carbon monoxide has been researched along with Pre-Eclampsia in 42 studies
Carbon Monoxide: Carbon monoxide (CO). A poisonous colorless, odorless, tasteless gas. It combines with hemoglobin to form carboxyhemoglobin, which has no oxygen carrying capacity. The resultant oxygen deprivation causes headache, dizziness, decreased pulse and respiratory rates, unconsciousness, and death. (From Merck Index, 11th ed)
carbon monoxide : A one-carbon compound in which the carbon is joined only to a single oxygen. It is a colourless, odourless, tasteless, toxic gas.
Pre-Eclampsia: A complication of PREGNANCY, characterized by a complex of symptoms including maternal HYPERTENSION and PROTEINURIA with or without pathological EDEMA. Symptoms may range between mild and severe. Pre-eclampsia usually occurs after the 20th week of gestation, but may develop before this time in the presence of trophoblastic disease.
| Excerpt | Relevance | Reference |
|---|---|---|
| "End-tidal breath carbon monoxide (CO) is abnormally low in women with preeclampsia (PE), while women smoking during pregnancy have shown an increase in CO levels and a 33% lower incidence of PE." | 7.79 | Chronic carbon monoxide inhalation during pregnancy augments uterine artery blood flow and uteroplacental vascular growth in mice. ( Adamson, SL; Casselman, R; Murphy, MS; Sled, JG; Smith, GN; Venditti, CC, 2013) |
| "Carbon monoxide (CO) in cigarette smoke may be the mechanism by which tobacco use during pregnancy decreases the risk of the development of preeclampsia." | 7.78 | Maternal exposure to moderate ambient carbon monoxide is associated with decreased risk of preeclampsia. ( Guo, Y; Krewski, D; Smith, G; Walker, M; Wen, SW; Zhai, D, 2012) |
| " We postulated that elevated carbon monoxide concentrations in serum of smoking women inhibits apoptosis and debris shedding of trophoblast cells exposed to ischemia-reperfusion injury because carbon monoxide has cytoprotective effects on endothelial and smooth muscle cells in culture." | 7.73 | Carbon monoxide inhibits hypoxia/reoxygenation-induced apoptosis and secondary necrosis in syncytiotrophoblast. ( Bainbridge, SA; Belkacemi, L; Dickinson, M; Graham, CH; Smith, GN, 2006) |
| "We sought to compare the end-tidal carbon monoxide breath levels in pregnant women with and without pregnancy-induced hypertension and preeclampsia." | 7.70 | End-tidal carbon monoxide measurements in women with pregnancy-induced hypertension and preeclampsia. ( Baum, M; Dennery, PA; Kreiser, D; Rosenthal, T; Schiff, E; Seidman, DS; Stevenson, DK, 2000) |
| "Although family clustering of preeclampsia has been observed, the existing genetic literature is limited by a failure to consider both mother and child." | 5.48 | A Family Based Study of Carbon Monoxide and Nitric Oxide Signalling Genes and Preeclampsia. ( Avery, CL; Bauer, AE; Engel, SM; Harmon, QE; Klungsøyr, K; Luo, J; Magnus, P; Manuck, TA; McGinnis, R; Morgan, L; Olshan, AF; Shi, M; Trogstad, L; Weinberg, CR; Williams, N; Wu, MC; Yang, J, 2018) |
