Page last updated: 2024-10-16

carbon monoxide and Cranial Nerve II Injuries

carbon monoxide has been researched along with Cranial Nerve II Injuries in 2 studies

Carbon Monoxide: Carbon monoxide (CO). A poisonous colorless, odorless, tasteless gas. It combines with hemoglobin to form carboxyhemoglobin, which has no oxygen carrying capacity. The resultant oxygen deprivation causes headache, dizziness, decreased pulse and respiratory rates, unconsciousness, and death. (From Merck Index, 11th ed)
carbon monoxide : A one-carbon compound in which the carbon is joined only to a single oxygen. It is a colourless, odourless, tasteless, toxic gas.

Research Excerpts

ExcerptRelevanceReference
"Carbon monoxide (CO) has been observed to elicit neuroprotection in various experimental models."1.48Preconditioning with carbon monoxide inhalation promotes retinal ganglion cell survival against optic nerve crush via inhibition of the apoptotic pathway. ( Chen, Z; Liu, L; Sun, A; Sun, Q; Wang, R; Wu, J; Xia, F, 2018)
"Carbon monoxide (CO) acts as a therapeutic agent against neural injury via its anti-apoptotic effect."1.43Low-dose carbon monoxide inhalation protects neuronal cells from apoptosis after optic nerve crush. ( Chen, Z; Liu, L; Sun, Q; Wang, R; Wu, J; Xia, F; Xu, J, 2016)

Research

Studies (2)

TimeframeStudies, this research(%)All Research%
pre-19900 (0.00)18.7374
1990's0 (0.00)18.2507
2000's0 (0.00)29.6817
2010's2 (100.00)24.3611
2020's0 (0.00)2.80

Authors

AuthorsStudies
Wang, R2
Chen, Z2
Wu, J2
Xia, F2
Sun, Q2
Sun, A1
Liu, L2
Xu, J1

Other Studies

2 other studies available for carbon monoxide and Cranial Nerve II Injuries

ArticleYear
Preconditioning with carbon monoxide inhalation promotes retinal ganglion cell survival against optic nerve crush via inhibition of the apoptotic pathway.
    Molecular medicine reports, 2018, Volume: 17, Issue:1

    Topics: Administration, Inhalation; Animals; Apoptosis; Carbon Monoxide; Cell Survival; Drug Evaluation, Pre

2018
Low-dose carbon monoxide inhalation protects neuronal cells from apoptosis after optic nerve crush.
    Biochemical and biophysical research communications, 2016, Jan-22, Volume: 469, Issue:4

    Topics: Administration, Inhalation; Animals; Apoptosis; Carbon Monoxide; Cells, Cultured; Dose-Response Rela

2016