baicalin has been researched along with Blood Poisoning in 10 studies
| Timeframe | Studies, this research(%) | All Research% |
|---|---|---|
| pre-1990 | 0 (0.00) | 18.7374 |
| 1990's | 0 (0.00) | 18.2507 |
| 2000's | 0 (0.00) | 29.6817 |
| 2010's | 7 (70.00) | 24.3611 |
| 2020's | 3 (30.00) | 2.80 |
| Authors | Studies |
|---|---|
| Ma, J; Wang, R; Xu, A; Xu, R; Yan, H; Zhang, J | 1 |
| Liu, P; Long, C; Lu, X; Luo, L; Xiao, Z; Yan, H; Zhang, X; Zhu, Y | 1 |
| Fan, H; Le, J; Sun, M; Zhu, J | 1 |
| Chen, G; Dou, D; Guo, S; Xu, Y; Zhang, M; Zhang, X | 1 |
| Guo, M; He, XX; Wu, Q; Ye, H; Zheng, XB; Zhu, YZ | 1 |
| Liu, D; Wang, H | 1 |
| Hu, X; Miao, P; Yu, R; Zheng, H | 1 |
| Chen, GR; Dou, DQ; Jing, J; Li, MY; Mackie, B; Wang, J; Zhang, G; Zhang, X | 1 |
| Fu, Y; Lin, H; Zhu, Y | 1 |
| Bao, R; Bo, L; Chen, F; Deng, X; Fan, X; Li, J; Lou, J; Sheng, Y; Wang, F; Wang, J; Wu, Y; Zhu, J; Zou, Y | 1 |
10 other study(ies) available for baicalin and Blood Poisoning
| Article | Year |
|---|---|
Protective Effects of Baicalin on Lipopolysaccharide-Induced Injury in Caenorhabditis elegans.
Topics: Animals; Anti-Inflammatory Agents; Behavior, Animal; Caenorhabditis elegans; Disease Models, Animal; Flavonoids; Lipopolysaccharides; NF-kappa B; Oxidative Stress; Protective Agents; Proto-Oncogene Proteins c-bcl-2; Sepsis; Toll-Like Receptor 4 | 2020 |
Baicalin suppresses Th1 and Th17 responses and promotes Treg response to ameliorate sepsis-associated pancreatic injury via the RhoA-ROCK pathway.
Topics: Animals; Anti-Inflammatory Agents; Cell Differentiation; Disease Models, Animal; Flavonoids; Interferon-gamma; Interleukin-10; Interleukin-17; Male; Mice, Inbred C57BL; Pancreas; rho-Associated Kinases; Sepsis; Signal Transduction; STAT Transcription Factors; T-Lymphocytes, Regulatory; Th1 Cells; Th17 Cells | 2020 |
[Protective effect and mechanism of Baicalin on acute kidney injury in septic mice].
Topics: Acute Kidney Injury; Animals; Flavonoids; Kidney; Ligation; Mice; Sepsis | 2021 |
Evaluation of anti-sepsis activity by compounds with high affinity to lipid a from HuanglianJiedu decoction.
Topics: Animals; Berberine; Berberine Alkaloids; Cell Line; Drugs, Chinese Herbal; Flavonoids; Interleukin-6; Iridoids; Lipid A; Lipopolysaccharides; Mice; RAW 264.7 Cells; Sepsis; Toll-Like Receptor 4; Tumor Necrosis Factor-alpha | 2017 |
[Protective effect of baicalin against LPS-induced intestinal injury].
Topics: Animals; Antioxidants; Drugs, Chinese Herbal; Flavonoids; Glutathione Peroxidase; Humans; Ileum; Intestinal Mucosa; Lipopolysaccharides; Male; Mice; Mice, Inbred BALB C; Protective Agents; Sepsis; Superoxide Dismutase | 2013 |
Baicalin inhibits high-mobility group box 1 release and improves survival in experimental sepsis.
Topics: Animals; Anti-Inflammatory Agents, Non-Steroidal; Cell Proliferation; Cells, Cultured; Cytokines; Dose-Response Relationship, Drug; Drug Evaluation, Preclinical; Flavonoids; HMGB1 Protein; Inflammation Mediators; Lipopolysaccharides; Liver; Lung; Macrophages; Male; Mice; Mice, Inbred BALB C; Sepsis; Survival Analysis; Translocation, Genetic | 2014 |
The immunoprotective activity of baicalin in mouse model of cecal ligation and puncture-induced sepsis.
Topics: Animals; Anti-Inflammatory Agents, Non-Steroidal; Flavonoids; Male; Mice; Mice, Inbred BALB C; Sepsis; Spleen; Thymus Gland | 2015 |
The effective components of Huanglian Jiedu Decoction against sepsis evaluated by a lipid A-based affinity biosensor.
Topics: Animals; Berberine; Biosensing Techniques; Drugs, Chinese Herbal; Female; Flavonoids; Interleukin-6; Iridoids; Kidney; Lipid A; Lipopolysaccharides; Liver; Lung; Male; Mice, Inbred BALB C; Myocardium; Sepsis; Tumor Necrosis Factor-alpha | 2016 |
Baicalin Inhibits Renal Cell Apoptosis and Protects Against Acute Kidney Injury in Pediatric Sepsis.
Topics: Acute Kidney Injury; Animals; Apoptosis; bcl-2-Associated X Protein; Cecum; Child; Child, Preschool; Disease Models, Animal; Flavonoids; Humans; Infant; Kidney; Kidney Function Tests; Ligation; Male; Mice, Inbred C57BL; Punctures; Sepsis | 2016 |
Baicalin improves survival in a murine model of polymicrobial sepsis via suppressing inflammatory response and lymphocyte apoptosis.
Topics: Adaptive Immunity; Animals; Anti-Inflammatory Agents, Non-Steroidal; Apoptosis; CD8-Positive T-Lymphocytes; Cytokines; Dendritic Cells; Disease Models, Animal; Flavonoids; Lymphocyte Count; Male; Mice; Sepsis; T-Lymphocytes, Regulatory | 2012 |