baicalin has been researched along with Fibrosis in 14 studies
| Timeframe | Studies, this research(%) | All Research% |
|---|---|---|
| pre-1990 | 0 (0.00) | 18.7374 |
| 1990's | 0 (0.00) | 18.2507 |
| 2000's | 0 (0.00) | 29.6817 |
| 2010's | 6 (42.86) | 24.3611 |
| 2020's | 8 (57.14) | 2.80 |
| Authors | Studies |
|---|---|
| El-Kashef, DH; Zaghloul, AM; Zaghloul, RA | 1 |
| Jiang, Q; Wang, H; Zhang, L | 1 |
| Li, L; Liu, Q; Shi, L; Wang, L; Wang, X; Wu, W; Wu, Z; Zhou, X | 1 |
| Hao, Y; Hu, H; Li, W; Liang, W; Peng, Z; Zou, Z | 1 |
| Alcalde-Estévez, E; Castillo, C; Castro, A; Costa, IG; Fernández, L; Herrero, JI; Jansen, J; Kramann, R; Lamas, S; Miguel, V; Nagai, J; Ranz, I; Reimer, KC; Rey-Serra, C; Rodríguez González-Moro, JM; Sancho, D; Sevilla, L; Sirera, B; Tituaña, J | 1 |
| Chen, G; Du, JW; Fan, XY; Feng, J; Jin, YL; Nie, Q; Zheng, XP | 1 |
| Liang, R; Wang, P; Xu, L; Yang, C; Zhang, S | 1 |
| He, SD; Huang, SG; Li, DY; Sun, CX; Tan, N; Zhu, HJ | 1 |
| Cai, Y; Li, X; Liu, F; Ma, W; Qiu, J; Wu, B; Xiao, Y; Zhang, G | 1 |
| Zhu, QJ | 1 |
| Cai, Y; Huang, B; Huang, J; Liu, X; Wu, B; Xiao, Y; Ye, J; Zhang, G; Zhou, Y; Zhu, L | 1 |
| Deng, Q; Lun, W; Wu, X; Zhang, W; Zhi, F | 1 |
| Mao, HX; Tan, YJ; Zhu, CL | 1 |
| Hu, C; Hu, M; Li, L; Lin, M; Rong, R; Sidikejiang, N; Wang, X; Zhang, C; Zheng, L | 1 |
1 review(s) available for baicalin and Fibrosis
| Article | Year |
|---|---|
[Research advances on baicalin and baicalein as potential therapeutic agents for fibrotic disease].
Topics: Fibrosis; Flavanones; Flavonoids; Humans | 2017 |
13 other study(ies) available for baicalin and Fibrosis
| Article | Year |
|---|---|
Hepatoprotective effect of Baicalin against thioacetamide-induced cirrhosis in rats: Targeting NOX4/NF-κB/NLRP3 inflammasome signaling pathways.
Topics: Animals; Antioxidants; Fibrosis; Flavonoids; Inflammasomes; Inflammation; Liver; Liver Cirrhosis; Male; NADPH Oxidase 4; NF-kappa B; NLR Family, Pyrin Domain-Containing 3 Protein; Oxidative Stress; Rats; Rats, Sprague-Dawley; Reactive Oxygen Species; Signal Transduction; Thioacetamide | 2022 |
Baicalin protects against renal interstitial fibrosis in mice by inhibiting the TGF-β/Smad signalling pathway.
Topics: Animals; Fibrosis; Flavonoids; Kidney; Kidney Diseases; Male; Mice; Mice, Inbred C57BL; Signal Transduction; Transforming Growth Factor beta; Transforming Growth Factor beta1; Ureteral Obstruction | 2022 |
Baicalin prevents fibrosis of human trabecular meshwork cells via inhibiting the MyD88/NF-κB pathway.
Topics: Blotting, Western; Cells, Cultured; Fibrosis; Glaucoma, Open-Angle; Humans; Myeloid Differentiation Factor 88; NF-kappa B; Toll-Like Receptor 4; Trabecular Meshwork; Transforming Growth Factor beta2 | 2023 |
Baicalin ameliorates renal fibrosis by upregulating CPT1α-mediated fatty acid oxidation in diabetic kidney disease.
Topics: Animals; Diabetes Mellitus; Diabetic Nephropathies; Fatty Acids; Fibrosis; Humans; Kidney; Mice; RNA, Messenger | 2024 |
Enhanced fatty acid oxidation through metformin and baicalin as therapy for COVID-19 and associated inflammatory states in lung and kidney.
