capsaicin has been researched along with Benign Neoplasms, Brain in 5 studies
Timeframe | Studies, this research(%) | All Research% |
---|---|---|
pre-1990 | 0 (0.00) | 18.7374 |
1990's | 0 (0.00) | 18.2507 |
2000's | 3 (60.00) | 29.6817 |
2010's | 1 (20.00) | 24.3611 |
2020's | 1 (20.00) | 2.80 |
Authors | Studies |
---|---|
Palka, J; Szoka, L | 1 |
Chirasani, SR; Cravatt, BF; Cristino, L; De Petrocellis, L; Di Marzo, V; Dittmar, G; Glass, R; Gurok, U; Imperatore, R; Kettenmann, H; Kumar, J; Lewin, GR; Ligresti, A; Matyash, V; Momma, S; Nuber, UA; Petrosino, S; Purfürst, B; Smith, ES; Stock, K; Synowitz, M; Wälzlein, JH; Wend, P | 1 |
Fowler, CJ; Jonsson, KO; Lambert, DM; Smart, D; Vandevoorde, S | 1 |
Amantini, C; Arcella, A; Caprodossi, S; Giangaspero, F; Lucciarini, R; Mosca, M; Nabissi, M; Santoni, G | 1 |
Kim, JA; Lee, YS; Nam, DH | 1 |
5 other study(ies) available for capsaicin and Benign Neoplasms, Brain
Article | Year |
---|---|
Capsaicin up-regulates pro-apoptotic activity of thiazolidinediones in glioblastoma cell line.
Topics: Apoptosis; Brain Neoplasms; Capsaicin; Cell Line, Tumor; Dose-Response Relationship, Drug; Drug Synergism; Glioblastoma; Humans; Ligands; Pioglitazone; PPAR gamma; Rosiglitazone; TRPV Cation Channels; Up-Regulation | 2020 |
Neural precursor cells induce cell death of high-grade astrocytomas through stimulation of TRPV1.
Topics: Aging; Amides; Amidohydrolases; Animals; Antineoplastic Agents; Apoptosis; Arachidonic Acids; Brain; Brain Neoplasms; Capsaicin; Cell Movement; Culture Media, Conditioned; Dopamine; Endocannabinoids; Ethanolamines; Female; Gene Expression Regulation, Neoplastic; Glioblastoma; Humans; Male; Mice; Mice, Inbred C57BL; Mice, Knockout; Mice, SCID; Neoplasm Proteins; Neural Stem Cells; Oleic Acids; Palmitic Acids; Polyunsaturated Alkamides; Real-Time Polymerase Chain Reaction; RNA, Small Interfering; TRPV Cation Channels; Tumor Cells, Cultured | 2012 |
N-Morpholino- and N-diethyl-analogues of palmitoylethanolamide increase the sensitivity of transfected human vanilloid receptors to activation by anandamide without affecting fatty acid amidohydrolase activity.
Topics: Amides; Amidohydrolases; Animals; Arachidonic Acids; Brain Neoplasms; Calcium Channel Blockers; Capsaicin; Endocannabinoids; Ethanolamines; Glioma; Humans; In Vitro Techniques; Kidney; Morpholines; Palmitic Acids; Polyunsaturated Alkamides; Radioligand Assay; Rats; Receptor, Cannabinoid, CB1; Receptors, Drug; Transfection; Tumor Cells, Cultured | 2003 |
Capsaicin-induced apoptosis of glioma cells is mediated by TRPV1 vanilloid receptor and requires p38 MAPK activation.
Topics: Antineoplastic Agents; Apoptosis; Astrocytes; Brain Neoplasms; Calcium Signaling; Capsaicin; Caspase 3; Cell Line, Tumor; Enzyme Activation; Gene Expression Regulation, Neoplastic; Glioblastoma; Glioma; Humans; Membrane Potential, Mitochondrial; p38 Mitogen-Activated Protein Kinases; RNA, Messenger; TRPV Cation Channels | 2007 |
Induction of apoptosis by capsaicin in A172 human glioblastoma cells.
Topics: Apoptosis; Brain Neoplasms; Calcium; Capsaicin; Dose-Response Relationship, Drug; Glioblastoma; Humans; Kinetics; Lipid Peroxidation; Reactive Oxygen Species; Receptors, Drug; Tumor Cells, Cultured | 2000 |