azacitidine and Cancer of Esophagus

azacitidine has been researched along with Cancer of Esophagus in 71 studies

Research

Studies (71)

TimeframeStudies, this research(%)All Research%
pre-19900 (0.00)18.7374
1990's1 (1.41)18.2507
2000's28 (39.44)29.6817
2010's41 (57.75)24.3611
2020's1 (1.41)2.80

Authors

AuthorsStudies
Cai, HQ; Cai, Y; Chang, C; Chen, DX; Guo, J; Hao, JJ; Wang, JJ; Wang, MR; Wei, WQ; Yang, LY; Zhang, Y1
Li, JR; Li, SY; Sun, LL; Yang, SM; Yu, HB1
Chen, C; Liu, X; Ma, Z; Yu, Z; Zhang, H1
Bai, G; Bao, Y; Chu, J; Eli, M; Wen, H1
da Costa, NM; de Almeida Simão, T; Gonzaga, IM; Herceg, Z; Hernandez-Vargas, H; Nicolau, MC; Nicolau-Neto, P; Ribeiro Pinto, LF; Soares Lima, SC1
Ding, C; Dong, Z; Guo, W; Guo, Y; Liang, J; Liu, S; Shan, B; Shen, S1
He, J; Li, Y; Luo, M; Shi, X; Sun, N; Yang, W; Zhou, F1
Baba, H; Baba, Y; Eto, K; Hiyoshi, Y; Ishimoto, T; Iwagami, S; Iwatsuki, M; Kiyozumi, Y; Miyamoto, Y; Ogata, Y; Okadome, K; Watanabe, M; Yagi, T; Yoshida, N1
Bultmann, I; Conradi, A; Kretschmer, C; Sterner-Kock, A1
Cheng, Y; Dong, M; Fan, X; Feng, X; Jin, Z; Meltzer, SJ; Mori, Y; Wang, L; Zhang, X; Zhao, Z1
Cao, Z; Chen, S; Cheng, Y; Dong, M; Fan, X; Feng, X; Gao, Y; Jin, Z; Liu, J; Meltzer, SJ; Mori, Y; Wang, L; Yu, H; Zhang, X; Zhang, Y; Zhao, Z1
Akhtar, J; Chen, HX; Liu, H; Wang, S; Wang, Z1
Chen, X; Dong, W; Fan, Q; Huang, L; Li, F; Liu, J; Wang, F; Wang, L; Wang, M; Yue, D; Zhang, B; Zhang, Y; Zhang, Z1
Ling, Y; Liu, Y; Lu, M; Xu, Y; Zhang, C; Zhou, T; Zhu, C; Zhu, J1
Chen, HX; Liu, H; Wang, S; Wang, Z1
Cui, L; Dong, Z; Guo, W; Guo, Y; Kuang, G; Shen, S; Wang, C1
Dong, Z; Guo, W; Guo, Y; Kuang, G; Shen, S; Yang, Z; Zhang, M1
Cao, Z; Chen, S; Cheng, Y; Dong, M; Fan, X; Feng, X; Gao, Y; Jin, Z; Liu, J; Meltzer, SJ; Mori, Y; Wang, L; Wu, W; Yu, H; Zhang, X; Zhao, Z1
Hou, SY; Liu, WH; Sang, MX; Shan, BE; Zhang, C1
Dong, Z; Guo, W; Guo, X; Guo, Y; Kuang, G; Shen, S; Wang, C1
Ahrens, TD; Boerries, M; Busch, H; Follo, M; Hembach, S; Hoeppner, J; Hopt, UT; Lassmann, S; Ostendorp, J; Timme, S; Werner, M1
Dong, Z; Guo, W; Guo, X; Guo, Y; Kuang, G; Liang, J; Liu, Y; Shen, S1
Li, X; Liao, J; Liu, R; Pan, E; Pu, Y; Wang, Y; Yang, M; Yin, L1
Ahrens, TD; Bogatyreva, L; Hauschke, D; Hoeppner, J; Hopt, UT; Lassmann, S; Ostendorp, J; Timme, S; Werner, M1
Cao, Y; Chen, Y; Huang, Y; Li, G; Liu, Z1
Bao, Y; Chen, Z; Dong, H; Guo, Y; Li, K; Shen, K; Wang, Q; Yang, W; Yang, Y; Zhang, H1
Chen, J; Gao, J; Gu, X; Liu, T; Lü, L; Mei, Z; Zeng, H1
Baba, H; Baba, Y; Harada, K; Ishimoto, T; Iwatsuki, M; Kiyozumi, Y; Kosumi, K; Kurashige, J; Miyake, K; Nakamura, K; Nakao, M; Ohuchi, M; Sakamoto, Y; Shigaki, H; Watanabe, M; Yoshida, N1
He, H; He, Y; Jiang, D; Li, W; Ma, H; Niu, Z; Wu, D; Xie, X; Zuo, X1
Duan, Y; Han, X; Jia, Y; Liu, L; Lv, W; Ma, M; Wang, J; Wang, X; Wang, Y1
Arai, M; Imazeki, F; Mikata, R; Ochiai, T; Sakai, Y; Seki, N; Shimada, H; Tada, M; Yokosuka, O1
Chen, H; El-Rifai, W; Peng, DF; Razvi, M; Roessner, A; Schneider-Stock, R; Washington, K1
Abraham, JM; Agarwal, R; Beer, DG; Cheng, Y; David, S; Hamilton, JP; Ito, T; Jin, Z; Kan, T; Meltzer, SJ; Mori, Y; Olaru, A; Paun, B; Sato, F; Selaru, FM; Shimada, Y; Yang, J1
Bonazzi, VF; Hayward, NK; Irwin, D1
Drew, PA; Jones, ME; Smith, E1
Claesson, MH; Krishnadath, KK; Milano, F; Pedersen, AW; Weinert, BT; Zocca, MB1
He, Y; Li, P; Wang, J; Wang, Y; Zhang, S; Zhu, S1
Li, P; Meng, XY; Wang, YJ; Zhang, ST; Zhu, ST; Zong, Y1
Dong, ZM; Fang, XH; Ge, MH; Hu, FJ; Li, P; Ling, ZQ; Mao, WM; Zhao, X1
Jiao, Y; Li, P; Meng, Y; Wang, JX; Wang, QG; Zhang, ST; Zhu, ST1
El-Rifai, W; Hu, TL; Jiang, A; Moskaluk, CA; Peng, D; Schneider-Stock, R; Washington, MK1
He, YL; Wang, JX; Yang, S; Zhang, ST; Zhu, ST1
Deng, X; Jing, Z; Li, G; Wu, SX; Zou, CL1
Guo, LP; Li, LW; Li, XY; Lu, SX; Yu, XY; Zhou, Y1
Cui, YT; Dong, SW; He, Z; Liang, SJ; Liu, YM; Sun, P; Wang, S; Wang, YG; Zhang, P1
Cui, YT; Dong, SW; He, Z; Liang, DC; Liang, SJ; Liu, YM; Wang, S; Wang, YG; Zhang, P; Zhong, RR1
Li, P; Meng, XY; Wang, YJ; Zhang, ST; Zhou, QZ; Zhu, ST1
Chen, XY; Guo, MZ; He, QY1
Bartram, CR; Imamura, M; Imoto, I; Inazawa, J; Inoue, J; Janssen, JW; Shimada, Y; Ueda, M1
Hoque, MO; Meltzer, SJ; Mori, M; Osada, M; Sato, F; Sidransky, D; Upadhyay, S; Xiao, Y; Yamashita, K1
Amagasa, T; Chin, K; Gray, JW; Hirohashi, S; Imamura, M; Imoto, I; Inazawa, J; Inoue, J; Shibata, T; Shimada, Y; Sonoda, I1
Beer, DG; Chen, GA; Fischette, MR; Guo, ZS; Hong, JA; Kassis, ES; Lee, S; Nguyen, DM; Schrump, DS; Steiner, FA; Trepel, JB; Weiser, TS1
Chen, D; Christman, JK; Fang, MZ; Jin, Z; Sun, Y; Yang, CS1
Chan, AT; Langford, C; Li, H; Murray, P; Putti, T; Seng, TJ; Srivastava, G; Tao, Q; Tsao, SW; Ying, J1
Ding, F; Guo, M; Li, J; Li, W; Liu, Z; Wang, Y; Wu, M; Yu, Z; Zhan, Q; Zhang, L1
Benhattar, J; Bosman, FT; Braunschweig, R; Clément, G; Pasquier, N1
Chen, GA; Figg, WD; Fischette, MR; Hancox, A; Hong, JA; Kunst, TF; Li, X; Murgo, AJ; Nguyen, DM; Pishchik, V; Schrump, DS; Steinberg, SM; Zhao, M1
Abraham, JM; Cheng, Y; Greenwald, BD; Hamilton, JP; Ito, T; Jin, Z; Kan, T; Meltzer, SJ; Mori, Y; Paun, BC; Sato, F; Wang, S; Yang, J1
Chan, AT; Chan, SL; Cui, Y; Jin, H; Lee, KY; Li, H; Ng, KM; Rha, SY; Robertson, KD; Srivastava, G; Tao, Q; Tsao, GS; van Hasselt, A; Wang, Y; Zhong, S1
Asuncion, L; Csiszar, K; Dietzsch, E; Fong, KS; Fong, SF; He, Q; Hollosi, P; Parker, MI1
Aiko, S; Imoto, I; Inazawa, J; Inoue, J; Iwai, T; Kawano, T; Kozaki, K; Shimada, Y; Tanaka, K; Tsuda, H; Yoshizumi, Y1
Abraham, JM; Agarwal, R; Beer, D; Cheng, Y; David, S; Hamilton, JP; Ito, T; Jin, Z; Kan, T; Meltzer, SJ; Montgomery, E; Mori, Y; Olaru, A; Paun, B; Sato, F; Yang, J1
Li, K; Li, Z; Teng, L; Wei, L; Wu, K; Yu, P; Zhang, C; Zhu, J1
Agarwal, R; Hamilton, JP; Ito, T; Jin, Z; Kan, T; Meltzer, SJ; Mori, Y; Olaru, A; Sato, F; Yang, J1
Hu, J; Long, C; Lu, Q; Yang, Y; Yin, B; Yu, F; Yuan, Y; Zhou, X1
Fukuyoshi, Y; Inoue, H; Ishii, H; Kita, Y; Mimori, K; Mori, M; Motoyama, K; Ohta, M; Yamashita, K1
Fitzgerald, RC; Gjerset, RA; Huang, Y; Peters, CJ1
Harada, H; Hatooka, S; Imamura, M; Ishizaki, K; Shimada, Y; Shinoda, M; Tanaka, H1
Chen, GA; Fischette, MR; Guo, ZS; Hong, JA; Nguyen, DM; Ohnmacht, GA; Schrump, DS; Weiser, TS1
Liao, J; Nie, Y; So, C; Song, Y; Wang, LD; Yang, CS; Yang, G; Zhao, X1
Cheung, AL; Lam, KY; Liu, Y; Shen, ZY; Si, HX; Srivastava, G; Tsao, SW; Wong, YC1

