carbenoxolone sodium has been researched along with Nerve Degeneration in 4 studies
Timeframe | Studies, this research(%) | All Research% |
---|---|---|
pre-1990 | 0 (0.00) | 18.7374 |
1990's | 0 (0.00) | 18.2507 |
2000's | 2 (50.00) | 29.6817 |
2010's | 2 (50.00) | 24.3611 |
2020's | 0 (0.00) | 2.80 |
Authors | Studies |
---|---|
Doi, Y; Jin, S; Kawanokuchi, J; Liang, J; Mizuno, T; Sawada, M; Suzuki, H; Suzumura, A; Takeuchi, H | 1 |
Suzumura, A | 1 |
Boone, DR; Crookshanks, JM; DeWitt, DS; Hellmich, HL; Masel, BE; Micci, MA; Prough, DS; Rojo, DR; Sell, SL | 1 |
Benabou, S; de Pina-Benabou, MH; Federoff, HJ; Kyrozis, A; Rempe, D; Rozental, R; Spray, DC; Stanton, PK; Szostak, V; Urban-Maldonado, M; Uziel, D | 1 |
4 other study(ies) available for carbenoxolone sodium and Nerve Degeneration
Article | Year |
---|---|
Blockade of microglial glutamate release protects against ischemic brain injury.
Topics: Animals; Brain Ischemia; Carbenoxolone; Diazooxonorleucine; Dose-Response Relationship, Drug; Gap Junctions; Gerbillinae; Glutamic Acid; Male; Microglia; Nerve Degeneration; Neurons | 2008 |
[Microglia and neuronal degeneration].
Topics: Alzheimer Disease; Amyotrophic Lateral Sclerosis; Animals; Carbenoxolone; Cytokines; Disease Models, Animal; Drug Design; Glutamates; Humans; Microglia; Molecular Targeted Therapy; Multiple Sclerosis; Nerve Degeneration; Neurogenic Inflammation | 2010 |
Pathway analysis reveals common pro-survival mechanisms of metyrapone and carbenoxolone after traumatic brain injury.
Topics: Animals; Brain Injuries; Carbenoxolone; Cell Death; Cell Survival; Gene Expression Regulation; Hippocampus; Humans; Male; Metyrapone; Nerve Degeneration; Neuroprotective Agents; Rats; Rats, Sprague-Dawley; Reactive Oxygen Species; Signal Transduction; Stress, Physiological | 2013 |
Blockade of gap junctions in vivo provides neuroprotection after perinatal global ischemia.
Topics: Animals; Anti-Ulcer Agents; Apoptosis; Brain; Carbenoxolone; Caspase 3; Caspases; Cell Communication; Connexins; Disease Models, Animal; DNA; Electrophysiology; Female; Gap Junctions; Glucose; Hippocampus; Hypoxia; Hypoxia-Ischemia, Brain; Ischemia; Male; Mice; Mice, Inbred C57BL; Microscopy, Fluorescence; Nerve Degeneration; Neurons; Neuroprotective Agents; Nucleosomes; Oxygen; Polymerase Chain Reaction; Propidium; Rats; Rats, Sprague-Dawley; RNA, Messenger; Time Factors | 2005 |