carbamates has been researched along with Hepatolenticular Degeneration in 3 studies
Hepatolenticular Degeneration: A rare autosomal recessive disease characterized by the deposition of copper in the BRAIN; LIVER; CORNEA; and other organs. It is caused by defects in the ATP7B gene encoding copper-transporting ATPase 2 (EC 3.6.3.4), also known as the Wilson disease protein. The overload of copper inevitably leads to progressive liver and neurological dysfunction such as LIVER CIRRHOSIS; TREMOR; ATAXIA and intellectual deterioration. Hepatic dysfunction may precede neurologic dysfunction by several years.
Timeframe | Studies, this research(%) | All Research% |
---|---|---|
pre-1990 | 3 (100.00) | 18.7374 |
1990's | 0 (0.00) | 18.2507 |
2000's | 0 (0.00) | 29.6817 |
2010's | 0 (0.00) | 24.3611 |
2020's | 0 (0.00) | 2.80 |
Authors | Studies |
---|---|
SUNDERMAN, FW | 2 |
WHITE, JC | 1 |
PETIT, H | 1 |
Lindquist, RR | 1 |
3 other studies available for carbamates and Hepatolenticular Degeneration
Article | Year |
---|---|
Metabolic balance studies in hepatolenticular degeneration treated with diethyldithiocarbamate.
Topics: Carbamates; Chelating Agents; Copper; Ditiocarb; Hepatolenticular Degeneration; Humans; Minerals; Ni | 1963 |
[CURRENT ASPECTS OF WILSON'S DISEASE].
Topics: Carbamates; Ceruloplasmin; Diagnosis; Diet; Diet Therapy; Dimercaprol; Drug Therapy; Esters; Genetic | 1964 |
Studies on the pathogenesis of hepatolenticular degeneration. II. Cytochemical methods for the localization of copper.
Topics: Amides; Animals; Carbamates; Carbazoles; Chemical Phenomena; Chemistry; Copper; Hepatolenticular Deg | 1969 |