bumetanide has been researched along with Brain Injuries in 6 studies
Brain Injuries: Acute and chronic (see also BRAIN INJURIES, CHRONIC) injuries to the brain, including the cerebral hemispheres, CEREBELLUM, and BRAIN STEM. Clinical manifestations depend on the nature of injury. Diffuse trauma to the brain is frequently associated with DIFFUSE AXONAL INJURY or COMA, POST-TRAUMATIC. Localized injuries may be associated with NEUROBEHAVIORAL MANIFESTATIONS; HEMIPARESIS, or other focal neurologic deficits.
| Excerpt | Relevance | Reference |
|---|---|---|
| "The development of cytotoxic brain edema resulting in increased intracranial pressure is a major cause of death occurring in the early phase of traumatic brain injury (TBI)." | 1.37 | Aquaporin-4 expression in cultured astrocytes after fluid percussion injury. ( Curtis, KM; Norenberg, MD; Rao, KV; Reddy, PV, 2011) |
| Timeframe | Studies, this research(%) | All Research% |
|---|---|---|
| pre-1990 | 0 (0.00) | 18.7374 |
| 1990's | 0 (0.00) | 18.2507 |
| 2000's | 3 (50.00) | 29.6817 |
| 2010's | 2 (33.33) | 24.3611 |
| 2020's | 1 (16.67) | 2.80 |
| Authors | Studies |
|---|---|
| Tóth, K | 1 |
| Lénárt, N | 1 |
| Berki, P | 1 |
| Fekete, R | 1 |
| Szabadits, E | 1 |
| Pósfai, B | 1 |
| Cserép, C | 1 |
| Alatshan, A | 1 |
| Benkő, S | 1 |
| Kiss, D | 1 |
| Hübner, CA | 1 |
| Gulyás, A | 1 |
| Kaila, K | 2 |
| Környei, Z | 1 |
| Dénes, Á | 1 |
| Shulga, A | 1 |
| Thomas-Crusells, J | 1 |
| Sigl, T | 1 |
| Blaesse, A | 1 |
| Mestres, P | 1 |
| Meyer, M | 1 |
| Yan, Q | 1 |
| Saarma, M | 1 |
| Rivera, C | 1 |
| Giehl, KM | 1 |
| Rao, KV | 1 |
| Reddy, PV | 1 |
| Curtis, KM | 1 |
| Norenberg, MD | 1 |
| Dzhala, V | 1 |
| Valeeva, G | 1 |
| Glykys, J | 1 |
| Khazipov, R | 1 |
| Staley, K | 1 |
| Lu, KT | 2 |
| Wu, CY | 2 |
| Cheng, NC | 1 |
| Wo, YY | 1 |
| Yang, JT | 1 |
| Yen, HH | 2 |
| Yang, YL | 2 |
| Peng, JH | 1 |
| Wang, CL | 1 |
6 other studies available for bumetanide and Brain Injuries
| Article | Year |
|---|---|
The NKCC1 ion transporter modulates microglial phenotype and inflammatory response to brain injury in a cell-autonomous manner.
Topics: Animals; Brain Edema; Brain Injuries; Bumetanide; Embryo, Mammalian; Gene Expression Regulation; Hip | 2022 |
Posttraumatic GABA(A)-mediated [Ca2+]i increase is essential for the induction of brain-derived neurotrophic factor-dependent survival of mature central neurons.
Topics: Animals; Animals, Newborn; Axons; Axotomy; Brain Injuries; Brain-Derived Neurotrophic Factor; Bumeta | 2008 |
Aquaporin-4 expression in cultured astrocytes after fluid percussion injury.
Topics: Analysis of Variance; Animals; Aquaporin 4; Astrocytes; Blotting, Western; Brain Edema; Brain Injuri | 2011 |
Traumatic alterations in GABA signaling disrupt hippocampal network activity in the developing brain.
Topics: 6-Cyano-7-nitroquinoxaline-2,3-dione; Action Potentials; Age Factors; Animals; Animals, Newborn; Ant | 2012 |
Inhibition of the Na+ -K+ -2Cl- -cotransporter in choroid plexus attenuates traumatic brain injury-induced brain edema and neuronal damage.
Topics: Animals; Brain Edema; Brain Injuries; Bumetanide; Choroid Plexus; Diuretics; Male; Neurons; Neuropro | 2006 |
Bumetanide administration attenuated traumatic brain injury through IL-1 overexpression.
Topics: Analysis of Variance; Animals; Brain Edema; Brain Injuries; Bumetanide; Disease Models, Animal; Hipp | 2007 |