Park, YH | 1 |
| Chung, JH | 1 |
| Lee, YS | 1 |
| Kang, KS | 1 |
| Lim, KM | 1 |
| Barry, KH | 1 |
| Zhang, Y | 1 |
| Lan, Q | 1 |
| Zahm, SH | 1 |
| Holford, TR | 1 |
| Leaderer, B | 1 |
| Boyle, P | 1 |
| Hosgood, HD | 1 |
| Chanock, S | 1 |
| Yeager, M | 1 |
| Rothman, N | 1 |
| Zheng, T | 1 |
| Wang, C | 1 |
| Liu, YR | 1 |
| Zhou, Y | 1 |
| Li, AP | 1 |
| Zhou, JW | 1 |
| Ma, W | 1 |
| Xia, X | 1 |
| Stafford, LJ | 1 |
| Yu, C | 1 |
| Wang, F | 1 |
| LeSage, G | 1 |
| Liu, M | 1 |
| Strnad, P | 1 |
| Tao, GZ | 1 |
| Zhou, Q | 1 |
| Harada, M | 1 |
| Toivola, DM | 1 |
| Brunt, EM | 1 |
| Omary, MB | 1 |
| Yu, J | 1 |
| Wu, CW | 1 |
| Chu, ES | 1 |
| Hui, AY | 1 |
| Cheng, AS | 1 |
| Go, MY | 1 |
| Ching, AK | 1 |
| Chui, YL | 1 |
| Chan, HL | 1 |
| Sung, JJ | 1 |
13 other studies available for carbon tetrachloride and Genetic Predisposition
| Article | Year |
|---|---|
Deficiency of apoptosis-stimulating protein 2 of p53 protects mice from acute hepatic injury induced by CCl
Topics: Alanine Transaminase; Animals; Apoptosis; Aspartate Aminotransferases; Autophagy; Carbon Tetrachlori | 2019 |
The Genetic Architecture of Carbon Tetrachloride-Induced Liver Fibrosis in Mice.
Topics: Animals; Carbon Tetrachloride; Disease Models, Animal; Gene Regulatory Networks; Genetic Predisposit | 2021 |
Fibronectin Type III Domain-Containing Protein 5 rs3480 A>G Polymorphism, Irisin, and Liver Fibrosis in Patients With Nonalcoholic Fatty Liver Disease.
Topics: Adult; Animals; Carbon Tetrachloride; Case-Control Studies; Diet, High-Fat; Female; Fibronectins; Ge | 2017 |
Systems genetics of hepatocellular damage in vivo and in vitro: identification of a critical network on chromosome 11 in mouse.
Topics: Animals; Carbon Tetrachloride; Chromosome Mapping; Chromosomes, Mammalian; Ethanol; Female; Gene Exp | 2013 |
Exploring multiple quantitative trait loci models of hepatic fibrosis in a mouse intercross.
Topics: Alleles; Animals; Carbon Tetrachloride; Chemical and Drug Induced Liver Injury, Chronic; Chimera; Ch | 2016 |
Epithelial Transforming Growth Factor-β Signaling Does Not Contribute to Liver Fibrosis but Protects Mice From Cholangiocarcinoma.
Topics: Animals; ATP Binding Cassette Transporter, Subfamily B; ATP-Binding Cassette Sub-Family B Member 4; | 2016 |
Increased susceptibility to exacerbated liver injury in hypercholesterolemic ApoE-deficient mice: potential involvement of oxysterols.
Topics: Animals; Apolipoproteins E; Carbon Tetrachloride; Chemical and Drug Induced Liver Injury; Chemokine | 2009 |
Predose blood gene expression profiles might identify the individuals susceptible to carbon tetrachloride-induced hepatotoxicity.
Topics: Animals; Carbon Tetrachloride; Chemical and Drug Induced Liver Injury; Gene Expression; Gene Express | 2010 |
Genetic variation in metabolic genes, occupational solvent exposure, and risk of non-hodgkin lymphoma.
Topics: Adult; Carbon Tetrachloride; Connecticut; Cytochrome P-450 CYP2E1; Epoxide Hydrolases; Female; Genet | 2011 |
[The study of susceptibility to carbon tetrachloride and benzene in offspring of expanded simple tandem repeats mutation mice exposed to formaldehyde].
Topics: Alanine Transaminase; Animals; Aspartate Aminotransferases; Benzene; Carbon Tetrachloride; Chemical | 2011 |
Expression of GCIP in transgenic mice decreases susceptibility to chemical hepatocarcinogenesis.
Topics: Animals; Carbon Tetrachloride; Carcinogens; Cell Line, Tumor; Female; Genetic Predisposition to Dise | 2006 |
Keratin mutation predisposes to mouse liver fibrosis and unmasks differential effects of the carbon tetrachloride and thioacetamide models.
Topics: Animals; Arsenamide; Carbon Tetrachloride; Disease Models, Animal; Female; Genetic Predisposition to | 2008 |
Elucidation of the role of COX-2 in liver fibrogenesis using transgenic mice.
Topics: Alanine Transaminase; Animals; Bilirubin; Body Weight; Carbon Tetrachloride; Collagen; Cyclooxygenas | 2008 |