Trials

1 trial(s) available for azacitidine and Cancer of Esophagus

ArticleYear
Phase I study of decitabine-mediated gene expression in patients with cancers involving the lungs, esophagus, or pleura.
    Clinical cancer research : an official journal of the American Association for Cancer Research, 2006, Oct-01, Volume: 12, Issue:19

    Topics: Adult; Aged; Antigens, Neoplasm; Antimetabolites, Antineoplastic; Azacitidine; Carcinoma, Non-Small-Cell Lung; Carcinoma, Squamous Cell; Decitabine; DNA Modification Methylases; Esophageal Neoplasms; Female; Gene Expression Regulation, Neoplastic; Genes, p16; Humans; Lung Neoplasms; Male; Maximum Tolerated Dose; Membrane Proteins; Mesothelioma; Middle Aged; Neoplasm Proteins; Pleural Neoplasms; Transcriptional Activation

2006

Other Studies

70 other study(ies) available for azacitidine and Cancer of Esophagus

ArticleYear
Dysregulation of CXCL14 promotes malignant phenotypes of esophageal squamous carcinoma cells via regulating SRC and EGFR signaling.
    Biochemical and biophysical research communications, 2022, 06-18, Volume: 609

    Topics: Azacitidine; Cell Line, Tumor; Cell Proliferation; Chemokines, CXC; DNA Methylation; ErbB Receptors; Esophageal Neoplasms; Esophageal Squamous Cell Carcinoma; Gene Expression Regulation, Neoplastic; Humans; Phenotype

2022
Repression of DOK7 mediated by DNMT3A promotes the proliferation and invasion of KYSE410 and TE-12 ESCC cells.
    Biomedicine & pharmacotherapy = Biomedecine & pharmacotherapie, 2017, Volume: 90

    Topics: Azacitidine; Carcinoma, Squamous Cell; Cell Line, Tumor; Cell Proliferation; Decitabine; DNA (Cytosine-5-)-Methyltransferases; DNA Methylation; DNA Methyltransferase 3A; Epigenesis, Genetic; Esophageal Neoplasms; Esophageal Squamous Cell Carcinoma; Gene Expression Regulation, Neoplastic; Gene Silencing; Humans; Muscle Proteins; Neoplasm Invasiveness; Promoter Regions, Genetic

2017
Krüppel-Like Factor 4 Enhances Sensitivity of Cisplatin to Esophageal Squamous Cell Carcinoma (ESCC) Cells.
    Medical science monitor : international medical journal of experimental and clinical research, 2017, Jul-11, Volume: 23

    Topics: Antineoplastic Agents; Apoptosis; Azacitidine; Carcinoma, Squamous Cell; Cell Cycle Checkpoints; Cell Line, Tumor; Cisplatin; DNA Methylation; Drug Resistance, Neoplasm; Esophageal Neoplasms; Esophageal Squamous Cell Carcinoma; Gene Silencing; Humans; Kruppel-Like Factor 4; Kruppel-Like Transcription Factors; Promoter Regions, Genetic

2017
PAQR3 overexpression suppresses the aggressive phenotype of esophageal squamous cell carcinoma cells via inhibition of ERK signaling.
    Biomedicine & pharmacotherapy = Biomedecine & pharmacotherapie, 2017, Volume: 94

    Topics: Animals; Antimetabolites, Antineoplastic; Azacitidine; Carcinoma, Squamous Cell; Cell Line, Tumor; Cell Proliferation; Decitabine; Esophageal Neoplasms; Esophageal Squamous Cell Carcinoma; Female; Gene Expression Regulation, Neoplastic; Humans; Intracellular Signaling Peptides and Proteins; Male; MAP Kinase Signaling System; Membrane Proteins; Mice; Mice, Nude; Middle Aged; Neoplasm Staging; Phenotype; RNA, Messenger; Signal Transduction

