bromodeoxyuridine has been researched along with Muscular Dystrophy in 3 studies
Timeframe | Studies, this research(%) | All Research% |
---|---|---|
pre-1990 | 0 (0.00) | 18.7374 |
1990's | 0 (0.00) | 18.2507 |
2000's | 2 (66.67) | 29.6817 |
2010's | 1 (33.33) | 24.3611 |
2020's | 0 (0.00) | 2.80 |
Authors | Studies |
---|---|
Aoki, Y; Nagata, T; Nakamura, A; Partridge, T; Takeda, S; Wood, MJ; Yokota, T | 1 |
Deng, B; Glanzman, D; Tidball, JG | 1 |
Eade, A; Hu, H; Qi, Y; Xiong, Y; Yang, Y | 1 |
3 other study(ies) available for bromodeoxyuridine and Muscular Dystrophy
Article | Year |
---|---|
Highly efficient in vivo delivery of PMO into regenerating myotubes and rescue in laminin-α2 chain-null congenital muscular dystrophy mice.
Topics: Alternative Splicing; Animals; Base Sequence; Bromodeoxyuridine; Cardiotoxins; Cell Line; Cell Membrane Permeability; Disease Models, Animal; Dystrophin; Exons; Gene Expression; Gene Order; Humans; Laminin; Mice; Mice, Knockout; Morpholinos; Muscle Fibers, Skeletal; Muscular Dystrophies; Muscular Dystrophy, Animal; Muscular Dystrophy, Duchenne; Regeneration | 2013 |
Nitric oxide generated by muscle corrects defects in hippocampal neurogenesis and neural differentiation caused by muscular dystrophy.
Topics: Animals; Antimetabolites; Apoptosis; Blotting, Western; Bromodeoxyuridine; Cell Differentiation; Cell Proliferation; Dystrophin; Hippocampus; Immunohistochemistry; Mice; Mice, Inbred mdx; Mice, Transgenic; Muscle, Skeletal; Muscular Dystrophies; Neurons; Nitric Oxide; Nitric Oxide Synthase Type I; Nitric Oxide Synthase Type II; Running | 2009 |
Breaches of the pial basement membrane and disappearance of the glia limitans during development underlie the cortical lamination defect in the mouse model of muscle-eye-brain disease.
Topics: Animals; Basement Membrane; Bromodeoxyuridine; Cell Movement; Cerebral Cortex; Disease Models, Animal; Embryonic Development; Green Fluorescent Proteins; Growth; Luminescent Proteins; Mice; Mice, Knockout; Mice, Transgenic; Muscular Dystrophies; Mutation; N-Acetylglucosaminyltransferases; Neuroglia; Neurons; Pia Mater | 2007 |