caffeic acid phenethyl ester has been researched along with Hepatocellular Carcinoma in 6 studies
| Timeframe | Studies, this research(%) | All Research% |
|---|---|---|
| pre-1990 | 0 (0.00) | 18.7374 |
| 1990's | 0 (0.00) | 18.2507 |
| 2000's | 2 (33.33) | 29.6817 |
| 2010's | 3 (50.00) | 24.3611 |
| 2020's | 1 (16.67) | 2.80 |
| Authors | Studies |
|---|---|
| Cao, Y; Du, Q; Jiang, Y; Jiao, X; Li, Y; Liu, X; Tang, M; Tian, C; Wang, Y; Wang, Z; Xie, P; Yang, J | 1 |
| Kim, AK; Kim, EY; Ryu, JH | 1 |
| Han, EH; Im, JH; Jeong, HG; Jin, SW; Kim, HG; Lee, EJ | 1 |
| Choi, IW; Choi, YH; Dilshara, MG; Jayasooriya, RG; Kim, GY; Park, SR | 1 |
| Arce-Popoca, E; Carrasco-Legleu, CE; Fattel-Fazenda, S; Márquez-Rosado, L; Pérez-Carreón, JI; Villa-Treviño, S | 1 |
| Chang, YC; Cho, G; Chung, TW; Kim, CH; Kim, JG; Kim, SH; Ko, JH; Lee, YC; Moon, SK | 1 |
6 other study(ies) available for caffeic acid phenethyl ester and Hepatocellular Carcinoma
| Article | Year |
|---|---|
Discovery of CAPE derivatives as dual EGFR and CSK inhibitors with anticancer activity in a murine model of hepatocellular carcinoma.
Topics: Animals; Antineoplastic Agents; Binding Sites; Caffeic Acids; Carcinoma, Hepatocellular; Catalytic Domain; Cell Line, Tumor; Cell Proliferation; Disease Models, Animal; Dose-Response Relationship, Drug; Down-Regulation; Drug Screening Assays, Antitumor; ErbB Receptors; Female; Liver Neoplasms; Male; Mice; Molecular Docking Simulation; Phenylethyl Alcohol; src-Family Kinases; Structure-Activity Relationship | 2021 |
CAPE promotes TRAIL-induced apoptosis through the upregulation of TRAIL receptors via activation of p38 and suppression of JNK in SK-Hep1 hepatocellular carcinoma cells.
Topics: Animals; Apoptosis; Bees; Caffeic Acids; Carcinoma, Hepatocellular; Cell Line, Tumor; Gene Expression Regulation, Neoplastic; Humans; Liver Neoplasms; MAP Kinase Kinase 4; p38 Mitogen-Activated Protein Kinases; Phenylethyl Alcohol; Propolis; Receptors, TNF-Related Apoptosis-Inducing Ligand; Signal Transduction; TNF-Related Apoptosis-Inducing Ligand; Up-Regulation | 2013 |
Caffeic acid phenethyl ester inhibits 3-MC-induced CYP1A1 expression through induction of hypoxia-inducible factor-1α.
Topics: Animals; Apoptosis; Caffeic Acids; Carcinoma, Hepatocellular; Cell Line, Tumor; Cell Survival; Cytochrome P-450 CYP1A1; Dose-Response Relationship, Drug; Gene Expression Regulation, Neoplastic; Hypoxia-Inducible Factor 1, alpha Subunit; Methylcholanthrene; Mice; Phenylethyl Alcohol | 2015 |
Caffeic acid phenethyl ester enhances TRAIL-mediated apoptosis via CHOP-induced death receptor 5 upregulation in hepatocarcinoma Hep3B cells.
Topics: Apoptosis; Caffeic Acids; Carcinoma, Hepatocellular; Cell Line, Tumor; Gene Expression Regulation, Neoplastic; Humans; Liver Neoplasms; Neoplasm Proteins; Phenylethyl Alcohol; Receptors, TNF-Related Apoptosis-Inducing Ligand; TNF-Related Apoptosis-Inducing Ligand; Transcription Factor CHOP; Up-Regulation | 2016 |
Chemoprotective effect of caffeic acid phenethyl ester on promotion in a medium-term rat hepatocarcinogenesis assay.
Topics: Animals; Blotting, Western; Caffeic Acids; Carcinoma, Hepatocellular; Cell Nucleus; Disease Models, Animal; Disease Progression; gamma-Glutamyltransferase; Gene Expression Regulation; Glutathione S-Transferase pi; Glutathione Transferase; Humans; I-kappa B Proteins; Isoenzymes; Liver Neoplasms, Experimental; Male; NF-kappa B; NF-KappaB Inhibitor alpha; Phenylethyl Alcohol; Precancerous Conditions; Protein Transport; Rats; Rats, Wistar; Reverse Transcriptase Polymerase Chain Reaction; RNA, Messenger | 2004 |
Novel and therapeutic effect of caffeic acid and caffeic acid phenyl ester on hepatocarcinoma cells: complete regression of hepatoma growth and metastasis by dual mechanism.
Topics: Animals; Antineoplastic Agents; Caffeic Acids; Carcinoma, Hepatocellular; Cell Line, Tumor; Cell Movement; Cell Proliferation; Cell Survival; Humans; Liver Neoplasms; Matrix Metalloproteinase 9; Matrix Metalloproteinase Inhibitors; Mice; Neoplasm Metastasis; NF-kappa B; Phenylethyl Alcohol; Transcription, Genetic | 2004 |