2017
    Clinical epigenetics, 2017, Volume: 9

    Topics: Adult; Aged; Azacitidine; Biomarkers, Tumor; Carcinoma, Squamous Cell; Cell Line, Tumor; Decitabine; DNA Methylation; Down-Regulation; Early Detection of Cancer; Epigenesis, Genetic; Esophageal Neoplasms; Esophageal Squamous Cell Carcinoma; Female; Gene Expression Regulation, Neoplastic; Humans; Male; Middle Aged; Promoter Regions, Genetic; Trefoil Factor-1

2017
Aberrant methylation-mediated silencing of lncRNA CTC-276P9.1 is associated with malignant progression of esophageal squamous cell carcinoma.
    Clinical & experimental metastasis, 2018, Volume: 35, Issue:1-2

    Topics: Adult; Aged; Azacitidine; Biomarkers, Tumor; Cell Line, Tumor; Cell Proliferation; CpG Islands; Disease Progression; DNA Methylation; Epithelial-Mesenchymal Transition; Esophageal Neoplasms; Esophageal Squamous Cell Carcinoma; Female; Gene Silencing; Humans; Luciferases; Male; Middle Aged; Neoplasm Invasiveness; Neoplasm Metastasis; Promoter Regions, Genetic; RNA, Long Noncoding; Sp1 Transcription Factor; Survival Analysis; Up-Regulation

2018
Aberrant methylation of EYA4 promotes epithelial-mesenchymal transition in esophageal squamous cell carcinoma.
    Cancer science, 2018, Volume: 109, Issue:6

    Topics: Animals; Azacitidine; Carcinoma, Squamous Cell; Cell Line, Tumor; Cell Movement; DNA Methylation; Enzyme Inhibitors; Epithelial-Mesenchymal Transition; Esophageal Neoplasms; Female; Gene Expression Regulation, Neoplastic; Humans; Male; Mice, Nude; Middle Aged; RNA Interference; Trans-Activators; Transplantation, Heterologous

2018
Indoleamine 2, 3-dioxygenase 1 promoter hypomethylation is associated with poor prognosis in patients with esophageal cancer.
    Cancer science, 2019, Volume: 110, Issue:6

    Topics: Aged; Azacitidine; Cell Line, Tumor; DNA Methylation; Enzyme Inhibitors; Esophageal Neoplasms; Female; Gene Expression Regulation, Neoplastic; Humans; Indoleamine-Pyrrole 2,3,-Dioxygenase; Male; Middle Aged; Prognosis; Promoter Regions, Genetic

2019
Latent transforming growth factor β-binding protein 4 is downregulated in esophageal cancer via promoter methylation.
    PloS one, 2013, Volume: 8, Issue:5

    Topics: Azacitidine; Base Sequence; Binding Sites; Carcinoma; Cell Line, Tumor; Cell Movement; Decitabine; Disease Progression; DNA Methylation; Down-Regulation; Esophageal Neoplasms; Gene Expression Regulation, Neoplastic; Gene Order; Humans; Latent TGF-beta Binding Proteins; Neoplasm Staging; Promoter Regions, Genetic; Protein Binding; Transcription Factors; Transforming Growth Factor beta1

2013
Endoglin promoter hypermethylation identifies a field defect in human primary esophageal cancer.
    Cancer, 2013, Oct-15, Volume: 119, Issue:20

    Topics: Adenocarcinoma; Aged; Antigens, CD; Antimetabolites, Antineoplastic; Azacitidine; Biomarkers, Tumor; Carcinoma, Squamous Cell; Case-Control Studies; Decitabine; DNA Methylation; Endoglin; Esophageal Neoplasms; Esophagus; Female; Follow-Up Studies; Humans; Middle Aged; Neoplasm Staging; Prognosis; Promoter Regions, Genetic; Real-Time Polymerase Chain Reaction; Receptors, Cell Surface; Reverse Transcriptase Polymerase Chain Reaction; RNA, Messenger; ROC Curve; Tumor Cells, Cultured

2013
MAL hypermethylation is a tissue-specific event that correlates with MAL mRNA expression in esophageal carcinoma.
    Scientific reports, 2013, Oct-03, Volume: 3

    Topics: Adenocarcinoma; Aged; Antimetabolites, Antineoplastic; Azacitidine; Barrett Esophagus; Carcinoma, Squamous Cell; Case-Control Studies; Decitabine; DNA Methylation; Esophageal Neoplasms; Esophagus; Female; Gene Expression Regulation, Neoplastic; Humans; Male; Middle Aged; Myelin and Lymphocyte-Associated Proteolipid Proteins; Neoplasm Grading; Prognosis; Promoter Regions, Genetic; Real-Time Polymerase Chain Reaction; Reverse Transcriptase Polymerase Chain Reaction; RNA, Messenger; ROC Curve; Tumor Cells, Cultured

2013
Alteration of runt-related transcription factor 3 gene expression and biologic behavior of esophageal carcinoma TE-1 cells after 5-azacytidine intervention.
    Asian Pacific journal of cancer prevention : APJCP, 2013, Volume: 14, Issue:9

    Topics: Animals; Antimetabolites, Antineoplastic; Azacitidine; Blotting, Western; Carcinoma, Squamous Cell; Cell Adhesion; Cell Movement; Cell Proliferation; Core Binding Factor Alpha 3 Subunit; DNA Methylation; DNA, Neoplasm; Esophageal Neoplasms; Female; Gene Expression Regulation, Neoplastic; Humans; Male; Mice; Mice, Inbred BALB C; Mice, Nude; Neoplasm Invasiveness; Promoter Regions, Genetic; Reverse Transcriptase Polymerase Chain Reaction; Tumor Cells, Cultured; Xenograft Model Antitumor Assays

2013
Epigenetic inactivation of SPINT2 is associated with tumor suppressive function in esophageal squamous cell carcinoma.
    Experimental cell research, 2014, Mar-10, Volume: 322, Issue:1

    Topics: Adult; Aged; Aged, 80 and over; Azacitidine; Carcinoma, Squamous Cell; DNA Methylation; Esophageal Neoplasms; Esophageal Squamous Cell Carcinoma; Female; Gene Expression Regulation, Neoplastic; Gene Silencing; Genes, Tumor Suppressor; Humans; Male; Membrane Glycoproteins; Middle Aged; Promoter Regions, Genetic; Tumor Cells, Cultured

2014
Promoter methylation‑associated silencing of p27kip1 gene with metastasis in esophageal squamous cell carcinoma.
    Molecular medicine reports, 2014, Volume: 9, Issue:3

    Topics: Antimetabolites, Antineoplastic; Azacitidine; Carcinoma, Squamous Cell; Cell Line, Tumor; Cyclin-Dependent Kinase Inhibitor p27; Decitabine; DNA Methylation; Esophageal Neoplasms; Esophageal Squamous Cell Carcinoma; Female; Gene Expression Regulation; Gene Silencing; Humans; Lymphatic Metastasis; Male; Promoter Regions, Genetic; RNA, Messenger

2014
Effects of 5-azacytidine on RUNX3 gene expression and the biological behavior of esophageal carcinoma cells.
    Molecular medicine reports, 2014, Volume: 9, Issue:4

    Topics: Animals; Azacitidine; Cell Line, Tumor; Cell Movement; Cell Proliferation; Cell Survival; Core Binding Factor Alpha 3 Subunit; Esophageal Neoplasms; Female; Gene Expression Regulation, Neoplastic; Humans; Male; Methylation; Mice; Mice, Inbred BALB C; Mice, Nude; Neoplasm Invasiveness; Xenograft Model Antitumor Assays

2014
Decreased expression of RASSF1A and up-regulation of RASSF1C is associated with esophageal squamous cell carcinoma.
    Clinical & experimental metastasis, 2014, Volume: 31, Issue:5

