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bisindolylmaleimide i and Muscular Dystrophy, Duchenne

bisindolylmaleimide i has been researched along with Muscular Dystrophy, Duchenne in 1 studies

bisindolylmaleimide I: a bis(indolyl)maleimide

Muscular Dystrophy, Duchenne: An X-linked recessive muscle disease caused by an inability to synthesize DYSTROPHIN, which is involved with maintaining the integrity of the sarcolemma. Muscle fibers undergo a process that features degeneration and regeneration. Clinical manifestations include proximal weakness in the first few years of life, pseudohypertrophy, cardiomyopathy (see MYOCARDIAL DISEASES), and an increased incidence of impaired mentation. Becker muscular dystrophy is a closely related condition featuring a later onset of disease (usually adolescence) and a slowly progressive course. (Adams et al., Principles of Neurology, 6th ed, p1415)

Research

Studies (1)

TimeframeStudies, this research(%)All Research%
pre-19900 (0.00)18.7374
1990's0 (0.00)18.2507
2000's0 (0.00)29.6817
2010's1 (100.00)24.3611
2020's0 (0.00)2.80

Authors

AuthorsStudies
Harisseh, R1
Chatelier, A1
Magaud, C1
Déliot, N1
Constantin, B1

Other Studies

1 other study available for bisindolylmaleimide i and Muscular Dystrophy, Duchenne

ArticleYear
Involvement of TRPV2 and SOCE in calcium influx disorder in DMD primary human myotubes with a specific contribution of α1-syntrophin and PLC/PKC in SOCE regulation.
    American journal of physiology. Cell physiology, 2013, May-01, Volume: 304, Issue:9

    Topics: Calcium Channel Blockers; Calcium Channels; Calcium Signaling; Calcium-Binding Proteins; Cells, Cult

2013