caffeine has been researched along with Acute Lung Injury in 2 studies
Acute Lung Injury: A condition of lung damage that is characterized by bilateral pulmonary infiltrates (PULMONARY EDEMA) rich in NEUTROPHILS, and in the absence of clinical HEART FAILURE. This can represent a spectrum of pulmonary lesions, endothelial and epithelial, due to numerous factors (physical, chemical, or biological).
| Excerpt | Relevance | Reference |
|---|---|---|
| " Similarly, the consumption of caffeine antagonizes A2ARs, resulting in exacerbation of ongoing acute inflammation." | 8.85 | The antihypoxia-adenosinergic pathogenesis as a result of collateral damage by overactive immune cells. ( Belikoff, B; Hatfield, S; Lukashev, D; Ohta, A; Sitkovsky, M, 2009) |
| " Similarly, the consumption of caffeine antagonizes A2ARs, resulting in exacerbation of ongoing acute inflammation." | 4.85 | The antihypoxia-adenosinergic pathogenesis as a result of collateral damage by overactive immune cells. ( Belikoff, B; Hatfield, S; Lukashev, D; Ohta, A; Sitkovsky, M, 2009) |
| Timeframe | Studies, this research(%) | All Research% |
|---|---|---|
| pre-1990 | 0 (0.00) | 18.7374 |
| 1990's | 0 (0.00) | 18.2507 |
| 2000's | 1 (50.00) | 29.6817 |
| 2010's | 1 (50.00) | 24.3611 |
| 2020's | 0 (0.00) | 2.80 |
| Authors | Studies |
|---|---|
| Hatfield, S | 1 |
| Belikoff, B | 1 |
| Lukashev, D | 1 |
| Sitkovsky, M | 1 |
| Ohta, A | 1 |
| Li, J | 1 |
| Li, G | 1 |
| Hu, JL | 1 |
| Fu, XH | 1 |
| Zeng, YJ | 1 |
| Zhou, YG | 1 |
| Xiong, G | 1 |
| Yang, N | 1 |
| Dai, SS | 1 |
| He, FT | 1 |
1 review available for caffeine and Acute Lung Injury
| Article | Year |
|---|---|
The antihypoxia-adenosinergic pathogenesis as a result of collateral damage by overactive immune cells.
Topics: Acute Lung Injury; Adenosine; Adenosine A2 Receptor Antagonists; Animals; Caffeine; Disease Models, | 2009 |
1 other study available for caffeine and Acute Lung Injury
| Article | Year |
|---|---|
Chronic or high dose acute caffeine treatment protects mice against oleic acid-induced acute lung injury via an adenosine A2A receptor-independent mechanism.
Topics: Acute Lung Injury; Animals; Caffeine; Cyclic AMP; Cytokines; Dose-Response Relationship, Drug; Drug | 2011 |