Topics: AMP-Activated Protein Kinases; Animals; COVID-19; Fatty Acids; Fibrosis; Humans; Inflammation; Kidney; Lung; Metformin; Respiratory Distress Syndrome; Transforming Growth Factor beta | 2023 |
Kidney-targeted baicalin-lysozyme conjugate ameliorates renal fibrosis in rats with diabetic nephropathy induced by streptozotocin.
Topics: Animals; Anti-Inflammatory Agents, Non-Steroidal; Blood Glucose; Cholesterol; Diabetes Mellitus, Experimental; Diabetic Nephropathies; Drug Carriers; Drug Delivery Systems; Fibrosis; Flavonoids; Insulin; Kidney; Malondialdehyde; Muramidase; Rats; Triglycerides | 2020 |
Baicalin suppresses renal fibrosis through microRNA-124/TLR4/NF-κB axis in streptozotocin-induced diabetic nephropathy mice and high glucose-treated human proximal tubule epithelial cells.
Topics: Animals; Cell Line; Diabetic Nephropathies; Epithelial Cells; Fibrosis; Flavonoids; Humans; Kidney; Kidney Tubules, Proximal; Male; Mice; Mice, Inbred C57BL; MicroRNAs; NF-kappa B; Toll-Like Receptor 4 | 2020 |
Baicalin attenuates adriamycin-induced nephrotic syndrome by regulating fibrosis procession and inflammatory reaction.
Topics: Animals; Apoptosis; Disease Models, Animal; Doxorubicin; Epithelial-Mesenchymal Transition; Fibrosis; Flavonoids; Gene Expression Regulation; Humans; Inflammation; Kidney; Nephrotic Syndrome; Rats | 2021 |
High doses of baicalin induces kidney injury and fibrosis through regulating TGF-β/Smad signaling pathway.
Topics: Actins; Animals; Anti-Inflammatory Agents, Non-Steroidal; Antineoplastic Agents; Antiviral Agents; Connective Tissue Growth Factor; Fibronectins; Fibrosis; Flavonoids; Kidney; Kidney Diseases; Rats, Sprague-Dawley; Scutellaria baicalensis; Signal Transduction; Smad3 Protein; Transforming Growth Factor beta1 | 2017 |
Baicalin inhibits pressure overload-induced cardiac fibrosis through regulating AMPK/TGF-β/Smads signaling pathway.
Topics: Angiotensin II; Animals; Cells, Cultured; Collagen; Connective Tissue Growth Factor; Dose-Response Relationship, Drug; Fibronectins; Fibrosis; Flavonoids; Heart Diseases; Myocardium; Proto-Oncogene Proteins c-akt; Rats; Rats, Sprague-Dawley; Signal Transduction; Smad Proteins; Transforming Growth Factor beta | 2018 |
Baicalin inhibits PDGF-BB-induced hepatic stellate cell proliferation, apoptosis, invasion, migration and activation via the miR-3595/ACSL4 axis.
Topics: Animals; Apoptosis; Becaplermin; Cell Proliferation; Coenzyme A Ligases; Enzyme Inhibitors; Fibrosis; Flavonoids; Hepatic Stellate Cells; Liver; MicroRNAs; Plant Extracts; Protective Agents; Proto-Oncogene Proteins c-sis; Rats; Scutellaria baicalensis; Signal Transduction | 2018 |
[Therapeutic effect of baicalin in treatment of renal interstitial fibrosis in rats with unliateral ureteral obstruction and related mechanisms].
Topics: Animals; Fibrosis; Flavonoids; Immunohistochemistry; Kidney; Male; Rats; Rats, Sprague-Dawley; Receptor, Notch1; Transforming Growth Factor beta1; Ureteral Obstruction | 2016 |
Baicalin ameliorates renal fibrosis via inhibition of transforming growth factor β1 production and downstream signal transduction.
Topics: Animals; Disease Models, Animal; Epithelial-Mesenchymal Transition; Fibrosis; Flavonoids; Immunohistochemistry; Kidney; Kidney Diseases; Male; Models, Biological; Phosphorylation; Rats, Sprague-Dawley; Signal Transduction; Smad Proteins; Transforming Growth Factor beta1; Ureteral Obstruction | 2017 |