bromodeoxyuridine has been researched along with Cardiac Remodeling, Ventricular in 2 studies
| Timeframe | Studies, this research(%) | All Research% |
|---|---|---|
| pre-1990 | 0 (0.00) | 18.7374 |
| 1990's | 0 (0.00) | 18.2507 |
| 2000's | 2 (100.00) | 29.6817 |
| 2010's | 0 (0.00) | 24.3611 |
| 2020's | 0 (0.00) | 2.80 |
| Authors | Studies |
|---|---|
| Esaki, M; Fujiwara, H; Fujiwara, T; Goto, K; Hayakawa, K; Kawase, Y; Koda, M; Kosai, K; Li, Y; Maruyama, R; Minatoguchi, S; Mizuguchi, H; Nagano, S; Okada, H; Takahashi, T; Takemura, G; Yuge, K | 1 |
| Ceddia, RB; Coe, IR; Li, RK; Sweeney, G; Tajmir, P | 1 |
2 other study(ies) available for bromodeoxyuridine and Cardiac Remodeling, Ventricular
| Article | Year |
|---|---|
Postinfarction treatment with an adenoviral vector expressing hepatocyte growth factor relieves chronic left ventricular remodeling and dysfunction in mice.
Topics: Adenoviridae; Animals; Bromodeoxyuridine; Cell Division; Chronic Disease; Disease Models, Animal; Disease Progression; Genetic Vectors; Hepatocyte Growth Factor; Hepatocytes; Humans; Immunohistochemistry; Male; Mice; Mice, Inbred C57BL; Myocardial Infarction; Myocardium; Survival Rate; Time Factors; Transduction, Genetic; Treatment Outcome; Ventricular Dysfunction, Left; Ventricular Remodeling | 2003 |
Leptin increases cardiomyocyte hyperplasia via extracellular signal-regulated kinase- and phosphatidylinositol 3-kinase-dependent signaling pathways.
Topics: Animals; Bromodeoxyuridine; Cell Line; Child, Preschool; Humans; Hyperplasia; Intracellular Membranes; Leptin; Mice; Mitogen-Activated Protein Kinases; Myocytes, Cardiac; Phosphatidylinositol 3-Kinases; Signal Transduction; Ventricular Remodeling | 2004 |