    Topics: Adult; Aged; Azacitidine; Carcinoma, Squamous Cell; Cell Line, Tumor; Cell Proliferation; Decitabine; DNA Methylation; Esophageal Neoplasms; Exons; Female; Gene Expression Regulation, Neoplastic; Gene Silencing; Humans; Male; Middle Aged; Neoplasm Grading; Neoplasm Metastasis; Neoplasm Staging; Promoter Regions, Genetic; RNA, Messenger; Tumor Suppressor Proteins

2014
Aberrant methylation and decreased expression of the TGF-β/Smad target gene FBXO32 in esophageal squamous cell carcinoma.
    Cancer, 2014, Aug-15, Volume: 120, Issue:16

    Topics: Adult; Aged; Azacitidine; Calmodulin-Binding Proteins; Carcinoma, Squamous Cell; Cell Growth Processes; Decitabine; DNA Methylation; Esophageal Neoplasms; Esophageal Squamous Cell Carcinoma; Female; Gene Silencing; Humans; Hydroxamic Acids; Immunohistochemistry; Male; Middle Aged; Muscle Proteins; Promoter Regions, Genetic; RNA, Messenger; SKP Cullin F-Box Protein Ligases; Smad Proteins; Transforming Growth Factor beta; Up-Regulation

2014
Temporal evolution in caveolin 1 methylation levels during human esophageal carcinogenesis.
    BMC cancer, 2014, May-20, Volume: 14

    Topics: Adenocarcinoma; Area Under Curve; Azacitidine; Barrett Esophagus; Carcinoma, Squamous Cell; Caveolin 1; Cell Line, Tumor; Decitabine; Disease Progression; DNA Methylation; DNA Modification Methylases; Enzyme Inhibitors; Esophageal Neoplasms; Gene Expression Regulation, Neoplastic; Humans; Promoter Regions, Genetic; RNA, Messenger; ROC Curve; Time Factors

2014
Low-dose decitabine induces MAGE-A expression and inhibits invasion via suppression of NF-κB2 and MMP2 in Eca109 cells.
    Biomedicine & pharmacotherapy = Biomedecine & pharmacotherapie, 2014, Volume: 68, Issue:6

    Topics: Antigens, Neoplasm; Antimetabolites, Antineoplastic; Azacitidine; Carcinoma, Squamous Cell; Cell Line, Tumor; Cell Proliferation; Decitabine; Esophageal Neoplasms; Esophageal Squamous Cell Carcinoma; Humans; Matrix Metalloproteinase 2; Neoplasm Invasiveness; Neoplasm Proteins; NF-kappa B p52 Subunit

2014
RASSF5A, a candidate tumor suppressor, is epigenetically inactivated in esophageal squamous cell carcinoma.
    Clinical & experimental metastasis, 2015, Volume: 32, Issue:1

    Topics: Adaptor Proteins, Signal Transducing; Adult; Aged; Alternative Splicing; Apoptosis; Apoptosis Regulatory Proteins; Azacitidine; Biomarkers, Tumor; Carcinoma, Squamous Cell; Cell Line, Tumor; Cell Proliferation; Cytidine Triphosphate; DNA Methylation; Esophageal Neoplasms; Esophagus; Female; Genes, Tumor Suppressor; Genetic Predisposition to Disease; Humans; Hydroxamic Acids; Male; Middle Aged; Monomeric GTP-Binding Proteins; Neoplasm Invasiveness; Prognosis; Promoter Regions, Genetic; Protein Isoforms; Protein Synthesis Inhibitors; RNA, Messenger, Stored

2015
Selective inhibition of esophageal cancer cells by combination of HDAC inhibitors and Azacytidine.
    Epigenetics, 2015, Volume: 10, Issue:5

    Topics: Acetylation; Adenocarcinoma; Antineoplastic Combined Chemotherapy Protocols; Apoptosis; Azacitidine; Benzamides; Carcinoma, Squamous Cell; Cell Cycle Checkpoints; Cell Line, Tumor; Cell Movement; Cell Survival; Decitabine; Depsipeptides; DNA (Cytosine-5-)-Methyltransferase 1; DNA (Cytosine-5-)-Methyltransferases; DNA Damage; Esophageal Neoplasms; Esophageal Squamous Cell Carcinoma; Histone Deacetylase Inhibitors; Histones; Humans; Hydroxamic Acids; Pyridines; Transcriptome; Vorinostat

2015
Methylation-mediated repression of potential tumor suppressor miR-203a and miR-203b contributes to esophageal squamous cell carcinoma development.
    Tumour biology : the journal of the International Society for Oncodevelopmental Biology and Medicine, 2016, Volume: 37, Issue:4

    Topics: Adult; Aged; Azacitidine; Biomarkers, Tumor; Carcinoma, Squamous Cell; Cell Line, Tumor; Cell Proliferation; CpG Islands; DNA Methylation; Esophageal Neoplasms; Esophageal Squamous Cell Carcinoma; Female; Gene Expression Regulation, Neoplastic; Humans; Lymphatic Metastasis; Male; MicroRNAs; Middle Aged; Prognosis; Promoter Regions, Genetic

2016
Epigenetic Repression of miR-218 Promotes Esophageal Carcinogenesis by Targeting ROBO1.
    International journal of molecular sciences, 2015, Nov-20, Volume: 16, Issue:11

    Topics: 3' Untranslated Regions; Azacitidine; Base Sequence; Binding Sites; Carcinoma, Squamous Cell; Cell Line, Tumor; Cell Proliferation; Cell Transformation, Neoplastic; CpG Islands; DNA Methylation; Epigenetic Repression; Esophageal Neoplasms; Esophageal Squamous Cell Carcinoma; G1 Phase Cell Cycle Checkpoints; Gene Expression Regulation, Neoplastic; Gene Order; Genetic Loci; Humans; MicroRNAs; Nerve Tissue Proteins; Receptors, Immunologic; RNA Interference; RNA, Messenger; Roundabout Proteins

2015
Response of esophageal cancer cells to epigenetic inhibitors is mediated via altered thioredoxin activity.
    Laboratory investigation; a journal of technical methods and pathology, 2016, Volume: 96, Issue:3

    Topics: Adult; Aged; Aged, 80 and over; Azacitidine; Carrier Proteins; Cell Line, Tumor; Epigenesis, Genetic; Esophageal Neoplasms; Female; Histone Deacetylase Inhibitors; Humans; Male; Middle Aged; Reactive Oxygen Species; Thioredoxin Reductase 1; Thioredoxins

2016
In vitro study of human mutL homolog 1 hypermethylation in inducing drug resistance of esophageal carcinoma.
    Irish journal of medical science, 2017, Volume: 186, Issue:2

    Topics: Azacitidine; Cell Line, Tumor; Cisplatin; Decitabine; DNA Methylation; Drug Resistance, Neoplasm; Esophageal Neoplasms; Humans; MutL Protein Homolog 1; Promoter Regions, Genetic; RNA, Messenger

2017
PRSS8 methylation and its significance in esophageal squamous cell carcinoma.
    Oncotarget, 2016, May-10, Volume: 7, Issue:19

    Topics: Azacitidine; Base Sequence; Carcinoma, Squamous Cell; Cell Line, Tumor; Cell Movement; Cell Proliferation; Decitabine; Disease-Free Survival; DNA Methylation; Down-Regulation; Enzyme Inhibitors; Esophageal Neoplasms; Female; Gene Expression Regulation, Neoplastic; HEK293 Cells; Humans; Male; Middle Aged; Promoter Regions, Genetic; RNA Interference; Sequence Homology, Nucleic Acid; Serine Endopeptidases

