carbon monoxide has been researched along with Hemorrhagic Shock in 37 studies
Carbon Monoxide: Carbon monoxide (CO). A poisonous colorless, odorless, tasteless gas. It combines with hemoglobin to form carboxyhemoglobin, which has no oxygen carrying capacity. The resultant oxygen deprivation causes headache, dizziness, decreased pulse and respiratory rates, unconsciousness, and death. (From Merck Index, 11th ed)
carbon monoxide : A one-carbon compound in which the carbon is joined only to a single oxygen. It is a colourless, odourless, tasteless, toxic gas.
| Excerpt | Relevance | Reference |
|---|---|---|
| "This study aimed to assess whether pretreatment with carbon monoxide-releasing molecule-2 (CORM-2) could ameliorate inflammation by regulating differentiation of CD4 + T cells in intestinal mucosa of rats undergoing hemorrhagic shock." | 8.12 | Carbon monoxide-releasing molecule 2 inhibits inflammation associated with intestinal ischemia-reperfusion injury in a rat model of hemorrhagic shock. ( Du, F; Niu, Q; Wang, X; Yang, X, 2022) |
| "Carbon monoxide (CO) releasing molecule (CORM)-3, a water-soluble CORM, has protective effects against inflammatory and ischemia/reperfusion injury." | 7.91 | Carbon monoxide-releasing molecule-3 protects against cortical pyroptosis induced by hemorrhagic shock and resuscitation via mitochondrial regulation. ( Fu, L; Kong, XJ; Li, CC; Li, Y; Qi, MM; Song, PP; Wang, XD; Wang, XP; Zhang, DX; Zhang, LM, 2019) |
| "Carbon monoxide-releasing molecule protected against hemorrhagic shock/resuscitation organ injury and systemic inflammation and reduced hepatic sinusoidal endothelial injury." | 7.81 | Carbon monoxide protects against hemorrhagic shock and resuscitation-induced microcirculatory injury and tissue injury. ( Brumfield, J; Escobar, D; Gallo, D; Gomez, H; Kautza, B; Loughran, P; Luciano, J; Nassour, I; Otterbein, LE; Rubin, M; Scott, J; Zuckerbraun, BS, 2015) |
| " Both nitrite and carbon monoxide (CO) may protect from this reperfusion injury by limiting mitochondrial free radial production." | 7.81 | Effects of inhalation of low-dose nitrite or carbon monoxide on post-reperfusion mitochondrial function and tissue injury in hemorrhagic shock swine. ( Airgood, H; Dezfulian, C; Escobar, D; Gómez, H; Haugaa, H; Holder, A; Kenny, E; Maberry, DR; Ogundele, O; Pinsky, MR; Quintero, AM; Shiva, S; Tønnessen, TI; Zuckerbraun, B, 2015) |
| " Carbon monoxide (CO), when bound to red blood cells (CO-RBC) has the potential to protect the hepatic CYP protein to produce a resuscitative effect in a hemorrhagic shock rat model." | 7.81 | Kupffer cell inactivation by carbon monoxide bound to red blood cells preserves hepatic cytochrome P450 via anti-oxidant and anti-inflammatory effects exerted through the HMGB1/TLR-4 pathway during resuscitation from hemorrhagic shock. ( Ishima, Y; Maeda, H; Maruyama, T; Ogaki, S; Otagiri, M; Taguchi, K; Watanabe, H, 2015) |
