Page last updated: 2024-08-17

bromodeoxyuridine and Muscular Dystrophy

bromodeoxyuridine has been researched along with Muscular Dystrophy in 3 studies

Research

Studies (3)

TimeframeStudies, this research(%)All Research%
pre-19900 (0.00)18.7374
1990's0 (0.00)18.2507
2000's2 (66.67)29.6817
2010's1 (33.33)24.3611
2020's0 (0.00)2.80

Authors

AuthorsStudies
Aoki, Y; Nagata, T; Nakamura, A; Partridge, T; Takeda, S; Wood, MJ; Yokota, T1
Deng, B; Glanzman, D; Tidball, JG1
Eade, A; Hu, H; Qi, Y; Xiong, Y; Yang, Y1

Other Studies

3 other study(ies) available for bromodeoxyuridine and Muscular Dystrophy

ArticleYear
Highly efficient in vivo delivery of PMO into regenerating myotubes and rescue in laminin-α2 chain-null congenital muscular dystrophy mice.
    Human molecular genetics, 2013, Dec-15, Volume: 22, Issue:24

    Topics: Alternative Splicing; Animals; Base Sequence; Bromodeoxyuridine; Cardiotoxins; Cell Line; Cell Membrane Permeability; Disease Models, Animal; Dystrophin; Exons; Gene Expression; Gene Order; Humans; Laminin; Mice; Mice, Knockout; Morpholinos; Muscle Fibers, Skeletal; Muscular Dystrophies; Muscular Dystrophy, Animal; Muscular Dystrophy, Duchenne; Regeneration

2013
Nitric oxide generated by muscle corrects defects in hippocampal neurogenesis and neural differentiation caused by muscular dystrophy.
    The Journal of physiology, 2009, Apr-15, Volume: 587, Issue:Pt 8

    Topics: Animals; Antimetabolites; Apoptosis; Blotting, Western; Bromodeoxyuridine; Cell Differentiation; Cell Proliferation; Dystrophin; Hippocampus; Immunohistochemistry; Mice; Mice, Inbred mdx; Mice, Transgenic; Muscle, Skeletal; Muscular Dystrophies; Neurons; Nitric Oxide; Nitric Oxide Synthase Type I; Nitric Oxide Synthase Type II; Running

2009
Breaches of the pial basement membrane and disappearance of the glia limitans during development underlie the cortical lamination defect in the mouse model of muscle-eye-brain disease.
    The Journal of comparative neurology, 2007, Mar-01, Volume: 501, Issue:1

    Topics: Animals; Basement Membrane; Bromodeoxyuridine; Cell Movement; Cerebral Cortex; Disease Models, Animal; Embryonic Development; Green Fluorescent Proteins; Growth; Luminescent Proteins; Mice; Mice, Knockout; Mice, Transgenic; Muscular Dystrophies; Mutation; N-Acetylglucosaminyltransferases; Neuroglia; Neurons; Pia Mater

2007