capsaicin has been researched along with Experimental Mammary Neoplasms in 6 studies
| Timeframe | Studies, this research(%) | All Research% |
|---|---|---|
| pre-1990 | 0 (0.00) | 18.7374 |
| 1990's | 0 (0.00) | 18.2507 |
| 2000's | 1 (16.67) | 29.6817 |
| 2010's | 4 (66.67) | 24.3611 |
| 2020's | 1 (16.67) | 2.80 |
| Authors | Studies |
|---|---|
| Chen, M; Jiang, W; Lian, B; Liang, Z; Qin, Q; Tan, Q; Wei, C; Xiao, C; Yang, W | 1 |
| Bin-Meferij, MM; El-Kott, AF | 1 |
| Wan, Y | 1 |
| Abbassi, S; Iwanski, GB; Karlan, B; Koeffler, HP; La, DT; Leiter, A; Lu, D; Mehta, R; O'Kelly, J; Thoennissen, NH | 1 |
| Demir, N; Duymuş, O; Erin, N; Oztürk, S | 1 |
| Bylander, J; Chase, G; Clawson, G; Erin, N; Zhao, W | 1 |
1 review(s) available for capsaicin and Experimental Mammary Neoplasms
| Article | Year |
|---|---|
New Mechanism of Bone Cancer Pain: Tumor Tissue-Derived Endogenous Formaldehyde Induced Bone Cancer Pain via TRPV1 Activation.
Topics: Animals; Bone Neoplasms; Cancer Pain; Capsaicin; Cells, Cultured; Formaldehyde; Glycine Hydroxymethyltransferase; Histone Demethylases; Humans; Insulin-Like Growth Factor I; Mammary Neoplasms, Experimental; Neoplasm Proteins; Osteoblasts; Peripheral Nerves; Podophyllotoxin; Protein Kinase Inhibitors; Signal Transduction; TRPV Cation Channels | 2016 |
5 other study(ies) available for capsaicin and Experimental Mammary Neoplasms
| Article | Year |
|---|---|
Capsaicin Inhibits Proliferation and Induces Apoptosis in Breast Cancer by Down-Regulating FBI-1-Mediated NF-κB Pathway.
Topics: Animals; Antineoplastic Agents; Apoptosis; Breast Neoplasms; Capsaicin; Cell Proliferation; DNA-Binding Proteins; Down-Regulation; Drug Screening Assays, Antitumor; Female; Humans; Mammary Neoplasms, Experimental; Mice; Mice, Inbred BALB C; Mice, Nude; NF-kappa B; Proto-Oncogene Mas; Transcription Factors; Tumor Cells, Cultured | 2021 |
Suppressive effects of capsaicin against N-nitrosomethylurea-induced mammary tumorigenesis in rats.
Topics: Animals; Antioxidants; Capsaicin; Carcinogenesis; Estradiol; Female; Mammary Glands, Animal; Mammary Neoplasms, Experimental; Methylnitrosourea; Progesterone; Proliferating Cell Nuclear Antigen; Rats; Rats, Sprague-Dawley | 2018 |
Capsaicin causes cell-cycle arrest and apoptosis in ER-positive and -negative breast cancer cells by modulating the EGFR/HER-2 pathway.
Topics: Animals; Apoptosis; Blotting, Western; Breast Neoplasms; Capsaicin; Cell Cycle; Cell Line; Cell Line, Tumor; Cell Movement; Cell Proliferation; Dose-Response Relationship, Drug; ErbB Receptors; Female; Humans; Mammary Neoplasms, Experimental; Mice; Mice, Nude; Models, Biological; Receptor, ErbB-2; Receptors, Estrogen; Reverse Transcriptase Polymerase Chain Reaction; Signal Transduction; Xenograft Model Antitumor Assays | 2010 |
Activation of vagus nerve by semapimod alters substance P levels and decreases breast cancer metastasis.
Topics: Animals; Antineoplastic Agents; Breast Neoplasms; Capsaicin; Cell Line, Tumor; Female; Hydrazones; Liver Neoplasms, Experimental; Lung Neoplasms; Mammary Neoplasms, Experimental; Mice; Mice, Inbred BALB C; Neoplasm Metastasis; Neprilysin; Sensory Receptor Cells; Substance P; Vagotomy; Vagus Nerve | 2012 |
Capsaicin-induced inactivation of sensory neurons promotes a more aggressive gene expression phenotype in breast cancer cells.
Topics: ADAM Proteins; ADAM10 Protein; Amyloid Precursor Protein Secretases; Animals; Capsaicin; Caspase 7; Cell Line, Tumor; Denervation; Down-Regulation; Female; Gene Expression Profiling; Gene Expression Regulation, Neoplastic; Heart; Heart Neoplasms; Hydrolysis; Lung; Lung Neoplasms; Mammary Neoplasms, Experimental; Membrane Proteins; Mice; Mice, Inbred BALB C; Neoplasm Invasiveness; Neurons, Afferent; Oligonucleotide Array Sequence Analysis; Phenotype; Proto-Oncogene Proteins c-ets; Substance P; Time Factors | 2006 |