2016
Aberrant methylation of microRNA-193b in human Barrett's esophagus and esophageal adenocarcinoma.
    Molecular medicine reports, 2016, Volume: 14, Issue:1

    Topics: Adenocarcinoma; Adult; Aged; Azacitidine; Barrett Esophagus; Biomarkers, Tumor; Cell Line, Tumor; DNA Methylation; Down-Regulation; Esophageal Neoplasms; Female; Gene Expression Regulation, Neoplastic; Genes, ras; Humans; Immunohistochemistry; Male; MicroRNAs; Middle Aged; Promoter Regions, Genetic

2016
UHRF1 regulates global DNA hypomethylation and is associated with poor prognosis in esophageal squamous cell carcinoma.
    Oncotarget, 2016, Sep-06, Volume: 7, Issue:36

    Topics: Aged; Azacitidine; Biomarkers, Tumor; Carcinoma, Squamous Cell; CCAAT-Enhancer-Binding Proteins; Cell Transformation, Neoplastic; Disease-Free Survival; DNA Methylation; Epigenesis, Genetic; Epigenomics; Esophageal Neoplasms; Esophageal Squamous Cell Carcinoma; Esophagus; Female; Humans; Long Interspersed Nucleotide Elements; Male; Middle Aged; Prognosis; Proportional Hazards Models; Sequence Analysis, DNA; Treatment Outcome; Ubiquitin; Ubiquitin-Protein Ligases

2016
5-Azacytidine suppresses EC9706 cell proliferation and metastasis by upregulating the expression of SOX17 and CDH1.
    International journal of molecular medicine, 2016, Volume: 38, Issue:4

    Topics: Antigens, CD; Azacitidine; Cadherins; Cell Line, Tumor; Cell Proliferation; DNA Methylation; Esophageal Neoplasms; Gene Expression Regulation, Neoplastic; Humans; Neoplasm Metastasis; Promoter Regions, Genetic; SOXF Transcription Factors; Up-Regulation

2016
Methylation decreases the Bin1 tumor suppressor in ESCC and restoration by decitabine inhibits the epithelial mesenchymal transition.
    Oncotarget, 2017, Mar-21, Volume: 8, Issue:12

    Topics: Adaptor Proteins, Signal Transducing; Adult; Aged; Animals; Antimetabolites, Antineoplastic; Apoptosis; Azacitidine; Biomarkers, Tumor; Carcinoma, Squamous Cell; Cell Cycle; Cell Movement; Cell Proliferation; Decitabine; DNA Methylation; Epithelial-Mesenchymal Transition; Esophageal Neoplasms; Female; Follow-Up Studies; Gene Expression Regulation, Neoplastic; Genes, Tumor Suppressor; Humans; Lymphatic Metastasis; Male; Mice; Mice, Inbred BALB C; Mice, Nude; Middle Aged; Neoplasm Invasiveness; Neoplasm Staging; Nuclear Proteins; Prognosis; Promoter Regions, Genetic; Signal Transduction; Survival Rate; Tumor Cells, Cultured; Tumor Suppressor Proteins; Xenograft Model Antitumor Assays

2017
Analysis of the methylation status of genes up-regulated by the demethylating agent, 5-aza-2'-deoxycytidine, in esophageal squamous cell carcinoma.
    Oncology reports, 2008, Volume: 20, Issue:2

    Topics: Antimetabolites, Antineoplastic; Azacitidine; Carcinoma, Squamous Cell; Cathepsin L; Cathepsins; Cell Cycle Proteins; Cell Line, Tumor; Cell Movement; Cell Proliferation; Cysteine Endopeptidases; Decitabine; DNA Methylation; DNA Modification Methylases; Esophageal Neoplasms; Esophagus; Fatty Acid-Binding Proteins; Gene Expression Regulation, Neoplastic; Humans; Neoplasm Proteins; Nuclear Proteins; Promoter Regions, Genetic; Reverse Transcriptase Polymerase Chain Reaction; RNA, Messenger

2008
DNA hypermethylation regulates the expression of members of the Mu-class glutathione S-transferases and glutathione peroxidases in Barrett's adenocarcinoma.
    Gut, 2009, Volume: 58, Issue:1

    Topics: Adenocarcinoma; Adult; Aged; Aged, 80 and over; Antimetabolites, Antineoplastic; Azacitidine; Barrett Esophagus; Cell Transformation, Neoplastic; CpG Islands; Decitabine; Disease Progression; DNA Methylation; DNA, Neoplasm; Down-Regulation; Epigenesis, Genetic; Esophageal Neoplasms; Gene Expression Regulation, Enzymologic; Glutathione Peroxidase; Glutathione Transferase; Humans; Hydroxamic Acids; Middle Aged; Promoter Regions, Genetic; Reverse Transcriptase Polymerase Chain Reaction; RNA, Messenger; RNA, Neoplasm; Tumor Cells, Cultured

2009
Promoter hypermethylation of CDH13 is a common, early event in human esophageal adenocarcinogenesis and correlates with clinical risk factors.
    International journal of cancer, 2008, Nov-15, Volume: 123, Issue:10

    Topics: Adenocarcinoma; Azacitidine; Base Sequence; Cadherins; Cell Line, Tumor; DNA Methylation; DNA Primers; Esophageal Neoplasms; Female; Humans; Male; Middle Aged; Promoter Regions, Genetic; Reverse Transcriptase Polymerase Chain Reaction; Risk Factors

2008
Identification of candidate tumor suppressor genes inactivated by promoter methylation in melanoma.
    Genes, chromosomes & cancer, 2009, Volume: 48, Issue:1

    Topics: Azacitidine; Carrier Proteins; Cell Line, Tumor; Colonic Neoplasms; CpG Islands; Decitabine; DNA Methylation; Esophageal Neoplasms; Gene Expression Profiling; Gene Silencing; Genes, Tumor Suppressor; Glioma; Heat-Shock Proteins; Humans; Hydroxamic Acids; Intracellular Signaling Peptides and Proteins; Melanoma; Membrane Proteins; Microfilament Proteins; Neuropeptides; Nuclear Proteins; Oligonucleotide Array Sequence Analysis; Phosphoprotein Phosphatases; Promoter Regions, Genetic; Reverse Transcriptase Polymerase Chain Reaction

2009
Quantitation of DNA methylation by melt curve analysis.
    BMC cancer, 2009, Apr-24, Volume: 9

    Topics: Algorithms; Antimetabolites, Antineoplastic; Azacitidine; Cell Line, Tumor; Colorectal Neoplasms; Cyclin-Dependent Kinase Inhibitor p16; Decitabine; DNA Methylation; DNA Modification Methylases; DNA Repair Enzymes; DNA, Neoplasm; Esophageal Neoplasms; Hot Temperature; Humans; Immunohistochemistry; Nucleic Acid Denaturation; Oligonucleotides; Polymerase Chain Reaction; Promoter Regions, Genetic; Tissue Inhibitor of Metalloproteinase-3; Tumor Suppressor Proteins

2009
Real-time PCR analysis of genes encoding tumor antigens in esophageal tumors and a cancer vaccine.
    Cancer immunity, 2009, Oct-09, Volume: 9

    Topics: Aged; Antigens, Neoplasm; Azacitidine; Cancer Vaccines; Carcinoma, Squamous Cell; Clone Cells; Decitabine; DNA Modification Methylases; Epithelial Cells; Esophageal Neoplasms; Female; Gene Expression Profiling; Humans; Male; Middle Aged; Neoplasm Proteins; Reverse Transcriptase Polymerase Chain Reaction

