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bepridil and Nerve Degeneration

bepridil has been researched along with Nerve Degeneration in 2 studies

Bepridil: A long-acting calcium-blocking agent with significant anti-anginal activity. The drug produces significant coronary vasodilation and modest peripheral effects. It has antihypertensive and selective anti-arrhythmia activities and acts as a calmodulin antagonist.
bepridil : A tertiary amine in which the substituents on nitrogen are benzyl, phenyl and 3-(2-methylpropoxy)-2-(pyrrolidin-1-yl)propyl.

Nerve Degeneration: Loss of functional activity and trophic degeneration of nerve axons and their terminal arborizations following the destruction of their cells of origin or interruption of their continuity with these cells. The pathology is characteristic of neurodegenerative diseases. Often the process of nerve degeneration is studied in research on neuroanatomical localization and correlation of the neurophysiology of neural pathways.

Research

Studies (2)

TimeframeStudies, this research(%)All Research%
pre-19900 (0.00)18.7374
1990's1 (50.00)18.2507
2000's1 (50.00)29.6817
2010's0 (0.00)24.3611
2020's0 (0.00)2.80

Authors

AuthorsStudies
Kapoor, R1
Davies, M1
Blaker, PA1
Hall, SM1
Smith, KJ1
George, EB1
Glass, JD1
Griffin, JW1

Other Studies

2 other studies available for bepridil and Nerve Degeneration

ArticleYear
Blockers of sodium and calcium entry protect axons from nitric oxide-mediated degeneration.
    Annals of neurology, 2003, Volume: 53, Issue:2

    Topics: Anesthetics, Local; Animals; Axons; Bepridil; Calcium; Calcium Channel Blockers; Electrophysiology;

2003
Axotomy-induced axonal degeneration is mediated by calcium influx through ion-specific channels.
    The Journal of neuroscience : the official journal of the Society for Neuroscience, 1995, Volume: 15, Issue:10

    Topics: Animals; Axons; Bepridil; Calcium; Calcium Channel Blockers; Calcium Channels; Calpain; Culture Tech

1995