bafilomycin-a1 and Carcinogenesis

bafilomycin-a1 has been researched along with Carcinogenesis* in 2 studies

Other Studies

2 other study(ies) available for bafilomycin-a1 and Carcinogenesis

Article	Year
Excess hepsin proteolytic activity limits oncogenic signaling and induces ER stress and autophagy in prostate cancer cells. Cell death & disease, 2019, 08-09, Volume: 10, Issue:8 Topics: Animals; Autophagy; Carcinogenesis; Cinnamates; Endoplasmic Reticulum Stress; Endoplasmic Reticulum-Associated Degradation; Gene Expression Regulation, Neoplastic; Heterografts; Humans; Macrolides; Male; Mice; Microtubule-Associated Proteins; Neoplastic Stem Cells; PC-3 Cells; Prostatic Neoplasms; Sequestosome-1 Protein; Serine Endopeptidases; Thiourea	2019
Methods for Monitoring Macroautophagy in Pancreatic Cancer Cells. Methods in molecular biology (Clifton, N.J.), 2019, Volume: 1882 Macroautophagy is a catabolic process through which redundant, aged, or damaged cellular structures are first enclosed within double-membrane vesicles (called autophagosomes), and thereafter degraded within lysosomes. Macroautophagy provides a primary route for the turnover of macromolecules, membranes and organelles, and as such plays a major role in cell homeostasis. As part of the stress response, autophagy is crucial to determine the cell fate in response to extracellular or intracellular injuries. Autophagy is involved in cancerogenesis and in cancer progression. Here we illustrate the essential methods for monitoring autophagy in pancreatic cancer cells. Topics: Animals; Autophagosomes; Autophagy; Autophagy-Related Proteins; Carcinogenesis; Cell Culture Techniques; Cell Line, Tumor; Chloroquine; Disease Progression; Electrophoresis, Polyacrylamide Gel; Fluorescent Dyes; Humans; Immunoblotting; Lysosomes; Macrolides; Mice; Microscopy, Fluorescence; Pancreas; Pancreatic Neoplasms	2019

Article

Year

Excess hepsin proteolytic activity limits oncogenic signaling and induces ER stress and autophagy in prostate cancer cells.

Cell death & disease, 2019, 08-09, Volume: 10, Issue:8

Topics: Animals; Autophagy; Carcinogenesis; Cinnamates; Endoplasmic Reticulum Stress; Endoplasmic Reticulum-Associated Degradation; Gene Expression Regulation, Neoplastic; Heterografts; Humans; Macrolides; Male; Mice; Microtubule-Associated Proteins; Neoplastic Stem Cells; PC-3 Cells; Prostatic Neoplasms; Sequestosome-1 Protein; Serine Endopeptidases; Thiourea

2019

Methods for Monitoring Macroautophagy in Pancreatic Cancer Cells.

Methods in molecular biology (Clifton, N.J.), 2019, Volume: 1882

Macroautophagy is a catabolic process through which redundant, aged, or damaged cellular structures are first enclosed within double-membrane vesicles (called autophagosomes), and thereafter degraded within lysosomes. Macroautophagy provides a primary route for the turnover of macromolecules, membranes and organelles, and as such plays a major role in cell homeostasis. As part of the stress response, autophagy is crucial to determine the cell fate in response to extracellular or intracellular injuries. Autophagy is involved in cancerogenesis and in cancer progression. Here we illustrate the essential methods for monitoring autophagy in pancreatic cancer cells.

Topics: Animals; Autophagosomes; Autophagy; Autophagy-Related Proteins; Carcinogenesis; Cell Culture Techniques; Cell Line, Tumor; Chloroquine; Disease Progression; Electrophoresis, Polyacrylamide Gel; Fluorescent Dyes; Humans; Immunoblotting; Lysosomes; Macrolides; Mice; Microscopy, Fluorescence; Pancreas; Pancreatic Neoplasms

2019