2009
Identification of GPX3 epigenetically silenced by CpG methylation in human esophageal squamous cell carcinoma.
    Digestive diseases and sciences, 2011, Volume: 56, Issue:3

    Topics: Antimetabolites, Antineoplastic; Azacitidine; Carcinoma, Squamous Cell; Cell Line, Tumor; CpG Islands; Decitabine; DNA Methylation; Down-Regulation; Esophageal Neoplasms; Female; Gene Expression Regulation, Neoplastic; Gene Silencing; Glutathione Peroxidase; Humans; Male; Middle Aged; Promoter Regions, Genetic; Tumor Suppressor Proteins

2011
Promoter hypermethylation of cyclooxygenase-2 gene in esophageal squamous cell carcinoma.
    Diseases of the esophagus : official journal of the International Society for Diseases of the Esophagus, 2011, Volume: 24, Issue:6

    Topics: Azacitidine; Carcinoma, Squamous Cell; Cell Line, Tumor; Cyclooxygenase 2; Decitabine; Dinoprostone; DNA Methylation; Esophageal Neoplasms; Gene Expression Regulation, Neoplastic; Gene Silencing; Humans; Promoter Regions, Genetic; RNA, Messenger; Sequence Analysis, DNA

2011
Hypermethylation-modulated down-regulation of CDH1 expression contributes to the progression of esophageal cancer.
    International journal of molecular medicine, 2011, Volume: 27, Issue:5

    Topics: Adult; Aged; Aged, 80 and over; Antigens, CD; Azacitidine; Cadherins; Carcinoma, Squamous Cell; Cell Adhesion; Cell Cycle; Cell Line, Tumor; Cell Movement; Cell Shape; Cell Survival; Decitabine; DNA Methylation; DNA Modification Methylases; Down-Regulation; Esophageal Neoplasms; Female; Humans; Male; Methylation; Middle Aged; Neoplasm Invasiveness; Neoplasm Staging; Promoter Regions, Genetic; Transcription, Genetic

2011
Epigenetic inactivation of the SFRP1 gene in esophageal squamous cell carcinoma.
    Digestive diseases and sciences, 2011, Volume: 56, Issue:11

    Topics: Antimetabolites, Antineoplastic; Azacitidine; Carcinoma, Squamous Cell; Cell Line, Tumor; Decitabine; DNA Methylation; Esophageal Neoplasms; Gene Silencing; Histone Deacetylase Inhibitors; Humans; Hydroxamic Acids; Intercellular Signaling Peptides and Proteins; Membrane Proteins

2011
Location-specific epigenetic regulation of the metallothionein 3 gene in esophageal adenocarcinomas.
    PloS one, 2011, Volume: 6, Issue:7

    Topics: Adenocarcinoma; Adult; Aged; Aged, 80 and over; Azacitidine; Cell Line, Tumor; CpG Islands; DNA Methylation; Down-Regulation; Epigenesis, Genetic; Esophageal Neoplasms; Gene Expression Regulation, Neoplastic; Genes, Neoplasm; Histones; Humans; Lymphatic Metastasis; Metallothionein 3; Middle Aged; Neoplasm Staging; Nerve Tissue Proteins; Promoter Regions, Genetic; Protein Processing, Post-Translational; Repressor Proteins

2011
Aberrant methylation of the 3q25 tumor suppressor gene PTX3 in human esophageal squamous cell carcinoma.
    World journal of gastroenterology, 2011, Oct-07, Volume: 17, Issue:37

    Topics: Antimetabolites, Antineoplastic; Azacitidine; C-Reactive Protein; Carcinoma, Squamous Cell; Cell Line, Tumor; Chromosomes, Human, Pair 3; Decitabine; DNA Methylation; Down-Regulation; Esophageal Neoplasms; Gene Expression Regulation, Neoplastic; Humans; Promoter Regions, Genetic; Serum Amyloid P-Component

2011
[Aberrant methylation of T-cadherin gene in human esophageal carcinoma cells].
    Zhonghua yi xue za zhi, 2011, Oct-11, Volume: 91, Issue:37

    Topics: Azacitidine; Cadherins; Cell Line, Tumor; DNA Methylation; Esophageal Neoplasms; Humans; Promoter Regions, Genetic

2011
[Mechanism of loss of human esophageal cancer-related gene 4 (ECRG4) gene expression in esophageal squamous cell carcinoma cell line EC9706].
    Zhonghua zhong liu za zhi [Chinese journal of oncology], 2011, Volume: 33, Issue:8

    Topics: Antimetabolites, Antineoplastic; Antineoplastic Agents; Arsenic Trioxide; Arsenicals; Azacitidine; Carcinoma, Squamous Cell; Cell Line, Tumor; CpG Islands; Decitabine; DNA Methylation; Epigenesis, Genetic; Esophageal Neoplasms; Exons; Gene Expression Regulation, Neoplastic; Humans; Mutation; Neoplasm Proteins; Oxides; Promoter Regions, Genetic; RNA, Messenger; Tumor Suppressor Proteins

2011
Study on RIZ1 gene promoter methylation status in human esophageal squamous cell carcinoma.
    World journal of gastroenterology, 2012, Feb-14, Volume: 18, Issue:6

    Topics: Antimetabolites, Antineoplastic; Azacitidine; Base Sequence; Carcinoma, Squamous Cell; Cell Line, Tumor; DNA Methylation; DNA-Binding Proteins; Epigenesis, Genetic; Esophageal Neoplasms; Female; Gene Silencing; Histone-Lysine N-Methyltransferase; Humans; Male; Middle Aged; Molecular Sequence Data; Nuclear Proteins; Promoter Regions, Genetic; Sequence Analysis, DNA; Transcription Factors

2012
Decreased expression of retinoblastoma protein-interacting zinc-finger gene 1 in human esophageal squamous cell cancer by DNA methylation.
    Clinical laboratory, 2012, Volume: 58, Issue:1-2

    Topics: Antimetabolites, Antineoplastic; Azacitidine; Base Sequence; Carcinoma, Squamous Cell; Cell Line, Tumor; Decitabine; DNA Methylation; DNA-Binding Proteins; Drug Screening Assays, Antitumor; Esophageal Neoplasms; Gene Expression Regulation, Neoplastic; Gene Silencing; Histone-Lysine N-Methyltransferase; Humans; Molecular Sequence Data; Nuclear Proteins; RNA, Messenger; Transcription Factors

2012
Promoter methylation regulates cigarette smoke-stimulated cyclooxygenase-2 expression in esophageal squamous cell carcinoma.
    Journal of digestive diseases, 2012, Volume: 13, Issue:4

    Topics: Antimetabolites, Antineoplastic; Azacitidine; Carcinoma, Squamous Cell; Cell Line, Tumor; Cyclooxygenase 2; Decitabine; DNA Methylation; Esophageal Neoplasms; Gene Expression Regulation, Enzymologic; Gene Expression Regulation, Neoplastic; Humans; Promoter Regions, Genetic; Smoking

2012
XAF1 is frequently methylated in human esophageal cancer.
    World journal of gastroenterology, 2012, Jun-14, Volume: 18, Issue:22

    Topics: Adaptor Proteins, Signal Transducing; Aged; Apoptosis Regulatory Proteins; Azacitidine; Biomarkers, Tumor; Blotting, Western; Carcinoma, Squamous Cell; Cell Line, Tumor; Chi-Square Distribution; China; Decitabine; DNA Methylation; DNA Modification Methylases; Enzyme Inhibitors; Epigenesis, Genetic; Esophageal Neoplasms; Female; Gene Expression Regulation, Neoplastic; Humans; Immunohistochemistry; Intracellular Signaling Peptides and Proteins; Logistic Models; Male; Middle Aged; Multivariate Analysis; Neoplasm Proteins; Promoter Regions, Genetic; Reverse Transcriptase Polymerase Chain Reaction