| "The response to exchange transfusion with red blood cells (RBCs) saturated with carbon monoxide (CO) in amelioration of microvascular function and providing tissue protection in hemorrhagic shock resuscitation was investigated in the hamster chamber window model." | 7.74 | Hemorrhagic shock resuscitation with carbon monoxide saturated blood. ( Cabrales, P; Intaglietta, M; Tsai, AG, 2007) |
| " Thus, we determined the differential role of carbon monoxide (CO) and nitric oxide (NO) in the intrinsic control of sinusoidal perfusion, mitochondrial redox state, and bile production in the isolated perfused rat liver after hemorrhagic shock." | 7.70 | Protective role of endogenous carbon monoxide in hepatic microcirculatory dysfunction after hemorrhagic shock in rats. ( Bauer, M; Clemens, MG; Geiger, KK; Hole, B; Köhler, N; Pannen, BH, 1998) |
| "Hemorrhagic shock was induced in adult male Sprague‑Dawley rats under sevoflurane anesthesia by bleeding using a heparinized syringe to maintain a mean arterial pressure of 30±5 mmHg for 60 min." | 5.56 | Exogenous carbon monoxide protects against mitochondrial DNA‑induced hippocampal pyroptosis in a model of hemorrhagic shock and resuscitation. ( Fu, L; Gui, CX; Kang, LQ; Kong, XJ; Li, Y; Liu, FH; Song, YC; Wang, XD; Wang, XP; Zhang, DX; Zhang, LM; Zheng, WC, 2020) |
| "To investigate this, we created a hemorrhagic shock model rat, followed by resuscitation with RBC and CO-RBC." | 5.40 | Carbon monoxide-bound red blood cell resuscitation ameliorates hepatic injury induced by massive hemorrhage and red blood cell resuscitation via hepatic cytochrome P450 protection in hemorrhagic shock rats. ( Ishima, Y; Maruyama, T; Ogaki, S; Otagiri, M; Taguchi, K; Watanabe, H, 2014) |
| "Even after successful resuscitation, hemorrhagic shock frequently causes pulmonary inflammation that induces acute lung injury (ALI)." | 5.39 | Inhalation of carbon monoxide following resuscitation ameliorates hemorrhagic shock-induced lung injury. ( Kawanishi, S; Maeda, S; Matsumi, M; Morimatsu, H; Morita, K; Nakao, A; Omori, E; Sato, K; Shimizu, H; Takahashi, T, 2013) |
| "CO protects against systemic effects of hemorrhagic shock and resuscitation." | 5.33 | Carbon monoxide prevents multiple organ injury in a model of hemorrhagic shock and resuscitation. ( Billiar, TR; Gallo, D; Ifedigbo, E; Liu, F; McCloskey, CA; Otterbein, LE; Zuckerbraun, BS, 2005) |
| "This study aimed to assess whether pretreatment with carbon monoxide-releasing molecule-2 (CORM-2) could ameliorate inflammation by regulating differentiation of CD4 + T cells in intestinal mucosa of rats undergoing hemorrhagic shock." | 4.12 | Carbon monoxide-releasing molecule 2 inhibits inflammation associated with intestinal ischemia-reperfusion injury in a rat model of hemorrhagic shock. ( Du, F; Niu, Q; Wang, X; Yang, X, 2022) |
| "Carbon monoxide (CO) releasing molecule (CORM)-3, a water-soluble CORM, has protective effects against inflammatory and ischemia/reperfusion injury." | 3.91 | Carbon monoxide-releasing molecule-3 protects against cortical pyroptosis induced by hemorrhagic shock and resuscitation via mitochondrial regulation. ( Fu, L; Kong, XJ; Li, CC; Li, Y; Qi, MM; Song, PP; Wang, XD; Wang, XP; Zhang, DX; Zhang, LM, 2019) |