| "Preeclampsia is a pregnancy complication which manifests as new-onset hypertension, proteinuria, and a spectrum of other symptoms." | 5.46 | Carbon Monoxide Releasing Molecules Blunt Placental Ischemia-Induced Hypertension. ( Arany, M; Cockrell, K; Gadepalli, RSV; George, EM; Granger, JP; Rimoldi, JM; Stec, DE, 2017) |
| "Decreased DDAH and HO with preeclampsia suggest that they are important points in the regulatory pathways of NO and CO production that are altered in preeclampsia." | 5.39 | Nitric oxide and carbon monoxide production and metabolism in preeclampsia. ( Chan, K; Chen, DB; Ehsanipoor, RM; Fitzmaurice, LE; Fortson, W; Liao, WX; Wing, DA, 2013) |
| "Preterm delivery and preeclampsia are common adverse pregnancy outcomes that have been inconsistently associated with ambient air pollutant exposures." | 5.37 | Ambient carbon monoxide and fine particulate matter in relation to preeclampsia and preterm delivery in western Washington State. ( Koenig, JQ; Rudra, CB; Schiff, MA; Sheppard, L; Williams, MA, 2011) |
| "The three known gasotransmitters, nitric oxide, carbon monoxide, and hydrogen sulfide are involved in key processes throughout pregnancy." | 5.05 | Maternal disease and gasotransmitters. ( Boeldt, DS; Mauro, AK; Rengarajan, A, 2020) |
| "Smokers have a significantly decreased risk of pre-eclampsia (PE), possibly attributed to an increase in blood carbon monoxide (CO) concentrations." | 3.88 | Endogenous carbon monoxide production by menadione. ( Odozor, CU; Peterson, N; Pudwell, J; Smith, GN, 2018) |
| "End-tidal breath carbon monoxide (CO) is abnormally low in women with preeclampsia (PE), while women smoking during pregnancy have shown an increase in CO levels and a 33% lower incidence of PE." | 3.79 | Chronic carbon monoxide inhalation during pregnancy augments uterine artery blood flow and uteroplacental vascular growth in mice. ( Adamson, SL; Casselman, R; Murphy, MS; Sled, JG; Smith, GN; Venditti, CC, 2013) |
| "Carbon monoxide (CO) in cigarette smoke may be the mechanism by which tobacco use during pregnancy decreases the risk of the development of preeclampsia." | 3.78 | Maternal exposure to moderate ambient carbon monoxide is associated with decreased risk of preeclampsia. ( Guo, Y; Krewski, D; Smith, G; Walker, M; Wen, SW; Zhai, D, 2012) |
| " We postulated that elevated carbon monoxide concentrations in serum of smoking women inhibits apoptosis and debris shedding of trophoblast cells exposed to ischemia-reperfusion injury because carbon monoxide has cytoprotective effects on endothelial and smooth muscle cells in culture." | 3.73 | Carbon monoxide inhibits hypoxia/reoxygenation-induced apoptosis and secondary necrosis in syncytiotrophoblast. ( Bainbridge, SA; Belkacemi, L; Dickinson, M; Graham, CH; Smith, GN, 2006) |
| "We sought to compare the end-tidal carbon monoxide breath levels in pregnant women with and without pregnancy-induced hypertension and preeclampsia." | 3.70 | End-tidal carbon monoxide measurements in women with pregnancy-induced hypertension and preeclampsia. ( Baum, M; Dennery, PA; Kreiser, D; Rosenthal, T; Schiff, E; Seidman, DS; Stevenson, DK, 2000) |
