Page last updated: 2024-10-23

bumetanide and Brain Infarction

bumetanide has been researched along with Brain Infarction in 3 studies

Brain Infarction: Tissue NECROSIS in any area of the brain, including the CEREBRAL HEMISPHERES, the CEREBELLUM, and the BRAIN STEM. Brain infarction is the result of a cascade of events initiated by inadequate blood flow through the brain that is followed by HYPOXIA and HYPOGLYCEMIA in brain tissue. Damage may be temporary, permanent, selective or pan-necrosis.

Research

Studies (3)

TimeframeStudies, this research(%)All Research%
pre-19900 (0.00)18.7374
1990's0 (0.00)18.2507
2000's1 (33.33)29.6817
2010's2 (66.67)24.3611
2020's0 (0.00)2.80

Authors

AuthorsStudies
Mu, XP1
Wang, HB1
Cheng, X1
Yang, L1
Sun, XY1
Qu, HL1
Zhao, SS1
Zhou, ZK1
Liu, TT1
Xiao, T1
Song, B1
Jolkkonen, J2
Zhao, CS1
Xu, W1
Mu, X1
Wang, H1
Song, C1
Ma, W1
Zhao, C1
Chen, H1
Luo, J1
Kintner, DB1
Shull, GE1
Sun, D1

Other Studies

3 other studies available for bumetanide and Brain Infarction

ArticleYear
Inhibition of Nkcc1 promotes axonal growth and motor recovery in ischemic rats.
    Neuroscience, 2017, Dec-04, Volume: 365

    Topics: Animals; Axons; Biotin; Brain Infarction; Brain Ischemia; Brain-Derived Neurotrophic Factor; Bumetan

2017
Chloride Co-transporter NKCC1 Inhibitor Bumetanide Enhances Neurogenesis and Behavioral Recovery in Rats After Experimental Stroke.
    Molecular neurobiology, 2017, Volume: 54, Issue:4

    Topics: Animals; Antigens, Nuclear; Behavior, Animal; Brain Infarction; Bromodeoxyuridine; Bumetanide; Cell

2017
Na(+)-dependent chloride transporter (NKCC1)-null mice exhibit less gray and white matter damage after focal cerebral ischemia.
    Journal of cerebral blood flow and metabolism : official journal of the International Society of Cerebral Blood Flow and Metabolism, 2005, Volume: 25, Issue:1

    Topics: Amyloid beta-Protein Precursor; Animals; Astrocytes; Brain Edema; Brain Infarction; Brain Ischemia;

2005