carbon monoxide has been researched along with Gastric Ulcer in 6 studies
Carbon Monoxide: Carbon monoxide (CO). A poisonous colorless, odorless, tasteless gas. It combines with hemoglobin to form carboxyhemoglobin, which has no oxygen carrying capacity. The resultant oxygen deprivation causes headache, dizziness, decreased pulse and respiratory rates, unconsciousness, and death. (From Merck Index, 11th ed)
carbon monoxide : A one-carbon compound in which the carbon is joined only to a single oxygen. It is a colourless, odourless, tasteless, toxic gas.
| Excerpt | Relevance | Reference |
|---|---|---|
| "Carbon monoxide (CO), a gaseous mediator produced by haem oxygenases (HOs), has been shown to prevent stress-, ethanol-, aspirin- and alendronate-induced gastric damage; however, its role in gastric ulcer healing has not been fully elucidated." | 7.85 | Carbon monoxide released from its pharmacological donor, tricarbonyldichlororuthenium (II) dimer, accelerates the healing of pre-existing gastric ulcers. ( Brzozowski, T; Chmura, A; Ginter, G; Hubalewska-Mazgaj, M; Kwiecien, S; Magierowska, K; Magierowski, M; Pajdo, R; Sliwowski, Z, 2017) |
| "The prevalence of gastric ulcers is increasing worldwide, especially those brought on by non-steroidal anti-inflammatory drugs (NSAIDS), so prevention is extremely crucial." | 5.91 | Potential effects of carbon monoxide donor and its nanoparticles on experimentally induced gastric ulcer in rats. ( Abu-Risha, SE; Elsisi, AE; Mekky, EF, 2023) |
| "Carbon monoxide (CO), a gaseous mediator produced by haem oxygenases (HOs), has been shown to prevent stress-, ethanol-, aspirin- and alendronate-induced gastric damage; however, its role in gastric ulcer healing has not been fully elucidated." | 3.85 | Carbon monoxide released from its pharmacological donor, tricarbonyldichlororuthenium (II) dimer, accelerates the healing of pre-existing gastric ulcers. ( Brzozowski, T; Chmura, A; Ginter, G; Hubalewska-Mazgaj, M; Kwiecien, S; Magierowska, K; Magierowski, M; Pajdo, R; Sliwowski, Z, 2017) |
| "The prevalence of gastric ulcers is increasing worldwide, especially those brought on by non-steroidal anti-inflammatory drugs (NSAIDS), so prevention is extremely crucial." | 1.91 | Potential effects of carbon monoxide donor and its nanoparticles on experimentally induced gastric ulcer in rats. ( Abu-Risha, SE; Elsisi, AE; Mekky, EF, 2023) |
| "Acute gastric ulcers were induced in C57BL/6 male mice using acetic acid." | 1.43 | Carbon monoxide promotes gastric wound healing in mice via the protein kinase C pathway. ( Hirata, I; Horie, R; Mizuhima, K; Naito, Y; Suzuki, T; Takagi, T; Tsuboi, H; Uchiyama, K; Yoshikawa, T, 2016) |
| "Carbon monoxide (CO) has been found to be produced in every living cell in a biochemical reaction catalyzed by heme-oxygenase (HO) enzyme which degrades heme into biliverdin, CO, and iron." | 1.38 | Inhibition of endogenous CO by ZnPP protects against stress-induced gastric lesion in adult male albino rats. ( Abdel-Hakim, S; Aziz, N; El-Sayed, S; Hassan, M; Ibrahim, I, 2012) |
| Timeframe | Studies, this research(%) | All Research% |
|---|---|---|
| pre-1990 | 0 (0.00) | 18.7374 |
| 1990's | 0 (0.00) | 18.2507 |
| 2000's | 0 (0.00) | 29.6817 |
| 2010's | 5 (83.33) | 24.3611 |
| 2020's | 1 (16.67) | 2.80 |
| Authors | Studies |
|---|---|
| Elsisi, AE | 1 |
| Mekky, EF | 1 |
| Abu-Risha, SE | 1 |
| Magierowski, M | 2 |
| Magierowska, K | 2 |
| Hubalewska-Mazgaj, M | 2 |
| Sliwowski, Z | 2 |
| Ginter, G | 2 |
| Pajdo, R | 1 |
| Chmura, A | 2 |
| Kwiecien, S | 2 |
| Brzozowski, T | 2 |
| Bakalarz, D | 1 |
| Wójcik, D | 1 |
| Licholai, S | 1 |
| Korbut, E | 1 |
| Ibrahim, MY | 1 |
| El-Sayed, SA | 1 |
| Abdel-Hakim, SM | 1 |
| Hassan, MK | 1 |
| Aziz, NM | 1 |
| Takagi, T | 1 |
| Naito, Y | 1 |
| Uchiyama, K | 1 |
| Mizuhima, K | 1 |
| Suzuki, T | 1 |
| Horie, R | 1 |
| Hirata, I | 1 |
| Tsuboi, H | 1 |
| Yoshikawa, T | 1 |
| Ibrahim, I | 1 |
| El-Sayed, S | 1 |
| Abdel-Hakim, S | 1 |
| Hassan, M | 1 |
| Aziz, N | 1 |
6 other studies available for carbon monoxide and Gastric Ulcer
| Article | Year |
|---|---|
Potential effects of carbon monoxide donor and its nanoparticles on experimentally induced gastric ulcer in rats.
Topics: Animals; Carbon Monoxide; Cyclooxygenase 2; Gastric Mucosa; Indomethacin; NF-E2-Related Factor 2; Ra | 2023 |
Carbon monoxide released from its pharmacological donor, tricarbonyldichlororuthenium (II) dimer, accelerates the healing of pre-existing gastric ulcers.
Topics: Acetic Acid; Animals; Carbon Monoxide; Cyclooxygenase 2; Epidermal Growth Factor; ErbB Receptors; Ga | 2017 |
Time-dependent course of gastric ulcer healing and molecular markers profile modulated by increased gastric mucosal content of carbon monoxide released from its pharmacological donor.
Topics: Animals; Carbon Monoxide; Dose-Response Relationship, Drug; Drug Liberation; Gastric Mucosa; Interce | 2019 |
The effect of induction of endogenous CO by heme-oxygenase inducer, hemin versus heme-oxygenase blocker, zinc mesoporphyrin on gastric secretion and ulceration under different conditions in adult male albino rats.
Topics: Animals; Carbon Monoxide; Cold-Shock Response; Cyclooxygenase Inhibitors; Gastric Juice; Gastric Muc | 2014 |
Carbon monoxide promotes gastric wound healing in mice via the protein kinase C pathway.
Topics: Animals; Carbon Monoxide; Mice; Mice, Inbred C57BL; Protein Kinase C; Stomach Ulcer; Wound Healing | 2016 |
Inhibition of endogenous CO by ZnPP protects against stress-induced gastric lesion in adult male albino rats.
Topics: Animals; Carbon Monoxide; Carboxyhemoglobin; Heme Oxygenase-1; Male; Protective Agents; Protoporphyr | 2012 |