azacitidine and Pulmonary Arterial Remodeling

azacitidine has been researched along with Pulmonary Arterial Remodeling in 2 studies

Research

Studies (2)

Timeframe	Studies, this research(%)	All Research%
pre-1990	0 (0.00)	18.7374
1990's	0 (0.00)	18.2507
2000's	0 (0.00)	29.6817
2010's	1 (50.00)	24.3611
2020's	1 (50.00)	2.80

Authors

Authors	Studies
Dubner, AM; Enyart, BT; Jolly, AJ; Koch, KA; LaBarbera, DV; Li, L; Lu, S; Moulton, KS; Mutryn, MF; Nemenoff, RA; Strand, KA; Weiser-Evans, MCM; Zhou, Q	1
Li, H; Luan, P; Peng, W; Wang, K; Xu, Y; Zhang, P; Zhuang, J	1

Other Studies

2 other study(ies) available for azacitidine and Pulmonary Arterial Remodeling

Article	Year
High Throughput Screen Identifies the DNMT1 (DNA Methyltransferase-1) Inhibitor, 5-Azacytidine, as a Potent Inducer of PTEN (Phosphatase and Tensin Homolog): Central Role for PTEN in 5-Azacytidine Protection Against Pathological Vascular Remodeling. Arteriosclerosis, thrombosis, and vascular biology, 2020, Volume: 40, Issue:8 Topics: Animals; Azacitidine; DNA (Cytosine-5-)-Methyltransferase 1; High-Throughput Screening Assays; Mice; Mice, Inbred C57BL; Myocytes, Smooth Muscle; Platelet-Derived Growth Factor; Promoter Regions, Genetic; PTEN Phosphohydrolase; Vascular Remodeling	2020
The Yin-Yang Dynamics of DNA Methylation Is the Key Regulator for Smooth Muscle Cell Phenotype Switch and Vascular Remodeling. Arteriosclerosis, thrombosis, and vascular biology, 2017, Volume: 37, Issue:1 Topics: 5-Methylcytosine; Animals; Aorta; Aortic Diseases; Apolipoproteins E; Atherosclerosis; Azacitidine; Cell Dedifferentiation; Cell Movement; Cell Proliferation; Cells, Cultured; Decitabine; Diet, High-Fat; Dioxygenases; Disease Models, Animal; DNA (Cytosine-5-)-Methyltransferase 1; DNA (Cytosine-5-)-Methyltransferases; DNA Methylation; DNA-Binding Proteins; Enzyme Inhibitors; Epigenesis, Genetic; Genetic Predisposition to Disease; Mice, Knockout; Muscle, Smooth, Vascular; Myocytes, Smooth Muscle; Nuclear Proteins; Phenotype; Promoter Regions, Genetic; Proto-Oncogene Proteins; Rats; Signal Transduction; Time Factors; Trans-Activators; Vascular Remodeling	2017

Article

Year

High Throughput Screen Identifies the DNMT1 (DNA Methyltransferase-1) Inhibitor, 5-Azacytidine, as a Potent Inducer of PTEN (Phosphatase and Tensin Homolog): Central Role for PTEN in 5-Azacytidine Protection Against Pathological Vascular Remodeling.

Arteriosclerosis, thrombosis, and vascular biology, 2020, Volume: 40, Issue:8

Topics: Animals; Azacitidine; DNA (Cytosine-5-)-Methyltransferase 1; High-Throughput Screening Assays; Mice; Mice, Inbred C57BL; Myocytes, Smooth Muscle; Platelet-Derived Growth Factor; Promoter Regions, Genetic; PTEN Phosphohydrolase; Vascular Remodeling

2020

The Yin-Yang Dynamics of DNA Methylation Is the Key Regulator for Smooth Muscle Cell Phenotype Switch and Vascular Remodeling.

Arteriosclerosis, thrombosis, and vascular biology, 2017, Volume: 37, Issue:1

Topics: 5-Methylcytosine; Animals; Aorta; Aortic Diseases; Apolipoproteins E; Atherosclerosis; Azacitidine; Cell Dedifferentiation; Cell Movement; Cell Proliferation; Cells, Cultured; Decitabine; Diet, High-Fat; Dioxygenases; Disease Models, Animal; DNA (Cytosine-5-)-Methyltransferase 1; DNA (Cytosine-5-)-Methyltransferases; DNA Methylation; DNA-Binding Proteins; Enzyme Inhibitors; Epigenesis, Genetic; Genetic Predisposition to Disease; Mice, Knockout; Muscle, Smooth, Vascular; Myocytes, Smooth Muscle; Nuclear Proteins; Phenotype; Promoter Regions, Genetic; Proto-Oncogene Proteins; Rats; Signal Transduction; Time Factors; Trans-Activators; Vascular Remodeling

2017