carbenoxolone sodium has been researched along with Epilepsy, Absence in 4 studies
Timeframe | Studies, this research(%) | All Research% |
---|---|---|
pre-1990 | 0 (0.00) | 18.7374 |
1990's | 0 (0.00) | 18.2507 |
2000's | 3 (75.00) | 29.6817 |
2010's | 1 (25.00) | 24.3611 |
2020's | 0 (0.00) | 2.80 |
Authors | Studies |
---|---|
Gigout, S; Louvel, J; Pumain, R | 2 |
Barresi, V; Belluardo, N; Citraro, R; Condorelli, D; De Sarro, G; Gareri, P; Ibbadu, GF; Mudò, G; Russo, E; Trovato-Salinaro, A; Trovato-Salinato, A | 1 |
El-Beheiry, M; Khokhotva, V; Kokarovtseva, L; Leshchenko, Y; Perez Velazquez, JL; Proulx, E; Snead, OC | 1 |
4 other study(ies) available for carbenoxolone sodium and Epilepsy, Absence
Article | Year |
---|---|
Decrease of neocortical paired-pulse depression in GAERS and possible implication of gap junctions.
Topics: Animals; Anticonvulsants; Carbenoxolone; Cerebral Cortex; Disease Models, Animal; Electric Stimulation; Epilepsy, Absence; Gap Junctions; In Vitro Techniques; Neocortex; Quinidine; Rats, Wistar; Species Specificity; Synaptic Transmission | 2015 |
Antiabsence effects of carbenoxolone in two genetic animal models of absence epilepsy (WAG/Rij rats and lh/lh mice).
Topics: Animals; Autoradiography; Carbenoxolone; Connexins; Disease Models, Animal; Dose-Response Relationship, Drug; Electroencephalography; Epilepsy, Absence; Gene Expression Regulation; Male; Mice; Mice, Neurologic Mutants; Rats; Rats, Inbred Strains; Thalamic Nuclei; Time Factors | 2005 |
Functional contribution of specific brain areas to absence seizures: role of thalamic gap-junctional coupling.
Topics: Animals; Animals, Newborn; Brain Mapping; Carbenoxolone; Disease Models, Animal; Electrodes; Electroencephalography; Epilepsy, Absence; Evoked Potentials; Functional Laterality; Gap Junctions; Methylamines; Rats; Rats, Long-Evans; Spectrum Analysis; Thalamus; Time Factors; trans-1,4-Bis(2-chlorobenzaminomethyl)cyclohexane Dihydrochloride | 2006 |
Effects in vitro and in vivo of a gap junction blocker on epileptiform activities in a genetic model of absence epilepsy.
Topics: 4-Aminopyridine; Animals; Anticonvulsants; Carbenoxolone; Cerebral Cortex; Disease Models, Animal; Electroencephalography; Epilepsy, Absence; Evoked Potentials; Gap Junctions; Potassium Channel Blockers; Rats; Rats, Inbred Strains; Rats, Wistar; Seizures; Sodium Chloride; Somatosensory Cortex; Thalamic Nuclei | 2006 |