| "Carbon monoxide-releasing molecule protected against hemorrhagic shock/resuscitation organ injury and systemic inflammation and reduced hepatic sinusoidal endothelial injury." | 3.81 | Carbon monoxide protects against hemorrhagic shock and resuscitation-induced microcirculatory injury and tissue injury. ( Brumfield, J; Escobar, D; Gallo, D; Gomez, H; Kautza, B; Loughran, P; Luciano, J; Nassour, I; Otterbein, LE; Rubin, M; Scott, J; Zuckerbraun, BS, 2015) |
| " Both nitrite and carbon monoxide (CO) may protect from this reperfusion injury by limiting mitochondrial free radial production." | 3.81 | Effects of inhalation of low-dose nitrite or carbon monoxide on post-reperfusion mitochondrial function and tissue injury in hemorrhagic shock swine. ( Airgood, H; Dezfulian, C; Escobar, D; Gómez, H; Haugaa, H; Holder, A; Kenny, E; Maberry, DR; Ogundele, O; Pinsky, MR; Quintero, AM; Shiva, S; Tønnessen, TI; Zuckerbraun, B, 2015) |
| " Carbon monoxide (CO), when bound to red blood cells (CO-RBC) has the potential to protect the hepatic CYP protein to produce a resuscitative effect in a hemorrhagic shock rat model." | 3.81 | Kupffer cell inactivation by carbon monoxide bound to red blood cells preserves hepatic cytochrome P450 via anti-oxidant and anti-inflammatory effects exerted through the HMGB1/TLR-4 pathway during resuscitation from hemorrhagic shock. ( Ishima, Y; Maeda, H; Maruyama, T; Ogaki, S; Otagiri, M; Taguchi, K; Watanabe, H, 2015) |
| " Thus, we determined the differential role of carbon monoxide (CO) and nitric oxide (NO) in the intrinsic control of sinusoidal perfusion, mitochondrial redox state, and bile production in the isolated perfused rat liver after hemorrhagic shock." | 3.70 | Protective role of endogenous carbon monoxide in hepatic microcirculatory dysfunction after hemorrhagic shock in rats. ( Bauer, M; Clemens, MG; Geiger, KK; Hole, B; Köhler, N; Pannen, BH, 1998) |
| "To investigate the role of the vasodilator systems heme oxygenase-1/heat shock protein 32 (HO-1/HSP32) and nitric oxide synthase-II (NOS-II), generating carbon monoxide and nitric oxide respectively, as modulators of liver injury in an experimental model of reversible hemorrhagic shock." | 3.70 | Differential expression pattern of heme oxygenase-1/heat shock protein 32 and nitric oxide synthase-II and their impact on liver injury in a rat model of hemorrhage and resuscitation. ( Bauer, I; Bauer, M; Datene, V; Pannen, BH; Pätau, C; Rensing, H, 1999) |
| "In a porcine model of hemorrhagic shock, the administration of rhIL-11 at the start of resuscitation significantly improved the cardiac output and blood pressure." | 2.71 | Administration of recombinant interleukin-11 improves the hemodynamic functions and decreases third space fluid loss in a porcine model of hemorrhagic shock and resuscitation. ( Alam, HB; Honma, K; Keith, JC; Koles, NL; Olsen, C; Pollack, M; Rhee, P; Rollwagen, FM, 2005) |
| " Although the effectiveness of HbVs, including their physicochemical characteristics and pharmacological effects, has been reported, data on the pharmacokinetic properties of HbVs are limited." | 2.58 | [Safety Evaluation of Cellular-type Artificial Blood Based on Pharmacokinetic Analysis and Its Use in Medical Gas Delivery]. ( Taguchi, K, 2018) |
