carbon monoxide has been researched along with Homocystinuria in 2 studies
Carbon Monoxide: Carbon monoxide (CO). A poisonous colorless, odorless, tasteless gas. It combines with hemoglobin to form carboxyhemoglobin, which has no oxygen carrying capacity. The resultant oxygen deprivation causes headache, dizziness, decreased pulse and respiratory rates, unconsciousness, and death. (From Merck Index, 11th ed)
carbon monoxide : A one-carbon compound in which the carbon is joined only to a single oxygen. It is a colourless, odourless, tasteless, toxic gas.
Homocystinuria: Autosomal recessive inborn error of methionine metabolism usually caused by a deficiency of CYSTATHIONINE BETA-SYNTHASE and associated with elevations of homocysteine in plasma and urine. Clinical features include a tall slender habitus, SCOLIOSIS, arachnodactyly, MUSCLE WEAKNESS, genu varus, thin blond hair, malar flush, lens dislocations, an increased incidence of MENTAL RETARDATION, and a tendency to develop fibrosis of arteries, frequently complicated by CEREBROVASCULAR ACCIDENTS and MYOCARDIAL INFARCTION. (From Adams et al., Principles of Neurology, 6th ed, p979)
| Excerpt | Relevance | Reference |
|---|---|---|
| " This theory is indirectly supported by research in the area of environmental toxicology, which has revealed that carbon monoxide and carbon disulfide, agents whose prolonged exposure is known to result in the development of arteriosclerotic changes, induced vitamin B6 deficiency states which predictably lead to a homocystinuria-like state." | 7.67 | Environmental validation of the homocystine theory of arteriosclerosis. ( Calabrese, EJ, 1984) |
| " This theory is indirectly supported by research in the area of environmental toxicology, which has revealed that carbon monoxide and carbon disulfide, agents whose prolonged exposure is known to result in the development of arteriosclerotic changes, induced vitamin B6 deficiency states which predictably lead to a homocystinuria-like state." | 3.67 | Environmental validation of the homocystine theory of arteriosclerosis. ( Calabrese, EJ, 1984) |
| Timeframe | Studies, this research(%) | All Research% |
|---|---|---|
| pre-1990 | 1 (50.00) | 18.7374 |
| 1990's | 0 (0.00) | 18.2507 |
| 2000's | 0 (0.00) | 29.6817 |
| 2010's | 1 (50.00) | 24.3611 |
| 2020's | 0 (0.00) | 2.80 |
| Authors | Studies |
|---|---|
| Kabe, Y | 1 |
| Yamamoto, T | 1 |
| Kajimura, M | 1 |
| Sugiura, Y | 1 |
| Koike, I | 1 |
| Ohmura, M | 1 |
| Nakamura, T | 1 |
| Tokumoto, Y | 1 |
| Tsugawa, H | 1 |
| Handa, H | 1 |
| Kobayashi, T | 1 |
| Suematsu, M | 1 |
| Calabrese, EJ | 1 |
1 review available for carbon monoxide and Homocystinuria
| Article | Year |
|---|---|
Cystathionine β-synthase and PGRMC1 as CO sensors.
Topics: Animals; Carbon Monoxide; Cystathionine beta-Synthase; Cytochrome P-450 Enzyme System; ErbB Receptor | 2016 |
1 other study available for carbon monoxide and Homocystinuria
| Article | Year |
|---|---|
Environmental validation of the homocystine theory of arteriosclerosis.
Topics: Adult; Air Pollutants; Animals; Arteriosclerosis; Carbon Disulfide; Carbon Monoxide; Child; Heterozy | 1984 |