azacitidine and Atherogenesis

azacitidine has been researched along with Atherogenesis in 6 studies

Research

Studies (6)

TimeframeStudies, this research(%)All Research%
pre-19900 (0.00)18.7374
1990's0 (0.00)18.2507
2000's1 (16.67)29.6817
2010's5 (83.33)24.3611
2020's0 (0.00)2.80

Authors

AuthorsStudies
Cao, M; Lü, CX; Quan, XQ; Ruan, L; Shang, SS; Wang, G; Wu, XF; Xu, RD; Yan, FQ; Zhang, CT1
Dunn, J; Fan, Y; Hoffman, R; Jang, I; Jjingo, D; Jo, H; Jordan, IK; Kim, CW; Kim, D; Kim, S; Pan, C; Qiu, H; Son, DJ1
Cao, Q; Das, SK; Diallo, A; Hawkins, GA; Jia, L; Mondal, AK; Parks, JS; Shi, H; Wang, X; Xue, B; Yu, L1
Dunn, J; Jo, H; Simmons, R; Thabet, S1
Li, H; Luan, P; Peng, W; Wang, K; Xu, Y; Zhang, P; Zhuang, J1
Dong, C; Goldschmidt-Clermont, PJ; Zhu, S1

Reviews

1 review(s) available for azacitidine and Atherogenesis

ArticleYear
The role of epigenetics in the endothelial cell shear stress response and atherosclerosis.
    The international journal of biochemistry & cell biology, 2015, Volume: 67

    Topics: Animals; Apolipoproteins E; Atherosclerosis; Azacitidine; Decitabine; DNA (Cytosine-5-)-Methyltransferase 1; DNA (Cytosine-5-)-Methyltransferases; DNA Methylation; DNA Methyltransferase 3A; Endothelial Cells; Epigenesis, Genetic; Hemodynamics; Homeodomain Proteins; Humans; Kruppel-Like Transcription Factors; Mechanotransduction, Cellular; Mice; RNA, Small Interfering; Stress, Mechanical

2015

Other Studies

5 other study(ies) available for azacitidine and Atherogenesis

ArticleYear
FOXP3 demethylation as a means of identifying quantitative defects in regulatory T cells in acute coronary syndrome.
    Atherosclerosis, 2013, Volume: 229, Issue:1

    Topics: Acute Coronary Syndrome; Adult; Aged; Atherosclerosis; Azacitidine; Biomarkers; CD4 Antigens; Cells, Cultured; Cholesterol, LDL; Decitabine; DNA Modification Methylases; Down-Regulation; Female; Flow Cytometry; Forkhead Transcription Factors; Humans; Interferon-gamma; Interleukin-10; Interleukin-2 Receptor alpha Subunit; Male; Methylation; Middle Aged; T-Lymphocytes, Regulatory; Transforming Growth Factor beta1; Troponin I; Up-Regulation

2013
Flow-dependent epigenetic DNA methylation regulates endothelial gene expression and atherosclerosis.
    The Journal of clinical investigation, 2014, Volume: 124, Issue:7

    Topics: Animals; Apolipoproteins E; Atherosclerosis; Azacitidine; Decitabine; Disease Models, Animal; DNA (Cytosine-5-)-Methyltransferase 1; DNA (Cytosine-5-)-Methyltransferases; DNA Methylation; Endothelial Cells; Epigenesis, Genetic; Gene Expression Regulation; Homeodomain Proteins; Human Umbilical Vein Endothelial Cells; Humans; Kruppel-Like Transcription Factors; Mice; Mice, Inbred C57BL; Mice, Knockout; Phosphoproteins; Plaque, Atherosclerotic; Promoter Regions, Genetic; Regional Blood Flow; RNA, Messenger; Stress, Mechanical; Transcription Factors

2014
Inhibiting DNA Methylation by 5-Aza-2'-deoxycytidine ameliorates atherosclerosis through suppressing macrophage inflammation.
    Endocrinology, 2014, Volume: 155, Issue:12

    Topics: Animals; Atherosclerosis; Azacitidine; Cell Adhesion; Cell Movement; Cells, Cultured; Decitabine; DNA Methylation; Endoplasmic Reticulum Stress; Lipids; Liver X Receptors; Macrophages; Male; Mice; Mice, Knockout; Orphan Nuclear Receptors; Plaque, Atherosclerotic; PPAR gamma; Promoter Regions, Genetic; Random Allocation; Rats

2014
The Yin-Yang Dynamics of DNA Methylation Is the Key Regulator for Smooth Muscle Cell Phenotype Switch and Vascular Remodeling.
    Arteriosclerosis, thrombosis, and vascular biology, 2017, Volume: 37, Issue:1

    Topics: 5-Methylcytosine; Animals; Aorta; Aortic Diseases; Apolipoproteins E; Atherosclerosis; Azacitidine; Cell Dedifferentiation; Cell Movement; Cell Proliferation; Cells, Cultured; Decitabine; Diet, High-Fat; Dioxygenases; Disease Models, Animal; DNA (Cytosine-5-)-Methyltransferase 1; DNA (Cytosine-5-)-Methyltransferases; DNA Methylation; DNA-Binding Proteins; Enzyme Inhibitors; Epigenesis, Genetic; Genetic Predisposition to Disease; Mice, Knockout; Muscle, Smooth, Vascular; Myocytes, Smooth Muscle; Nuclear Proteins; Phenotype; Promoter Regions, Genetic; Proto-Oncogene Proteins; Rats; Signal Transduction; Time Factors; Trans-Activators; Vascular Remodeling

2017
Inactivation of monocarboxylate transporter MCT3 by DNA methylation in atherosclerosis.
    Circulation, 2005, Aug-30, Volume: 112, Issue:9

    Topics: Aorta; Atherosclerosis; Azacitidine; Biological Transport; Cell Proliferation; Cells, Cultured; CpG Islands; Decitabine; DNA Methylation; Humans; Lactic Acid; Membrane Transport Proteins; Monocarboxylic Acid Transporters; Muscle, Smooth, Vascular; Polymerase Chain Reaction; RNA, Messenger; RNA, Small Interfering; Symporters

2005