| "Carbon monoxide (CO) is a toxic gas that causes neuropathy." | 1.72 | Effect of carbon monoxide administration using haemoglobin-vesicles on the hippocampal tissue. ( Okuda, C; Sakai, H, 2022) |
| "Hemorrhagic shock was induced in adult male Sprague‑Dawley rats under sevoflurane anesthesia by bleeding using a heparinized syringe to maintain a mean arterial pressure of 30±5 mmHg for 60 min." | 1.56 | Exogenous carbon monoxide protects against mitochondrial DNA‑induced hippocampal pyroptosis in a model of hemorrhagic shock and resuscitation. ( Fu, L; Gui, CX; Kang, LQ; Kong, XJ; Li, Y; Liu, FH; Song, YC; Wang, XD; Wang, XP; Zhang, DX; Zhang, LM; Zheng, WC, 2020) |
| "To investigate this, we created a hemorrhagic shock model rat, followed by resuscitation with RBC and CO-RBC." | 1.40 | Carbon monoxide-bound red blood cell resuscitation ameliorates hepatic injury induced by massive hemorrhage and red blood cell resuscitation via hepatic cytochrome P450 protection in hemorrhagic shock rats. ( Ishima, Y; Maruyama, T; Ogaki, S; Otagiri, M; Taguchi, K; Watanabe, H, 2014) |
| "Even after successful resuscitation, hemorrhagic shock frequently causes pulmonary inflammation that induces acute lung injury (ALI)." | 1.39 | Inhalation of carbon monoxide following resuscitation ameliorates hemorrhagic shock-induced lung injury. ( Kawanishi, S; Maeda, S; Matsumi, M; Morimatsu, H; Morita, K; Nakao, A; Omori, E; Sato, K; Shimizu, H; Takahashi, T, 2013) |
| "CO protects against systemic effects of hemorrhagic shock and resuscitation." | 1.33 | Carbon monoxide prevents multiple organ injury in a model of hemorrhagic shock and resuscitation. ( Billiar, TR; Gallo, D; Ifedigbo, E; Liu, F; McCloskey, CA; Otterbein, LE; Zuckerbraun, BS, 2005) |
| "It is well known that hemorrhagic shock induces inflammatory changes." | 1.31 | Hemorrhage and resuscitation induce delayed inflammation and pulmonary dysfunction in mice. ( Claridge, JA; Enelow, RI; Young, JS, 2000) |
| Timeframe | Studies, this research(%) | All Research% |
|---|---|---|
| pre-1990 | 8 (21.62) | 18.7374 |
| 1990's | 4 (10.81) | 18.2507 |
| 2000's | 9 (24.32) | 29.6817 |
| 2010's | 12 (32.43) | 24.3611 |
| 2020's | 4 (10.81) | 2.80 |
| Authors | Studies |
|---|---|
| Okuda, C | 1 |
| Sakai, H | 3 |
| Niu, Q | 3 |
| Du, F | 3 |
| Yang, X | 6 |
| Wang, X | 3 |
| Zhang, LM | 3 |
| Xin, Y | 1 |
| Song, RX | 1 |
| Zheng, WC | 2 |
| Hu, JS | 1 |
| Wang, JX | 1 |
| Wu, ZY | 1 |
| Zhang, DX | 3 |
| Fu, L | 2 |
| Song, YC | 1 |
| Liu, FH | 1 |
| Li, Y | 2 |
| Wang, XP | 2 |
| Wang, XD | 2 |
| Gui, CX | 1 |
| Kong, XJ | 2 |
| Kang, LQ | 1 |
| Taguchi, K | 4 |
| Qi, MM | 1 |
| Li, CC | 1 |
| Song, PP | 1 |
| Ogaki, S | 3 |
| Watanabe, H | 3 |
| Ishima, Y | 2 |
| Otagiri, M | 3 |
| Maruyama, T | 3 |
| Nassour, I | 2 |
| Kautza, B | 2 |
| Rubin, M | 1 |
| Escobar, D | 3 |
| Luciano, J | 2 |
| Loughran, P | 2 |
| Gomez, H | 3 |
| Scott, J | 1 |
| Gallo, D | 2 |
| Brumfield, J | 1 |