| "Pregnancy is accompanied by several adaptations in the mother, such as increased blood volume, higher cardiac output and reduced peripheral vascular resistance." | 2.49 | Heme oxygenase-1 in placental development and pathology. ( Baczyk, D; Drewlo, S; Kingdom, J; Levytska, K, 2013) |
| "The incidence of preeclampsia is reduced by a third in smokers, but not in snuff users." | 2.47 | New insights into the etiology of preeclampsia: identification of key elusive factors for the vascular complications. ( Ahmed, A, 2011) |
| "Although family clustering of preeclampsia has been observed, the existing genetic literature is limited by a failure to consider both mother and child." | 1.48 | A Family Based Study of Carbon Monoxide and Nitric Oxide Signalling Genes and Preeclampsia. ( Avery, CL; Bauer, AE; Engel, SM; Harmon, QE; Klungsøyr, K; Luo, J; Magnus, P; Manuck, TA; McGinnis, R; Morgan, L; Olshan, AF; Shi, M; Trogstad, L; Weinberg, CR; Williams, N; Wu, MC; Yang, J, 2018) |
| "Preeclampsia is a pregnancy complication which manifests as new-onset hypertension, proteinuria, and a spectrum of other symptoms." | 1.46 | Carbon Monoxide Releasing Molecules Blunt Placental Ischemia-Induced Hypertension. ( Arany, M; Cockrell, K; Gadepalli, RSV; George, EM; Granger, JP; Rimoldi, JM; Stec, DE, 2017) |
| "Decreased DDAH and HO with preeclampsia suggest that they are important points in the regulatory pathways of NO and CO production that are altered in preeclampsia." | 1.39 | Nitric oxide and carbon monoxide production and metabolism in preeclampsia. ( Chan, K; Chen, DB; Ehsanipoor, RM; Fitzmaurice, LE; Fortson, W; Liao, WX; Wing, DA, 2013) |
| "Preterm delivery and preeclampsia are common adverse pregnancy outcomes that have been inconsistently associated with ambient air pollutant exposures." | 1.37 | Ambient carbon monoxide and fine particulate matter in relation to preeclampsia and preterm delivery in western Washington State. ( Koenig, JQ; Rudra, CB; Schiff, MA; Sheppard, L; Williams, MA, 2011) |
| "We observed elevated risks for preeclampsia, preterm birth, and very preterm birth from maternal exposures to traffic air pollutants measured at ambient stations (CO, NO, NO(2), and NO(x)) and modeled through CALINE4 (NO(x) and PM(2." | 1.37 | Comparing exposure assessment methods for traffic-related air pollution in an adverse pregnancy outcome study. ( Chung, J; Ritz, B; Wilhelm, M; Wu, J, 2011) |
| "During preeclampsia, vasculotoxic factors are released into the maternal circulation by the diseased placenta." | 1.36 | Pulmonary and systemic vascular dysfunction in young offspring of mothers with preeclampsia. ( Allemann, Y; Hutter, D; Jayet, PY; Nicod, P; Rexhaj, E; Rimoldi, SF; Salmòn, CS; Sartori, C; Sartori-Cucchia, C; Scherrer, U; Schwab, M; Stuber, T; Thalmann, S; Turini, P; Villena, M, 2010) |
| "Preeclampsia is characterized clinically by hypertension and proteinuria." | 1.34 | Negative regulation of soluble Flt-1 and soluble endoglin release by heme oxygenase-1. ( Abbas, A; Ahmad, S; Ahmed, A; Al-Ani, B; Chudasama, K; Coxall, H; Cudmore, M; Devey, LR; Fujisawa, T; Hewett, PW; Wigmore, SJ, 2007) |
| Timeframe | Studies, this research(%) | All Research% |
|---|---|---|
| pre-1990 | 0 (0.00) | 18.7374 |
| 1990's | 0 (0.00) | 18.2507 |
