barbital has been researched along with Precancerous Conditions in 3 studies
5,5-diethylbarbituric acid : A member of the class of barbiturates, the structure of which is that of barbituric acid substituted at C-5 by two ethyl groups. Formerly used as a hypnotic (sleeping aid).
Precancerous Conditions: Pathological conditions that tend eventually to become malignant.
Excerpt | Relevance | Reference |
---|---|---|
" Rats were dosed with sodium barbital in the feed at 0, 50, 250, 500, 1000, 2000 or 4000 p." | 1.31 | Promotion by sodium barbital induces early development but does not increase the multiplicity of hereditary renal tumors in Eker rats. ( David, CS; Donner, EM; Everitt, JI; Goldsworthy, TL; Harden, R; Janszen, DB; Wolf, DC, 2000) |
Timeframe | Studies, this research(%) | All Research% |
---|---|---|
pre-1990 | 0 (0.00) | 18.7374 |
1990's | 2 (66.67) | 18.2507 |
2000's | 1 (33.33) | 29.6817 |
2010's | 0 (0.00) | 24.3611 |
2020's | 0 (0.00) | 2.80 |
Authors | Studies |
---|---|
Kurata, Y | 1 |
Diwan, BA | 1 |
Uno, H | 1 |
Rice, JM | 1 |
Ward, JM | 1 |
Gerez, E | 1 |
Vazquez, E | 1 |
Caballero, F | 1 |
Polo, C | 1 |
Batlle, A | 1 |
Wolf, DC | 1 |
Goldsworthy, TL | 1 |
Janszen, DB | 1 |
Harden, R | 1 |
Donner, EM | 1 |
David, CS | 1 |
Everitt, JI | 1 |
3 other studies available for barbital and Precancerous Conditions
Article | Year |
---|---|
Pathology of preneoplastic and neoplastic renal tubular lesions induced in F-344 rats by sodium barbital, a nongenotoxic renal carcinogen and nephrotoxin.
Topics: Adenoma; Animals; Barbital; Carcinogens; Carcinoma, Renal Cell; Hyperplasia; Immunohistochemistry; K | 1993 |
Altered heme pathway regulation and drug metabolizing enzyme system in a mouse model of hepatocarcinogenesis: effect of veronal.
Topics: 5-Aminolevulinate Synthetase; Animals; Barbital; Catalase; Cytochrome P-450 Enzyme System; Glucuroni | 1997 |
Promotion by sodium barbital induces early development but does not increase the multiplicity of hereditary renal tumors in Eker rats.
Topics: Animals; Barbital; Cell Division; Cocarcinogenesis; Dose-Response Relationship, Drug; Genetic Predis | 2000 |