| Otterbein, LE | 2 |
| Zuckerbraun, BS | 3 |
| Haugaa, H | 1 |
| Maberry, DR | 1 |
| Holder, A | 2 |
| Ogundele, O | 2 |
| Quintero, AM | 1 |
| Tønnessen, TI | 1 |
| Airgood, H | 1 |
| Dezfulian, C | 1 |
| Kenny, E | 1 |
| Shiva, S | 2 |
| Zuckerbraun, B | 1 |
| Pinsky, MR | 2 |
| Maeda, H | 1 |
| Duan, C | 1 |
| Yang, G | 2 |
| Li, T | 2 |
| Liu, L | 2 |
| Botero, AM | 1 |
| Gordon, L | 1 |
| Martinez, S | 1 |
| Rosengart, MR | 1 |
| Pinsky, M | 1 |
| Horinouchi, H | 1 |
| Tsuchida, E | 1 |
| Kobayashi, K | 2 |
| Kanagawa, F | 1 |
| Takahashi, T | 2 |
| Inoue, K | 1 |
| Shimizu, H | 2 |
| Omori, E | 2 |
| Morimatsu, H | 2 |
| Maeda, S | 2 |
| Katayama, H | 1 |
| Nakao, A | 2 |
| Morita, K | 2 |
| Takeoka, S | 1 |
| Kawanishi, S | 1 |
| Sato, K | 1 |
| Matsumi, M | 1 |
| Siegel, JH | 1 |
| Fabian, M | 1 |
| Smith, JA | 1 |
| Kingston, EP | 1 |
| Steele, KA | 1 |
| Wells, MR | 1 |
| Kaplan, LJ | 1 |
| CAHILL, JM | 2 |
| JOUASSET-STRIEDER, D | 2 |
| BYRNE, JJ | 2 |
| McCloskey, CA | 1 |
| Liu, F | 1 |
| Ifedigbo, E | 1 |
| Billiar, TR | 2 |
| Honma, K | 1 |
| Koles, NL | 1 |
| Alam, HB | 1 |
| Rhee, P | 1 |
| Rollwagen, FM | 1 |
| Olsen, C | 1 |
| Keith, JC | 1 |
| Pollack, M | 1 |
| Clavijo-Alvarez, JA | 1 |
| Sims, CA | 1 |
| Puyana, JC | 1 |
| Cabrales, P | 1 |
| Tsai, AG | 1 |
| Intaglietta, M | 1 |
| Zhang, Y | 2 |
| Liu, LM | 1 |
| Ming, J | 1 |
| Xu, J | 1 |
| Chen, W | 1 |
| Sheldon, CA | 1 |
| Cerra, FB | 1 |
| Bohnhoff, N | 1 |
| Belani, K | 1 |
| Frieswyk, D | 1 |
| Dhanalal, K | 1 |
| Leonard, AS | 1 |
| Bauer, M | 3 |
| Rensing, H | 2 |
| Bauer, C | 1 |
| Bauer, I | 2 |
| Larsen, R | 1 |
| Pannen, BH | 2 |
| Köhler, N | 1 |
| Hole, B | 1 |
| Clemens, MG | 1 |
| Geiger, KK | 1 |
| Datene, V | 1 |
| Pätau, C | 1 |
| Claridge, JA | 1 |
| Enelow, RI | 1 |
| Young, JS | 1 |
| Malmberg, PO | 1 |
| Hlastala, MP | 1 |
| Woodson, RD | 1 |
| Kallos, T | 1 |
| Wyche, MQ | 1 |
| Marshall, BE | 1 |
| Coburn, RF | 2 |
| Ploegmakers, F | 1 |
| Gondrie, P | 1 |
| Abboud, R | 2 |
| Abel, FL | 1 |
| Waldhausen, JA | 1 |
| Daly, WJ | 1 |
| Pearce, WL | 1 |
| Wallace, HW | 1 |
3 reviews available for carbon monoxide and Hemorrhagic Shock
| Article | Year |
|---|---|
[Safety Evaluation of Cellular-type Artificial Blood Based on Pharmacokinetic Analysis and Its Use in Medical Gas Delivery].
Topics: Acute Disease; Animals; Blood Substitutes; Carbon Monoxide; Disease Models, Animal; Drug Carriers; D | 2018 |
Advances in Vascular Hyporeactivity After Shock: The Mechanisms and Managements.
Topics: Acidosis; Adult; Age Factors; Aged; Analgesics, Opioid; Blood Vessels; Burns; Calcium; Carbon Monoxi | 2015 |
Gas bioengineering using hemoglobin-vesicles for versatile clinical applications.
Topics: Animals; Blood Substitutes; Carbon Monoxide; Clinical Medicine; Hemoglobins; Humans; Models, Biologi | 2011 |
1 trial available for carbon monoxide and Hemorrhagic Shock
| Article | Year |
|---|---|
Administration of recombinant interleukin-11 improves the hemodynamic functions and decreases third space fluid loss in a porcine model of hemorrhagic shock and resuscitation.