| 2000's | 11 (26.19) | 29.6817 |
| 2010's | 28 (66.67) | 24.3611 |
| 2020's | 3 (7.14) | 2.80 |
| Authors | Studies |
|---|---|
| Ren, CL | 1 |
| Slaven, JE | 1 |
| Haas, DM | 1 |
| Haneline, LS | 1 |
| Tiller, C | 1 |
| Hogg, G | 1 |
| Bjerregaard, J | 1 |
| Tepper, RS | 1 |
| Rengarajan, A | 1 |
| Mauro, AK | 1 |
| Boeldt, DS | 1 |
| Dastoorpoor, M | 1 |
| Khanjani, N | 1 |
| Moradgholi, A | 1 |
| Sarizadeh, R | 1 |
| Cheraghi, M | 1 |
| Estebsari, F | 1 |
| George, EM | 4 |
| Cockrell, K | 1 |
| Arany, M | 1 |
| Stec, DE | 1 |
| Rimoldi, JM | 1 |
| Gadepalli, RSV | 1 |
| Granger, JP | 4 |
| Das, UN | 1 |
| Bauer, AE | 1 |
| Avery, CL | 1 |
| Shi, M | 1 |
| Weinberg, CR | 1 |
| Olshan, AF | 1 |
| Harmon, QE | 1 |
| Luo, J | 1 |
| Yang, J | 1 |
| Manuck, TA | 1 |
| Wu, MC | 1 |
| Williams, N | 1 |
| McGinnis, R | 1 |
| Morgan, L | 1 |
| Klungsøyr, K | 1 |
| Trogstad, L | 1 |
| Magnus, P | 1 |
| Engel, SM | 1 |
| McRae, KE | 2 |
| Smith, GN | 11 |
| Odozor, CU | 1 |
| Peterson, N | 2 |
| Pudwell, J | 1 |
| Dickson, MA | 1 |
| Levytska, K | 1 |
| Kingdom, J | 1 |
| Baczyk, D | 1 |
| Drewlo, S | 1 |
| Holwerda, KM | 1 |
| Faas, MM | 1 |
| van Goor, H | 1 |
| Lely, AT | 1 |
| Venditti, CC | 3 |
| Casselman, R | 3 |
| Murphy, MS | 1 |
| Adamson, SL | 1 |
| Sled, JG | 1 |
| Craici, IM | 1 |
| Wagner, SJ | 1 |
| Weissgerber, TL | 1 |
| Grande, JP | 1 |
| Garovic, VD | 1 |
| Warrington, JP | 1 |
| Spradley, FT | 1 |
| Palei, AC | 1 |
| Pedersen, M | 1 |
| Stayner, L | 1 |
| Slama, R | 1 |
| Sørensen, M | 1 |
| Figueras, F | 1 |
| Nieuwenhuijsen, MJ | 1 |
| Raaschou-Nielsen, O | 1 |
| Dadvand, P | 1 |
| Young, I | 1 |
| Karumanchi, SA | 1 |
| Ahmed, A | 5 |
| Ramma, W | 2 |
| Mendola, P | 1 |
| Wallace, M | 1 |
| Liu, D | 1 |
| Robledo, C | 1 |
| Mӓnnistӧ, T | 1 |
| Grantz, KL | 1 |
| Cudmore, MJ | 2 |
| Al-Ani, B | 2 |
| Hewett, PW | 2 |
| Fujisawa, T | 2 |
| Saifeddine, M | 1 |
| Williams, H | 1 |
| Sissaoui, S | 1 |
| Jayaraman, PS | 1 |
| Ohba, M | 1 |
| Ahmad, S | 2 |
| Hollenberg, MD | 1 |
| Tranquilli, AL | 1 |
| Landi, B | 1 |
| Wang, Y | 1 |
| Jayet, PY | 1 |
| Rimoldi, SF | 1 |
| Stuber, T | 1 |
| Salmòn, CS | 1 |
| Hutter, D | 1 |
| Rexhaj, E | 1 |
| Thalmann, S | 1 |
| Schwab, M | 1 |
| Turini, P | 1 |
| Sartori-Cucchia, C | 1 |
| Nicod, P | 1 |
| Villena, M | 1 |
| Allemann, Y | 1 |
| Scherrer, U | 1 |
| Sartori, C | 1 |
| Rudra, CB | 1 |
| Williams, MA | 1 |
| Sheppard, L | 1 |
| Koenig, JQ | 1 |
| Schiff, MA | 1 |
| Wu, J | 1 |
| Wilhelm, M | 1 |
| Chung, J | 1 |
| Ritz, B | 1 |
| Zhai, D | 1 |
| Guo, Y | 1 |
| Smith, G | 1 |
| Krewski, D | 1 |
| Walker, M | 1 |
| Wen, SW | 1 |
| Harper, LM | 1 |
| Keegan, MB | 1 |
| McPherson, JA | 1 |
| Trudell, AS | 1 |
| Ehsanipoor, RM | 1 |
| Fortson, W | 1 |
| Fitzmaurice, LE | 1 |
| Liao, WX | 1 |
| Wing, DA | 1 |
| Chen, DB | 1 |
| Chan, K | 1 |
| Lyall, F | 2 |
| Kreiser, D | 2 |
| Baum, M | 2 |
| Seidman, DS | 2 |
| Fanaroff, A | 1 |
| Shah, D | 1 |
| Hendler, I | 1 |
| Stevenson, DK | 2 |
| Schiff, E | 2 |
| Druzin, ML | 1 |
| Bainbridge, SA | 3 |
| Sidle, EH | 2 |
| Belkacemi, L | 1 |
| Dickinson, M | 1 |
| Graham, CH | 2 |
| Cudmore, M | 1 |
