Page last updated: 2024-08-17

bisphenol a and Cirrhosis

bisphenol a has been researched along with Cirrhosis in 3 studies

Research

Studies (3)

TimeframeStudies, this research(%)All Research%
pre-19900 (0.00)18.7374
1990's0 (0.00)18.2507
2000's0 (0.00)29.6817
2010's3 (100.00)24.3611
2020's0 (0.00)2.80

Authors

AuthorsStudies
Belcher, SM; Gear, R; Kendziorski, JA1
Belcher, SM; Kendziorski, JA1
Fang, Z; Hou, L; Hu, Y; Ju, J; Li, J; Li, Q; Li, Z; Qin, W; Wu, X; Xu, C; Zhang, L; Zhang, Q; Zhang, Y; Zhou, T1

Other Studies

3 other study(ies) available for bisphenol a and Cirrhosis

ArticleYear
Effects of bisphenol A on incidence and severity of cardiac lesions in the NCTR-Sprague-Dawley rat: A CLARITY-BPA study.
    Toxicology letters, 2017, Jun-05, Volume: 275

    Topics: Age Factors; Animals; Benzhydryl Compounds; Body Weight; Cardiomyopathies; Cardiotoxicity; Environmental Pollutants; Female; Fibrosis; Male; Myocardium; Organ Size; Phenols; Rats, Sprague-Dawley; Severity of Illness Index; Sex Factors

2017
Strain-specific induction of endometrial periglandular fibrosis in mice exposed during adulthood to the endocrine disrupting chemical bisphenol A.
    Reproductive toxicology (Elmsford, N.Y.), 2015, Volume: 58

    Topics: Age Factors; Animals; Benzhydryl Compounds; Collagen Type I; Collagen Type III; Endocrine Disruptors; Endometriosis; Endometrium; Female; Fibrosis; Matrix Metalloproteinase 14; Matrix Metalloproteinase 2; Mice, Inbred C57BL; Phenols; Phenotype; Species Specificity

2015
Bisphenol A, an environmental estrogen-like toxic chemical, induces cardiac fibrosis by activating the ERK1/2 pathway.
    Toxicology letters, 2016, May-27, Volume: 250-251

    Topics: Animals; Benzhydryl Compounds; Cardiomyopathies; Cell Proliferation; Cells, Cultured; Collagen; Dose-Response Relationship, Drug; Endocrine Disruptors; Environmental Pollutants; Enzyme Activation; Estrogen Antagonists; Fibroblasts; Fibrosis; Mitogen-Activated Protein Kinase 1; Mitogen-Activated Protein Kinase 3; Myocardium; Phenols; Phosphorylation; Protein Kinase Inhibitors; Rats, Sprague-Dawley; Signal Transduction; Time Factors; Transforming Growth Factor beta1

2016