Topics: Animals; Blood Pressure; Carbon Monoxide; Cardiac Output; Disease Models, Animal; Hemodynamics; Inte | 2005 |
33 other studies available for carbon monoxide and Hemorrhagic Shock
| Article | Year |
|---|---|
Effect of carbon monoxide administration using haemoglobin-vesicles on the hippocampal tissue.
Topics: Animals; Carbon Monoxide; Carbon Monoxide Poisoning; Hemoglobins; Hippocampus; Rats; Shock, Hemorrha | 2022 |
Carbon monoxide-releasing molecule 2 inhibits inflammation associated with intestinal ischemia-reperfusion injury in a rat model of hemorrhagic shock.
Topics: Animals; Carbon Monoxide; Cytokines; Inflammation; Interferon-gamma; Interleukin-17; Rats; Reperfusi | 2022 |
Carbon monoxide-releasing molecule 2 inhibits inflammation associated with intestinal ischemia-reperfusion injury in a rat model of hemorrhagic shock.
Topics: Animals; Carbon Monoxide; Cytokines; Inflammation; Interferon-gamma; Interleukin-17; Rats; Reperfusi | 2022 |
Carbon monoxide-releasing molecule 2 inhibits inflammation associated with intestinal ischemia-reperfusion injury in a rat model of hemorrhagic shock.
Topics: Animals; Carbon Monoxide; Cytokines; Inflammation; Interferon-gamma; Interleukin-17; Rats; Reperfusi | 2022 |
Carbon monoxide-releasing molecule 2 inhibits inflammation associated with intestinal ischemia-reperfusion injury in a rat model of hemorrhagic shock.
Topics: Animals; Carbon Monoxide; Cytokines; Inflammation; Interferon-gamma; Interleukin-17; Rats; Reperfusi | 2022 |
Carbon monoxide-releasing molecule 2 inhibits inflammation associated with intestinal ischemia-reperfusion injury in a rat model of hemorrhagic shock.
Topics: Animals; Carbon Monoxide; Cytokines; Inflammation; Interferon-gamma; Interleukin-17; Rats; Reperfusi | 2022 |
Carbon monoxide-releasing molecule 2 inhibits inflammation associated with intestinal ischemia-reperfusion injury in a rat model of hemorrhagic shock.
Topics: Animals; Carbon Monoxide; Cytokines; Inflammation; Interferon-gamma; Interleukin-17; Rats; Reperfusi | 2022 |
Carbon monoxide-releasing molecule 2 inhibits inflammation associated with intestinal ischemia-reperfusion injury in a rat model of hemorrhagic shock.
Topics: Animals; Carbon Monoxide; Cytokines; Inflammation; Interferon-gamma; Interleukin-17; Rats; Reperfusi | 2022 |
Carbon monoxide-releasing molecule 2 inhibits inflammation associated with intestinal ischemia-reperfusion injury in a rat model of hemorrhagic shock.
Topics: Animals; Carbon Monoxide; Cytokines; Inflammation; Interferon-gamma; Interleukin-17; Rats; Reperfusi | 2022 |
Carbon monoxide-releasing molecule 2 inhibits inflammation associated with intestinal ischemia-reperfusion injury in a rat model of hemorrhagic shock.
Topics: Animals; Carbon Monoxide; Cytokines; Inflammation; Interferon-gamma; Interleukin-17; Rats; Reperfusi | 2022 |
CORM-3 alleviates the intestinal injury in a rodent model of hemorrhage shock and resuscitation: roles of GFAP-positive glia.
Topics: Animals; Carbon Monoxide; Claudin-1; Glial Fibrillary Acidic Protein; Neuroglia; NLR Family, Pyrin D | 2023 |
Exogenous carbon monoxide protects against mitochondrial DNA‑induced hippocampal pyroptosis in a model of hemorrhagic shock and resuscitation.
Topics: Animals; Carbon Monoxide; Disease Models, Animal; DNA, Mitochondrial; Hippocampus; Male; Pyroptosis; | 2020 |
Carbon monoxide-releasing molecule-3 protects against cortical pyroptosis induced by hemorrhagic shock and resuscitation via mitochondrial regulation.