| Coxall, H | 1 |
| Chudasama, K | 1 |
| Devey, LR | 1 |
| Wigmore, SJ | 1 |
| Abbas, A | 1 |
| Dennery, PA | 1 |
| Rosenthal, T | 1 |
| Myatt, L | 1 |
| Appleton, SD | 1 |
| Marks, GS | 1 |
| Nakatsu, K | 1 |
| Brien, JF | 1 |
| Trial | Phase | Enrollment | Study Type | Start Date | Status | ||
|---|---|---|---|---|---|---|---|
| Pravastatin to Prevent Preeclampsia and Reduce Maternal-Neonatal Mortality and Morbidity in High Risk Preeclampsia Patients[NCT03648970] | Phase 2 | 280 participants (Anticipated) | Interventional | 2018-03-01 | Recruiting | ||
| Imaging Innovations for Placental Assessment in Response to Environmental Pollution[NCT02786420] | 199 participants (Actual) | Observational | 2015-10-02 | Completed | |||
| [information is prepared from clinicaltrials.gov, extracted Sep-2024] | |||||||
12 reviews available for carbon monoxide and Pre-Eclampsia
| Article | Year |
|---|---|
Maternal disease and gasotransmitters.
Topics: Animals; Carbon Monoxide; Female; Gasotransmitters; Humans; Hydrogen Sulfide; Nitric Oxide; Parturit | 2020 |
Heme oxygenase-1 in placental development and pathology.
Topics: Abortion, Spontaneous; Animals; Carbon Monoxide; Female; Heme Oxygenase-1; Humans; Placenta; Placent | 2013 |
Gasotransmitters: a solution for the therapeutic dilemma in preeclampsia?
Topics: Carbon Monoxide; Female; Humans; Hydrogen Sulfide; Neurotransmitter Agents; Nitric Oxide; Placenta; | 2013 |
Advances in the pathophysiology of pre-eclampsia and related podocyte injury.
Topics: Adaptive Immunity; Animals; Carbon Monoxide; Carrier Proteins; Endothelin-1; Endothelium, Vascular; | 2014 |
The heme oxygenases: important regulators of pregnancy and preeclampsia.
Topics: Animals; Bilirubin; Carbon Monoxide; Female; Heme; Heme Oxygenase (Decyclizing); Humans; Placenta; P | 2014 |
Ambient air pollution and pregnancy-induced hypertensive disorders: a systematic review and meta-analysis.
Topics: Adult; Air Pollution; Carbon Monoxide; Female; Humans; Hypertension, Pregnancy-Induced; Nitrogen Oxi | 2014 |
Unravelling the theories of pre-eclampsia: are the protective pathways the new paradigm?
Topics: Animals; Carbon Monoxide; Drug Design; Female; Humans; Hydrogen Sulfide; Inflammation; Oxidative Str | 2015 |
Involvement of the heme oxygenase system in the development of preeclampsia and as a possible therapeutic target.
Topics: Carbon Monoxide; Female; Heme Oxygenase (Decyclizing); Humans; Pre-Eclampsia; Pregnancy; Smoking | 2014 |
New insights into the etiology of preeclampsia: identification of key elusive factors for the vascular complications.
Topics: Antigens, CD; Carbon Monoxide; Endoglin; Female; Heme Oxygenase-1; Humans; Pre-Eclampsia; Pregnancy; | 2011 |
Heme oxygenase in pregnancy and preeclampsia.
Topics: Animals; Bilirubin; Blood Pressure; Carbon Monoxide; Female; Heme Oxygenase (Decyclizing); Humans; P | 2013 |
Development of the utero-placental circulation: the role of carbon monoxide and nitric oxide in trophoblast invasion and spiral artery transformation.
Topics: Arteries; Carbon Monoxide; Female; Fetal Growth Retardation; Humans; Immunohistochemistry; Microscop | 2003 |
HO in pregnancy.