2012
MYEOV, a gene at 11q13, is coamplified with CCND1, but epigenetically inactivated in a subset of esophageal squamous cell carcinomas.
    Journal of human genetics, 2002, Volume: 47, Issue:9

    Topics: Azacitidine; Blotting, Northern; Blotting, Southern; Carcinoma, Squamous Cell; Chromosomes, Human, Pair 11; Cyclin D1; Decitabine; DNA Methylation; Esophageal Neoplasms; Gene Amplification; Gene Silencing; Humans; Neoplasm Proteins; Oncogene Proteins; Proto-Oncogene Proteins; Tumor Cells, Cultured

2002
Pharmacologic unmasking of epigenetically silenced tumor suppressor genes in esophageal squamous cell carcinoma.
    Cancer cell, 2002, Volume: 2, Issue:6

    Topics: Azacitidine; Base Sequence; Carcinoma, Squamous Cell; CpG Islands; Decitabine; DNA Methylation; Down-Regulation; Enzyme Inhibitors; Esophageal Neoplasms; Gene Expression Regulation, Neoplastic; Gene Silencing; Genes, Tumor Suppressor; Humans; Hydroxamic Acids; Molecular Sequence Data; Oligonucleotide Array Sequence Analysis; Promoter Regions, Genetic; Reverse Transcriptase Polymerase Chain Reaction; Tumor Cells, Cultured

2002
Frequent silencing of low density lipoprotein receptor-related protein 1B (LRP1B) expression by genetic and epigenetic mechanisms in esophageal squamous cell carcinoma.
    Cancer research, 2004, Jun-01, Volume: 64, Issue:11

    Topics: Acetylation; Antimetabolites, Antineoplastic; Azacitidine; Carcinoma, Squamous Cell; Cell Division; Cell Line, Tumor; CpG Islands; Decitabine; DNA Methylation; Esophageal Neoplasms; Gene Deletion; Gene Expression Regulation, Neoplastic; Gene Silencing; Histones; Humans; Nucleic Acid Hybridization; Receptors, LDL; Reverse Transcriptase Polymerase Chain Reaction; RNA, Messenger

2004
Sequential 5-Aza 2'-deoxycytidine/depsipeptide FK228 treatment induces tissue factor pathway inhibitor 2 (TFPI-2) expression in cancer cells.
    Oncogene, 2005, Mar-31, Volume: 24, Issue:14

    Topics: Azacitidine; Base Sequence; Cell Line, Tumor; Decitabine; Depsipeptides; DNA Primers; Esophageal Neoplasms; Glycoproteins; Humans; Lung Neoplasms

2005
Reversal of hypermethylation and reactivation of p16INK4a, RARbeta, and MGMT genes by genistein and other isoflavones from soy.
    Clinical cancer research : an official journal of the American Association for Cancer Research, 2005, Oct-01, Volume: 11, Issue:19 Pt 1

    Topics: Anticarcinogenic Agents; Antineoplastic Agents; Azacitidine; Carcinoma, Squamous Cell; Cell Line, Tumor; Cell Proliferation; Cyclin-Dependent Kinase Inhibitor p16; Decitabine; DNA Methylation; Dose-Response Relationship, Drug; Esophageal Neoplasms; Genistein; Glycine max; Humans; Hydroxamic Acids; Isoflavones; Isothiocyanates; O(6)-Methylguanine-DNA Methyltransferase; Receptors, Retinoic Acid; Reverse Transcriptase Polymerase Chain Reaction; RNA, Messenger; Sulfites; Sulfoxides; Tea; Thiocyanates

2005
Functional epigenetics identifies a protocadherin PCDH10 as a candidate tumor suppressor for nasopharyngeal, esophageal and multiple other carcinomas with frequent methylation.
    Oncogene, 2006, Feb-16, Volume: 25, Issue:7

    Topics: Azacitidine; Cadherins; Carcinoma; Cell Movement; Decitabine; DNA (Cytosine-5-)-Methyltransferase 1; DNA (Cytosine-5-)-Methyltransferases; DNA Methylation; DNA Methyltransferase 3B; Epigenesis, Genetic; Esophageal Neoplasms; Gene Silencing; Genes, Tumor Suppressor; Humans; Nasopharyngeal Neoplasms; Promoter Regions, Genetic; Protocadherins; Sequence Deletion; Transcription, Genetic

2006
5-Aza-2'-deoxycytidine induces retinoic acid receptor-beta(2) demethylation and growth inhibition in esophageal squamous carcinoma cells.
    Cancer letters, 2005, Dec-18, Volume: 230, Issue:2

    Topics: Adult; Aged; Aged, 80 and over; Antimetabolites, Antineoplastic; Azacitidine; Carcinoma, Squamous Cell; Cell Line, Tumor; Cell Proliferation; Cell Survival; Decitabine; DNA Methylation; DNA Modification Methylases; Esophageal Neoplasms; Esophagus; Female; Gene Expression Regulation, Neoplastic; Humans; Male; Middle Aged; Receptors, Retinoic Acid; RNA, Messenger

2005
Alterations of the Wnt signaling pathway during the neoplastic progression of Barrett's esophagus.
    Oncogene, 2006, May-18, Volume: 25, Issue:21

    Topics: Adenocarcinoma; Azacitidine; Barrett Esophagus; beta Catenin; Cell Division; Cell Line, Tumor; CpG Islands; Decitabine; Disease Progression; DNA Methylation; DNA, Neoplasm; Esophageal Neoplasms; Gene Expression Regulation; Gene Expression Regulation, Neoplastic; Gene Silencing; Genes, APC; Humans; Intercellular Signaling Peptides and Proteins; Membrane Proteins; Mucous Membrane; Neoplasm Proteins; Precancerous Conditions; Promoter Regions, Genetic; RNA, Messenger; RNA, Neoplasm; RNA, Small Interfering; Signal Transduction; Transfection; Wnt Proteins; Wnt2 Protein

2006
Reprimo methylation is a potential biomarker of Barrett's-Associated esophageal neoplastic progression.
    Clinical cancer research : an official journal of the American Association for Cancer Research, 2006, Nov-15, Volume: 12, Issue:22

    Topics: Adenocarcinoma; Aged; Azacitidine; Barrett Esophagus; Carcinoma, Squamous Cell; Cell Cycle Proteins; Cell Line, Tumor; Decitabine; Disease Progression; DNA Methylation; Esophageal Diseases; Esophageal Neoplasms; Female; Genetic Markers; Glycoproteins; Humans; Male; Middle Aged; Polymerase Chain Reaction; Precancerous Conditions

2006
The tumor suppressor Wnt inhibitory factor 1 is frequently methylated in nasopharyngeal and esophageal carcinomas.
    Laboratory investigation; a journal of technical methods and pathology, 2007, Volume: 87, Issue:7

    Topics: Adaptor Proteins, Signal Transducing; Animals; Azacitidine; Biomarkers, Tumor; Carcinoma; Cell Line; Cell Line, Tumor; Cell Proliferation; Decitabine; DNA Methylation; Down-Regulation; Esophageal Neoplasms; Gene Expression Regulation, Neoplastic; Gene Silencing; Humans; Nasopharyngeal Neoplasms; Promoter Regions, Genetic; Repressor Proteins; Tumor Suppressor Proteins; Wnt1 Protein

2007
Lysyl oxidase-like 2 expression is increased in colon and esophageal tumors and associated with less differentiated colon tumors.
    Genes, chromosomes & cancer, 2007, Volume: 46, Issue:7