Topics: Animals; Carbon Monoxide; Cyclic GMP; Disease Models, Animal; Humans; Mitochondria; Neurons; Neuropr | 2019 |
Carbon monoxide-bound red blood cell resuscitation ameliorates hepatic injury induced by massive hemorrhage and red blood cell resuscitation via hepatic cytochrome P450 protection in hemorrhagic shock rats.
Topics: Animals; Carbon Monoxide; Cytochrome P-450 Enzyme System; Dapsone; Disease Models, Animal; Erythrocy | 2014 |
Carbon monoxide protects against hemorrhagic shock and resuscitation-induced microcirculatory injury and tissue injury.
Topics: Animals; Carbon Monoxide; Cell Adhesion; Cell Adhesion Molecules; Cells, Cultured; Cytokines; Drug E | 2015 |
Effects of inhalation of low-dose nitrite or carbon monoxide on post-reperfusion mitochondrial function and tissue injury in hemorrhagic shock swine.
Topics: Administration, Inhalation; Animals; Carbon Monoxide; Microdialysis; Mitochondria; Nitrites; Oxygen | 2015 |
Kupffer cell inactivation by carbon monoxide bound to red blood cells preserves hepatic cytochrome P450 via anti-oxidant and anti-inflammatory effects exerted through the HMGB1/TLR-4 pathway during resuscitation from hemorrhagic shock.
Topics: Animals; Antioxidants; Carbon Monoxide; Chromans; Cytochrome P-450 Enzyme System; Cytokines; Erythro | 2015 |
Inhaled Carbon Monoxide Protects against the Development of Shock and Mitochondrial Injury following Hemorrhage and Resuscitation.
Topics: Adenosine Triphosphate; Administration, Inhalation; Animals; Carbon Monoxide; Cells, Cultured; Inter | 2015 |
Hemoglobin vesicles and red blood cells as carriers of carbon monoxide prior to oxygen for resuscitation after hemorrhagic shock in a rat model.
Topics: Animals; Carbon Monoxide; Disease Models, Animal; Erythrocytes; Hemoglobins; Liver; Lung; Male; Rats | 2009 |
Protective effect of carbon monoxide inhalation on lung injury after hemorrhagic shock/resuscitation in rats.
Topics: Acute Lung Injury; Administration, Inhalation; Animals; Carbon Monoxide; Carboxyhemoglobin; Disease | 2010 |
Carbon monoxide-bound red blood cells protect red blood cell transfusion-induced hepatic cytochrome P450 impairment in hemorrhagic-shock rats.
Topics: Animals; Blotting, Western; Carbon Monoxide; Cytochrome P-450 Enzyme System; Erythrocyte Transfusion | 2013 |
Inhalation of carbon monoxide following resuscitation ameliorates hemorrhagic shock-induced lung injury.
Topics: Acute Lung Injury; Animals; Apoptosis; Carbon Monoxide; Carboxyhemoglobin; Disease Models, Animal; G | 2013 |
Oxygen debt criteria quantify the effectiveness of early partial resuscitation after hypovolemic hemorrhagic shock.
Topics: Albumins; Animals; Blood Volume; Carbon Monoxide; Disease Models, Animal; Dogs; Fluid Therapy; Kidne | 2003 |
LUNG FUNCTION IN SHOCK.
Topics: Anemia; Animals; Blood Pressure; Blood Volume; Capillaries; Carbon Monoxide; Dogs; Endotoxins; Eryth | 1965 |
Carbon monoxide prevents multiple organ injury in a model of hemorrhagic shock and resuscitation.
Topics: Adenosine Triphosphate; Air; Animals; Carbon Monoxide; Cell Death; Cell Survival; Cytokines; Disease | 2005 |
Monitoring skeletal muscle and subcutaneous tissue acid-base status and oxygenation during hemorrhagic shock and resuscitation.
Topics: Animals; Blood Pressure; Carbon Dioxide; Carbon Monoxide; Carotid Arteries; Coloring Agents; Evans B | 2005 |
Hemorrhagic shock resuscitation with carbon monoxide saturated blood.