Topics: Bilirubin; Biliverdine; Carbon Monoxide; Female; Heme Oxygenase (Decyclizing); Humans; Pre-Eclampsia | 2005 |
30 other studies available for carbon monoxide and Pre-Eclampsia
| Article | Year |
|---|---|
Forced expiratory flows and diffusion capacity in infants born from mothers with pre-eclampsia.
Topics: Carbon Monoxide; Female; Forced Expiratory Volume; Humans; Infant; Infant, Newborn; Infant, Prematur | 2022 |
Prenatal exposure to ambient air pollution and adverse pregnancy outcomes in Ahvaz, Iran: a generalized additive model.
Topics: Air Pollutants; Air Pollution; Carbon Monoxide; Diabetes, Gestational; Female; Fetal Macrosomia; Hum | 2021 |
Carbon Monoxide Releasing Molecules Blunt Placental Ischemia-Induced Hypertension.
Topics: Animals; Antihypertensive Agents; Blood Pressure; Carbon Monoxide; Disease Models, Animal; Female; G | 2017 |
Angiogenic, Antiangiogenic Molecules, and Bioactive Lipids in Preeclampsia.
Topics: Carbon Monoxide; Female; Humans; Hypertension; Lipids; Placenta; Pre-Eclampsia; Pregnancy | 2017 |
A Family Based Study of Carbon Monoxide and Nitric Oxide Signalling Genes and Preeclampsia.
Topics: Adult; Carbon Monoxide; Case-Control Studies; Female; Gene Frequency; Genes; Genetic Predisposition | 2018 |
Using Carbon Monoxide Releasing Molecules in Models of Preeclampsia: When Should We Be Monitoring Vascular Effects?
Topics: Carbon Monoxide; Female; Humans; Hypertension; Ischemia; Placenta; Pre-Eclampsia; Pregnancy | 2017 |
Response to: Using Carbon Monoxide Releasing Molecules in Models of Pre-Eclampsia: When Should We Be Monitoring Vascular Effects?
Topics: Carbon Monoxide; Female; Humans; Hypertension; Ischemia; Placenta; Pre-Eclampsia; Pregnancy | 2017 |
Endogenous carbon monoxide production by menadione.
Topics: Animals; Carbon Monoxide; Drug Evaluation, Preclinical; Female; Mice; Placenta; Pre-Eclampsia; Pregn | 2018 |
CORM-A1 treatment leads to increased carbon monoxide in pregnant mice.
Topics: Animals; Blood Gas Analysis; Boranes; Carbon Monoxide; Carbonates; Disease Models, Animal; Female; H | 2018 |
Chronic carbon monoxide inhalation during pregnancy augments uterine artery blood flow and uteroplacental vascular growth in mice.
Topics: Animals; Carbon Monoxide; Female; Mice; Placenta; Pre-Eclampsia; Pregnancy; Regional Blood Flow; Ute | 2013 |
Carbon monoxide prevents hypertension and proteinuria in an adenovirus sFlt-1 preeclampsia-like mouse model.
Topics: Adenoviridae; Animals; Carbon Monoxide; Disease Models, Animal; Female; Hypertension; Mice; Mothers; | 2014 |
Air pollution exposure and preeclampsia among US women with and without asthma.
Topics: Adult; Air Pollutants; Air Pollution; Asthma; Carbon Monoxide; Female; Humans; Nitrogen Oxides; Part | 2016 |
Can the biology of VEGF and haem oxygenases help solve pre-eclampsia?
Topics: Animals; Carbon Monoxide; Endothelium, Vascular; Female; Heme Oxygenase-1; Homeostasis; Humans; Hydr | 2009 |
Activation of proteinase-activated receptor 2 stimulates soluble vascular endothelial growth factor receptor 1 release via epidermal growth factor receptor transactivation in endothelial cells.
Topics: Carbon Monoxide; Cells, Cultured; Down-Regulation; Endothelial Cells; ErbB Receptors; Factor Xa; Fem | 2010 |
The origin of pre-eclampsia: from decidual "hyperoxia" to late hypoxia.
