bumetanide has been researched along with Allodynia in 9 studies
| Excerpt | Relevance | Reference |
|---|---|---|
| "Recent behavioral and electrophysiological studies have attributed an important role to dorsal root reflexes (DRRs) in the initiation and development of neurogenic inflammation produced by intradermal capsaicin (CAP)." | 7.73 | Role of the Na+-K+-2Cl- cotransporter in the development of capsaicin-induced neurogenic inflammation. ( Castañeda-Hernandez, G; Lawand, NB; Lin, Q; Valencia-de Ita, S; Willis, WD, 2006) |
| " Accordingly, intracolonic capsaicin, which generates robust secondary (referred) allodynia on the abdomen of mice, also causes an increased spinal c-Fos labeling." | 3.79 | Stimulation of cutaneous low threshold mechanoreceptors in mice after intracolonic capsaicin increases spinal c-Fos labeling in an NKCC1-dependent fashion. ( Cervero, F; Nieto, FR; Pitcher, MH, 2013) |
| "Recent behavioral and electrophysiological studies have attributed an important role to dorsal root reflexes (DRRs) in the initiation and development of neurogenic inflammation produced by intradermal capsaicin (CAP)." | 3.73 | Role of the Na+-K+-2Cl- cotransporter in the development of capsaicin-induced neurogenic inflammation. ( Castañeda-Hernandez, G; Lawand, NB; Lin, Q; Valencia-de Ita, S; Willis, WD, 2006) |
| "Treatment with bumetanide, an NKCC inhibitor, had no effect on mechanical hypersensitivity seen in mice with EAE even though it reversed the changes in the levels of NKCC1 and KCC2." | 1.46 | The chloride co-transporters, NKCC1 and KCC2, in experimental autoimmune encephalomyelitis (EAE). ( Kerr, B; Tenorio, G; Yousuf, MS; Zubkow, K, 2017) |
| "Neuropathic pain is a common problem following spinal cord injury (SCI)." | 1.36 | Role of NKCC1 and KCC2 in the development of chronic neuropathic pain following spinal cord injury. ( Ahmed, MM; Hasbargen, T; Kahle, KT; Li, L; Miranpuri, G; Resnick, D; Sun, D, 2010) |
| "The rats developed hyperalgesia between days 21 and 42 post-SCI." | 1.35 | The role of cation-dependent chloride transporters in neuropathic pain following spinal cord injury. ( Allcock, B; Baggott, C; Cain, J; Cramer, SW; Miranpuri, G; Rajpal, S; Resnick, D; Sun, D; Tilghman, J, 2008) |
| Timeframe | Studies, this research(%) | All Research% |
|---|---|---|
| pre-1990 | 0 (0.00) | 18.7374 |
| 1990's | 0 (0.00) | 18.2507 |
| 2000's | 2 (22.22) | 29.6817 |
| 2010's | 7 (77.78) | 24.3611 |
| 2020's | 0 (0.00) | 2.80 |
| Authors | Studies |
|---|---|
| Mòdol, L | 1 |
| Cobianchi, S | 1 |
| Navarro, X | 1 |
| Shisler, D | 1 |
| Austin, TM | 1 |
| Delpire, E | 1 |
| Sawyer, DB | 1 |
| Pandey, AK | 1 |
| Huang, YJ | 1 |
| Lee, KH | 1 |
| Murphy, L | 1 |
| Garraway, SM | 1 |
| Grau, JW | 1 |
| Yousuf, MS | 1 |
| Zubkow, K | 1 |
| Tenorio, G | 1 |
| Kerr, B | 1 |
| Cramer, SW | 1 |
| Baggott, C | 1 |
| Cain, J | 1 |
| Tilghman, J | 1 |
| Allcock, B | 1 |
| Miranpuri, G | 2 |
| Rajpal, S | 1 |
| Sun, D | 2 |
| Resnick, D | 2 |
| Hasbargen, T | 1 |
| Ahmed, MM | 1 |
| Li, L | 1 |
| Kahle, KT | 1 |
| Wei, H | 1 |
| Hao, B | 1 |
| Huang, JL | 1 |
| Ma, AN | 1 |
| Li, XY | 1 |
| Wang, YX | 1 |
| Pertovaara, A | 1 |
| Pitcher, MH | 1 |
| Nieto, FR | 1 |
| Cervero, F | 1 |
| Valencia-de Ita, S | 1 |
| Lawand, NB | 1 |
| Lin, Q | 1 |
| Castañeda-Hernandez, G | 1 |
| Willis, WD | 1 |
9 other studies available for bumetanide and Allodynia
| Article | Year |
|---|---|
Prevention of NKCC1 phosphorylation avoids downregulation of KCC2 in central sensory pathways and reduces neuropathic pain after peripheral nerve injury.
Topics: Afferent Pathways; Animals; Bumetanide; Down-Regulation; Female; Hyperalgesia; K Cl- Cotransporters; | 2014 |
Syndrome of severe pain associated with a continuous bumetanide infusion.
Topics: Aged; Bumetanide; Diuretics; Female; Heart Failure; Humans; Hyperalgesia; Infusions, Intravenous; Mu | 2014 |
Acute spinal cord injury (SCI) transforms how GABA affects nociceptive sensitization.
Topics: Acetates; Analysis of Variance; Animals; Bicuculline; Bumetanide; Capsaicin; Disease Models, Animal; | 2016 |
The chloride co-transporters, NKCC1 and KCC2, in experimental autoimmune encephalomyelitis (EAE).
Topics: Animals; Bumetanide; Disease Progression; Encephalomyelitis, Autoimmune, Experimental; Female; Gangl | 2017 |
The role of cation-dependent chloride transporters in neuropathic pain following spinal cord injury.
Topics: Animals; Bumetanide; Hyperalgesia; K Cl- Cotransporters; Male; Pain; Rats; Rats, Sprague-Dawley; Sod | 2008 |
Role of NKCC1 and KCC2 in the development of chronic neuropathic pain following spinal cord injury.
Topics: Animals; Bumetanide; Chronic Disease; gamma-Aminobutyric Acid; Hyperalgesia; Inflammation; K Cl- Cot | 2010 |
Intrathecal administration of a gap junction decoupler, an inhibitor of Na(+)-K(+)-2Cl(-) cotransporter 1, or a GABA(A) receptor agonist attenuates mechanical pain hypersensitivity induced by REM sleep deprivation in the rat.
Topics: Animals; Behavior, Animal; Bumetanide; Carbenoxolone; Chloride Channels; GABA Agonists; Gap Junction | 2010 |
Stimulation of cutaneous low threshold mechanoreceptors in mice after intracolonic capsaicin increases spinal c-Fos labeling in an NKCC1-dependent fashion.
Topics: Abdomen; Animals; Behavior, Animal; Bumetanide; Capsaicin; Colon; Diuretics; Dose-Response Relations | 2013 |
Role of the Na+-K+-2Cl- cotransporter in the development of capsaicin-induced neurogenic inflammation.
Topics: Animals; Bumetanide; Capsaicin; Ganglia, Spinal; Hyperalgesia; Inflammation; Male; Rats; Rats, Sprag | 2006 |