Topics: Animals; Blood Transfusion; Carbon Monoxide; Cricetinae; Infusions, Intravenous; Mesocricetus; Resus | 2007 |
[The changes in the HIF-1alpha expression during hypovolemic shock and its role in the pathogenesis of vascular hyporeactivity].
Topics: Animals; Blood Vessels; Carbon Monoxide; Heme Oxygenase-1; Hypoxia-Inducible Factor 1, alpha Subunit | 2006 |
regulatory effects of hypoxia-inducible factor 1alpha on vascular reactivity and its mechanisms following hemorrhagic shock in rats.
Topics: Animals; Carbon Monoxide; Cyclooxygenase 2; Heme Oxygenase-1; Hypoxia-Inducible Factor 1, alpha Subu | 2008 |
Peripheral postcapillary venous pressure: a new, more sensitive monitor of effective blood volume during hemorrhagic shock and resuscitation.
Topics: Animals; Blood Circulation; Blood Volume Determination; Carbon Monoxide; Cardiac Output; Central Ven | 1983 |
[Organ specific expression pattern of a carbon monoxide generating stress protein (hemoxygenase-1/heatshock protein 32) following hemorrhagic shock].
Topics: Animals; Blood Pressure; Blotting, Western; Carbon Monoxide; Heme Oxygenase (Decyclizing); Heme Oxyg | 1997 |
Protective role of endogenous carbon monoxide in hepatic microcirculatory dysfunction after hemorrhagic shock in rats.
Topics: Animals; Bile; Carbon Monoxide; Heme Oxygenase (Decyclizing); Hemodynamics; In Vitro Techniques; Isc | 1998 |
Differential expression pattern of heme oxygenase-1/heat shock protein 32 and nitric oxide synthase-II and their impact on liver injury in a rat model of hemorrhage and resuscitation.
Topics: Analysis of Variance; Animals; Carbon Monoxide; Gene Expression Regulation, Enzymologic; Glutathione | 1999 |
The diverging roles of carbon monoxide and nitric oxide in resuscitated hemorrhagic shock.
Topics: Carbon Monoxide; Humans; Liver; Nitric Oxide; Nitric Oxide Synthase; Resuscitation; Shock, Hemorrhag | 1999 |
Hemorrhage and resuscitation induce delayed inflammation and pulmonary dysfunction in mice.
Topics: Animals; Blood Pressure; Carbon Monoxide; Female; Inflammation; Lung; Mice; Peak Expiratory Flow Rat | 2000 |
Effect of increased blood-oxygen affinity on oxygen transport in hemorrhagic shock.
Topics: Animals; Carbon Monoxide; Cardiac Output; Exchange Transfusion, Whole Blood; Hydrogen-Ion Concentrat | 1979 |
Effects of hemorrhagic shock on pulmonary diffusion and capillary blood volume of the dog.
Topics: Animals; Blood Pressure; Carbon Monoxide; Cardiac Output; Dogs; Erythrocytes; Microcirculation; Neon | 1973 |
Myocardial myoglobin oxygen tension.
Topics: Animals; Biopsy; Carbon Dioxide; Carbon Isotopes; Carbon Monoxide; Carboxyhemoglobin; Dogs; Hemoglob | 1973 |
Pulmonary blood volume in hemorrhagic shock in the dog and primate.
Topics: Animals; Blood Pressure; Blood Volume; Carbon Monoxide; Cardiac Output; Dogs; Haplorhini; Heart Rate | 1967 |
Redistribution of body carbon monoxide after hemorrhage.
Topics: Animals; Blood Pressure; Blood Volume; Carbon Isotopes; Carbon Monoxide; Chromium Isotopes; Dogs; He | 1971 |
Pulmonary capillary blood volume in dogs during shock and after retransfusion.
Topics: Animals; Blood Transfusion; Blood Volume; Capillaries; Carbon Monoxide; Dogs; Hemodynamics; Pulmonar | 1966 |