carbocyanines and Colitis--Ulcerative

carbocyanines has been researched along with Colitis--Ulcerative* in 2 studies

Other Studies

2 other study(ies) available for carbocyanines and Colitis--Ulcerative

Article	Year
Standards for bacterial identification by fluorescence In situ hybridization within eukaryotic tissue using ribosomal rRNA-based probes. Inflammatory bowel diseases, 2006, Volume: 12, Issue:8 Topics: Bacteroides; Bifidobacterium; Carbocyanines; Colitis, Ulcerative; Crohn Disease; Fluorescent Dyes; Humans; In Situ Hybridization, Fluorescence; Microscopy, Phase-Contrast; RNA Probes; RNA, Bacterial; RNA, Ribosomal	2006
Colonic inflammation increases Na+ currents in bladder sensory neurons. Neuroreport, 2004, Dec-03, Volume: 15, Issue:17 The purpose of this study was to determine whether sensitization of pre-spinal afferents induced by colonic inflammation can affect the physiology of the urinary bladder. Tetrodotoxin-resistant (TTX-R) Na+ currents were examined in bladder sensory neurons after experimental colitis. Cell bodies of bladder dorsal root ganglia were retrogradely labeled and TTX-R Na+ currents were recorded in capsaicin-sensitive neurons. Colitis significantly enhanced response of bladder neurons to capsaicin by approximately 60% and the peak amplitude of TTX-R Na+ current by 51%. These results suggest that colonic inflammation leads to increased excitability of nociceptive bladder neurons. Topics: Animals; Capsaicin; Carbocyanines; Cells, Cultured; Colitis, Ulcerative; Dextran Sulfate; Drug Interactions; Electric Stimulation; Ganglia, Spinal; Ion Channel Gating; Male; Membrane Potentials; Neurons; Patch-Clamp Techniques; Rats; Rats, Sprague-Dawley; Sodium Channel Blockers; Sodium Channels; Tetrodotoxin; Urinary Bladder	2004

Article

Year

Standards for bacterial identification by fluorescence In situ hybridization within eukaryotic tissue using ribosomal rRNA-based probes.

Inflammatory bowel diseases, 2006, Volume: 12, Issue:8

Topics: Bacteroides; Bifidobacterium; Carbocyanines; Colitis, Ulcerative; Crohn Disease; Fluorescent Dyes; Humans; In Situ Hybridization, Fluorescence; Microscopy, Phase-Contrast; RNA Probes; RNA, Bacterial; RNA, Ribosomal

2006

Colonic inflammation increases Na+ currents in bladder sensory neurons.

Neuroreport, 2004, Dec-03, Volume: 15, Issue:17

The purpose of this study was to determine whether sensitization of pre-spinal afferents induced by colonic inflammation can affect the physiology of the urinary bladder. Tetrodotoxin-resistant (TTX-R) Na+ currents were examined in bladder sensory neurons after experimental colitis. Cell bodies of bladder dorsal root ganglia were retrogradely labeled and TTX-R Na+ currents were recorded in capsaicin-sensitive neurons. Colitis significantly enhanced response of bladder neurons to capsaicin by approximately 60% and the peak amplitude of TTX-R Na+ current by 51%. These results suggest that colonic inflammation leads to increased excitability of nociceptive bladder neurons.

Topics: Animals; Capsaicin; Carbocyanines; Cells, Cultured; Colitis, Ulcerative; Dextran Sulfate; Drug Interactions; Electric Stimulation; Ganglia, Spinal; Ion Channel Gating; Male; Membrane Potentials; Neurons; Patch-Clamp Techniques; Rats; Rats, Sprague-Dawley; Sodium Channel Blockers; Sodium Channels; Tetrodotoxin; Urinary Bladder

2004