Page last updated: 2024-08-23

captopril and Nerve Degeneration

captopril has been researched along with Nerve Degeneration in 4 studies

Research

Studies (4)

TimeframeStudies, this research(%)All Research%
pre-19900 (0.00)18.7374
1990's0 (0.00)18.2507
2000's2 (50.00)29.6817
2010's2 (50.00)24.3611
2020's0 (0.00)2.80

Authors

AuthorsStudies
Coleman, C; Gadad, BS; German, DC; Harris, SL; Li, W; Richardson, JR; Sonsalla, PK; Wong, LY1
AbdAlla, S; Abdelbaset, A; El Hakim, A; Elfaramawy, Y; Quitterer, U1
Flora, SJ; Mehta, A; Mishra, D; Pachauri, V; Saxena, G1
Guerra, MJ; Labandeira-Garcia, JL; Mendez-Alvarez, E; Muñoz, A; Rey, P; Soto-Otero, R1

Other Studies

4 other study(ies) available for captopril and Nerve Degeneration

ArticleYear
The angiotensin converting enzyme inhibitor captopril protects nigrostriatal dopamine neurons in animal models of parkinsonism.
    Experimental neurology, 2013, Volume: 250

    Topics: Angiotensin-Converting Enzyme Inhibitors; Animals; Brain; Captopril; Dopaminergic Neurons; Immunohistochemistry; Male; Mice; MPTP Poisoning; Nerve Degeneration; Neuroprotective Agents; Rats

2013
Inhibition of ACE Retards Tau Hyperphosphorylation and Signs of Neuronal Degeneration in Aged Rats Subjected to Chronic Mild Stress.
    BioMed research international, 2015, Volume: 2015

    Topics: Alzheimer Disease; Angiotensin-Converting Enzyme Inhibitors; Animals; Captopril; gamma-Synuclein; Gene Expression Regulation; Glutamate Decarboxylase; Hippocampus; Humans; Microtubule-Associated Proteins; Nerve Degeneration; Neurons; Peptidyl-Dipeptidase A; Phosphorylation; Rats; tau Proteins

2015
Combinational chelation therapy abrogates lead-induced neurodegeneration in rats.
    Toxicology and applied pharmacology, 2009, Oct-15, Volume: 240, Issue:2

    Topics: Animals; Antioxidants; Apoptosis; Behavior, Animal; Biogenic Amines; Calcium; Captopril; Chelating Agents; Cytochromes c; Disease Models, Animal; DNA Damage; Drug Therapy, Combination; Exploratory Behavior; Lead Poisoning, Nervous System; Learning; Male; Membrane Potential, Mitochondrial; Memory; Mitochondria; Motor Activity; Nerve Degeneration; Neurons; Nitric Oxide; Organometallic Compounds; Oxidative Stress; Rats; Rats, Wistar; Reactive Oxygen Species; Succimer

2009
Reduction of dopaminergic degeneration and oxidative stress by inhibition of angiotensin converting enzyme in a MPTP model of parkinsonism.
    Neuropharmacology, 2006, Volume: 51, Issue:1

    Topics: 1-Methyl-4-phenyl-1,2,3,6-tetrahydropyridine; Angiotensin-Converting Enzyme Inhibitors; Animals; Antiparkinson Agents; Captopril; Dopamine; Dopamine Agents; Dopamine Plasma Membrane Transport Proteins; Immunohistochemistry; In Vitro Techniques; Lipid Peroxidation; Male; Mesencephalon; Mice; Mice, Inbred C57BL; Mitochondria; Monoamine Oxidase; Neostriatum; Nerve Degeneration; Nerve Endings; Nerve Tissue Proteins; Neuroprotective Agents; Oxidative Stress; Parkinson Disease, Secondary; Pyridinium Compounds; Synaptosomes; Tyrosine 3-Monooxygenase

2006