cantharidin has been researched along with Cancer of the Tongue in 2 studies
Cantharidin: A toxic compound, isolated from the Spanish fly or blistering beetle (Lytta (Cantharis) vesicatoria) and other insects. It is a potent and specific inhibitor of protein phosphatases 1 (PP1) and 2A (PP2A). This compound can produce severe skin inflammation, and is extremely toxic if ingested orally.
cantharidin : A monoterpenoid with an epoxy-bridged cyclic dicarboxylic anhydride structure secreted by many species of blister beetle, and most notably by the Spanish fly, Lytta vesicatoria. Natural toxin inhibitor of protein phosphatases 1 and 2A.
Excerpt | Relevance | Reference |
---|---|---|
"Most of oral cancer is histopathologically diagnosed as oral squamous cell carcinoma (OSCC)." | 1.43 | Cantharidin Induced Oral Squamous Cell Carcinoma Cell Apoptosis via the JNK-Regulated Mitochondria and Endoplasmic Reticulum Stress-Related Signaling Pathways. ( Chen, MK; Kuo, CY; Lee, KI; Liu, SH; Su, CC; Tang, CH, 2016) |
Timeframe | Studies, this research(%) | All Research% |
---|---|---|
pre-1990 | 0 (0.00) | 18.7374 |
1990's | 0 (0.00) | 18.2507 |
2000's | 0 (0.00) | 29.6817 |
2010's | 2 (100.00) | 24.3611 |
2020's | 0 (0.00) | 2.80 |
Authors | Studies |
---|---|
Tian, X | 1 |
Zeng, G | 1 |
Li, X | 1 |
Wu, Z | 1 |
Wang, L | 1 |
Su, CC | 1 |
Lee, KI | 1 |
Chen, MK | 1 |
Kuo, CY | 1 |
Tang, CH | 1 |
Liu, SH | 1 |
2 other studies available for cantharidin and Cancer of the Tongue
Article | Year |
---|---|
Cantharidin inhibits cell proliferation and promotes apoptosis in tongue squamous cell carcinoma through suppression of miR-214 and regulation of p53 and Bcl-2/Bax.
Topics: Antineoplastic Agents; Apoptosis; bcl-2-Associated X Protein; Cantharidin; Carcinoma, Squamous Cell; | 2015 |
Cantharidin Induced Oral Squamous Cell Carcinoma Cell Apoptosis via the JNK-Regulated Mitochondria and Endoplasmic Reticulum Stress-Related Signaling Pathways.
Topics: Antineoplastic Agents; Apoptosis; Blotting, Western; Cantharidin; Carcinoma, Squamous Cell; Cell Lin | 2016 |