Topics: Carbon Monoxide; Female; Humans; Hyperoxia; Hypoxia; Leptin; Oxidative Stress; Pre-Eclampsia; Pregna | 2010 |
Re: Cudihy D, Lee RV. 2009. The pathophysiology of pre-eclampsia: current clinical concepts. Journal of Obstetrics and Gynaecology 29(7):576-582.
Topics: Carbon Monoxide; Female; Humans; Pre-Eclampsia; Pregnancy; Smoking | 2010 |
Pulmonary and systemic vascular dysfunction in young offspring of mothers with preeclampsia.
Topics: Adolescent; Age Factors; Carbon Monoxide; Child; Echocardiography, Doppler; Female; Humans; Hyperten | 2010 |
Ambient carbon monoxide and fine particulate matter in relation to preeclampsia and preterm delivery in western Washington State.
Topics: Adult; Air Pollutants; Carbon Monoxide; Cohort Studies; Female; Humans; Maternal Exposure; Particula | 2011 |
Comparing exposure assessment methods for traffic-related air pollution in an adverse pregnancy outcome study.
Topics: Adult; Air Pollutants; Air Pollution; California; Carbon Monoxide; Female; Humans; Maternal Exposure | 2011 |
Maternal exposure to moderate ambient carbon monoxide is associated with decreased risk of preeclampsia.
Topics: Adult; Air Pollutants; Air Pollution; Birth Weight; Carbon Monoxide; Cohort Studies; Female; Fetal G | 2012 |
Discussion: 'Moderate ambient level of carbon monoxide and risk of preeclampsia' by Zhai et al.
Topics: Air Pollutants; Air Pollution; Carbon Monoxide; Female; Humans; Maternal Exposure; Pre-Eclampsia; Pr | 2012 |
Nitric oxide and carbon monoxide production and metabolism in preeclampsia.
Topics: Adult; Amidohydrolases; Arginine; Biomarkers; Blotting, Western; Carbon Monoxide; Case-Control Studi | 2013 |
End tidal carbon monoxide levels are lower in women with gestational hypertension and pre-eclampsia.
Topics: Adult; Blood Pressure; Breath Tests; Carbon Monoxide; Case-Control Studies; Female; Humans; Hyperten | 2004 |
Direct placental effects of cigarette smoke protect women from pre-eclampsia: the specific roles of carbon monoxide and antioxidant systems in the placenta.
Topics: Administration, Inhalation; Antioxidants; Carbon Monoxide; Female; Humans; Models, Biological; Nicot | 2005 |
Carbon monoxide inhibits hypoxia/reoxygenation-induced apoptosis and secondary necrosis in syncytiotrophoblast.
Topics: Apoptosis; Carbon Monoxide; Cell Differentiation; Cell Hypoxia; Cells, Cultured; Female; Humans; Nec | 2006 |
Negative regulation of soluble Flt-1 and soluble endoglin release by heme oxygenase-1.
Topics: Animals; Antigens, CD; Antioxidants; Carbon Monoxide; Cell Hypoxia; Cells, Cultured; Culture Media, | 2007 |
Effect of cigarette smoke on placental antioxidant enzyme expression.
Topics: Antioxidants; Carbon Dioxide; Carbon Monoxide; Catalase; Cell Line, Tumor; Choriocarcinoma; Female; | 2007 |
End-tidal carbon monoxide measurements in women with pregnancy-induced hypertension and preeclampsia.
Topics: Adult; Carbon Monoxide; Female; Humans; Hypertension; Pre-Eclampsia; Pregnancy; Pregnancy Complicati | 2000 |
The role of the placenta in pre-eclampsia--a workshop report.
Topics: Adult; Apoptosis; Carbon Monoxide; Female; Fetal Growth Retardation; Heme Oxygenase (Decyclizing); H | 2002 |
Heme oxygenase activity in placenta: direct dependence on oxygen availability.
Topics: Carbon Monoxide; Chorionic Villi; Female; Heme; Heme Oxygenase (Decyclizing); Heme Oxygenase-1; Huma | 2002 |