    Topics: Adult; Aged; Aged, 80 and over; Amino Acid Oxidoreductases; Azacitidine; Blotting, Western; Cell Differentiation; Cell Line; Colonic Neoplasms; CpG Islands; Decitabine; DNA Primers; Esophageal Neoplasms; Female; Gene Expression Regulation, Neoplastic; Humans; Immunohistochemistry; Loss of Heterozygosity; Male; Middle Aged; Promoter Regions, Genetic; Reverse Transcriptase Polymerase Chain Reaction

2007
Frequent methylation-associated silencing of a candidate tumor-suppressor, CRABP1, in esophageal squamous-cell carcinoma.
    Oncogene, 2007, Sep-27, Volume: 26, Issue:44

    Topics: Azacitidine; Biomarkers, Tumor; Carcinoma, Squamous Cell; Cell Differentiation; Cell Proliferation; DNA Methylation; Epigenesis, Genetic; Esophageal Neoplasms; Female; Gene Expression Regulation, Neoplastic; Gene Silencing; Humans; Lymphatic Metastasis; Male; Middle Aged; Promoter Regions, Genetic; Receptors, Retinoic Acid

2007
Hypermethylation of the nel-like 1 gene is a common and early event and is associated with poor prognosis in early-stage esophageal adenocarcinoma.
    Oncogene, 2007, Sep-20, Volume: 26, Issue:43

    Topics: Aged; Azacitidine; Calcium-Binding Proteins; Cell Line, Tumor; Cytidine Triphosphate; DNA Methylation; Esophageal Neoplasms; Humans; Middle Aged; Neoplasm Staging; Nerve Tissue Proteins; Prognosis; Promoter Regions, Genetic; RNA, Messenger; Survival Rate; Time Factors

2007
p300 expression repression by hypermethylation associated with tumour invasion and metastasis in oesophageal squamous cell carcinoma.
    Journal of clinical pathology, 2007, Volume: 60, Issue:11

    Topics: Azacitidine; Carcinoma, Squamous Cell; Decitabine; DNA Methylation; DNA, Neoplasm; E1A-Associated p300 Protein; Esophageal Neoplasms; Female; Gene Expression Regulation, Neoplastic; Gene Silencing; Humans; Lymphatic Metastasis; Male; Neoplasm Invasiveness; Neoplasm Proteins; Promoter Regions, Genetic; Reverse Transcriptase Polymerase Chain Reaction; RNA, Messenger; RNA, Neoplasm

2007
Hypermethylation of the somatostatin promoter is a common, early event in human esophageal carcinogenesis.
    Cancer, 2008, Jan-01, Volume: 112, Issue:1

    Topics: Azacitidine; Barrett Esophagus; Cell Line, Tumor; DNA Methylation; Esophageal Neoplasms; Gene Silencing; Humans; Polymerase Chain Reaction; Promoter Regions, Genetic; Somatostatin

2008
Promoter hypermethylation of the RUNX3 gene in esophageal squamous cell carcinoma.
    Cancer investigation, 2007, Volume: 25, Issue:8

    Topics: Aged; Azacitidine; Carcinoma, Squamous Cell; Cell Line, Tumor; Core Binding Factor Alpha 3 Subunit; DNA Methylation; Esophageal Neoplasms; Female; Humans; Male; Middle Aged; Promoter Regions, Genetic; RNA, Messenger; Signal Transduction; Transforming Growth Factor beta

2007
Clinical significance of the reduced expression of G protein gamma 7 (GNG7) in oesophageal cancer.
    British journal of cancer, 2008, Jan-29, Volume: 98, Issue:2

    Topics: Adult; Aged; Aged, 80 and over; Antimetabolites, Antineoplastic; Azacitidine; Carcinoma; Cell Line, Tumor; Chromosomes, Human, Pair 19; DNA Methylation; Down-Regulation; Esophageal Neoplasms; Female; Follow-Up Studies; Gene Expression Regulation, Neoplastic; GTP-Binding Protein gamma Subunits; Humans; Loss of Heterozygosity; Male; MicroRNAs; Middle Aged; Neoplasm Invasiveness; Prognosis; RNA, Messenger

2008
Progressive silencing of p14ARF in oesophageal adenocarcinoma.
    Journal of cellular and molecular medicine, 2009, Volume: 13, Issue:2

    Topics: Adenocarcinoma; Adult; Aged; Aged, 80 and over; Antimetabolites, Antineoplastic; Azacitidine; Barrett Esophagus; Base Sequence; Cell Line; Decitabine; Disease Progression; Epithelial Cells; Esophageal Neoplasms; Female; Gene Silencing; Histones; Humans; Male; Methylation; Middle Aged; Molecular Sequence Data; Survival Rate; Tumor Suppressor Protein p14ARF

2009
Methylation of the 5' CpG island of the FHIT gene is closely associated with transcriptional inactivation in esophageal squamous cell carcinomas.
    Cancer research, 1998, Aug-01, Volume: 58, Issue:15

    Topics: Acid Anhydride Hydrolases; Antimetabolites, Antineoplastic; Azacitidine; Base Sequence; Carcinoma, Squamous Cell; CpG Islands; Decitabine; DNA Methylation; DNA, Neoplasm; Esophageal Neoplasms; Exons; Gene Deletion; Gene Expression Regulation, Neoplastic; Humans; Molecular Sequence Data; Neoplasm Proteins; Polymerase Chain Reaction; Polymorphism, Single-Stranded Conformational; Proteins; Transcriptional Activation; Tumor Cells, Cultured

1998
Induction of MAGE-3 expression in lung and esophageal cancer cells.
    The Annals of thoracic surgery, 2001, Volume: 71, Issue:1

    Topics: Adenocarcinoma; Anti-Bacterial Agents; Antibiotics, Antineoplastic; Antigens, Neoplasm; Azacitidine; Carcinoma, Non-Small-Cell Lung; Decitabine; Depsipeptides; DNA Modification Methylases; Enzyme Inhibitors; Esophageal Neoplasms; Histone Deacetylase Inhibitors; Humans; Immunohistochemistry; Lung Neoplasms; Melanoma; Neoplasm Proteins; Peptides, Cyclic; Reverse Transcriptase Polymerase Chain Reaction; RNA, Messenger; Tumor Cells, Cultured

2001
DNA hypermethylation is a mechanism for loss of expression of the HLA class I genes in human esophageal squamous cell carcinomas.
    Carcinogenesis, 2001, Volume: 22, Issue:10

    Topics: Azacitidine; Carcinoma, Squamous Cell; Chromosomes, Human, Pair 6; CpG Islands; Decitabine; DNA Methylation; DNA Modification Methylases; DNA Primers; DNA, Neoplasm; Down-Regulation; Esophageal Neoplasms; Gene Deletion; Genes, MHC Class I; Histocompatibility Antigens Class I; Humans; Immunoenzyme Techniques; Loss of Heterozygosity; Microsatellite Repeats; Reverse Transcriptase Polymerase Chain Reaction; RNA, Messenger

2001
E-cadherin expression is commonly downregulated by CpG island hypermethylation in esophageal carcinoma cells.
    Cancer letters, 2001, Nov-08, Volume: 173, Issue:1

    Topics: Azacitidine; Cadherins; Carcinoma; CpG Islands; Decitabine; DNA Methylation; DNA Modification Methylases; Down-Regulation; Enzyme Inhibitors; Esophageal Neoplasms; Humans; Promoter Regions, Genetic; RNA, Neoplasm; Transcription, Genetic; Tumor Cells, Cultured

2001