azacitidine and Abnormalities, Multiple

azacitidine has been researched along with Abnormalities, Multiple in 3 studies

Research

Studies (3)

TimeframeStudies, this research(%)All Research%
pre-19900 (0.00)18.7374
1990's0 (0.00)18.2507
2000's3 (100.00)29.6817
2010's0 (0.00)24.3611
2020's0 (0.00)2.80

Authors

AuthorsStudies
Hatta, T; Matsumoto, A; Moriyama, K; Otani, H1
Delmas, AL; Fields, CR; Jin, B; Liu, X; Peng, J; Qiu, J; Robertson, KD; Soo, HM; Tao, Q; Wu, W; Ying, J1
Kumar, A1

Other Studies

3 other study(ies) available for azacitidine and Abnormalities, Multiple

ArticleYear
Opposite effects of the maternal immune system activated by interleukin-1beta vs. PSK and OK432 on 5-azacytidine-induced birth defects.
    Congenital anomalies, 2003, Volume: 43, Issue:1

    Topics: Abnormalities, Multiple; Adjuvants, Immunologic; Animals; Antimetabolites, Antineoplastic; Antineoplastic Agents; Azacitidine; Body Weight; Brain; Congenital Abnormalities; Female; Interleukin-1; Mice; Mice, Inbred ICR; Microscopy, Electron, Scanning; Phagocytes; Picibanil; Placenta; Pregnancy; Pregnancy, Animal; Proteoglycans; Teratogens; Time Factors

2003
DNA methyltransferase 3B (DNMT3B) mutations in ICF syndrome lead to altered epigenetic modifications and aberrant expression of genes regulating development, neurogenesis and immune function.
    Human molecular genetics, 2008, Mar-01, Volume: 17, Issue:5

    Topics: Abnormalities, Multiple; Acetylation; Azacitidine; B-Lymphocytes; Case-Control Studies; Cell Line, Transformed; Cell Transformation, Viral; Cells, Cultured; Decitabine; DNA (Cytosine-5-)-Methyltransferases; DNA Methylation; DNA Methyltransferase 3B; Enzyme Inhibitors; Epigenesis, Genetic; Female; Gene Expression Profiling; Gene Expression Regulation, Developmental; Genes, Recessive; Histones; Humans; Hydroxamic Acids; Immunologic Deficiency Syndromes; Intellectual Disability; Male; Mutation; Neurons; Oligonucleotide Array Sequence Analysis; Promoter Regions, Genetic; Time Factors

2008
Rett and ICF syndromes: methylation moves into medicine.
    Journal of biosciences, 2000, Volume: 25, Issue:3

    Topics: Abnormalities, Multiple; Animals; Azacitidine; Chromosomal Proteins, Non-Histone; Craniofacial Abnormalities; DNA (Cytosine-5-)-Methyltransferases; DNA Methylation; DNA Methyltransferase 3A; DNA Methyltransferase 3B; DNA Mutational Analysis; DNA-Binding Proteins; Embryonic and Fetal Development; Female; Gene Expression Regulation, Developmental; Gene Silencing; Genes, Lethal; Genotype; Heterochromatin; Humans; Immunologic Deficiency Syndromes; Infant, Newborn; Male; Methyl-CpG-Binding Protein 2; Mice; Mutation, Missense; Phenotype; Repressor Proteins; Rett Syndrome; Sequence Deletion; Syndrome; X Chromosome

2000