azacitidine has been researched along with Abnormalities, Multiple in 3 studies
Timeframe | Studies, this research(%) | All Research% |
---|---|---|
pre-1990 | 0 (0.00) | 18.7374 |
1990's | 0 (0.00) | 18.2507 |
2000's | 3 (100.00) | 29.6817 |
2010's | 0 (0.00) | 24.3611 |
2020's | 0 (0.00) | 2.80 |
Authors | Studies |
---|---|
Hatta, T; Matsumoto, A; Moriyama, K; Otani, H | 1 |
Delmas, AL; Fields, CR; Jin, B; Liu, X; Peng, J; Qiu, J; Robertson, KD; Soo, HM; Tao, Q; Wu, W; Ying, J | 1 |
Kumar, A | 1 |
3 other study(ies) available for azacitidine and Abnormalities, Multiple
Article | Year |
---|---|
Opposite effects of the maternal immune system activated by interleukin-1beta vs. PSK and OK432 on 5-azacytidine-induced birth defects.
Topics: Abnormalities, Multiple; Adjuvants, Immunologic; Animals; Antimetabolites, Antineoplastic; Antineoplastic Agents; Azacitidine; Body Weight; Brain; Congenital Abnormalities; Female; Interleukin-1; Mice; Mice, Inbred ICR; Microscopy, Electron, Scanning; Phagocytes; Picibanil; Placenta; Pregnancy; Pregnancy, Animal; Proteoglycans; Teratogens; Time Factors | 2003 |
DNA methyltransferase 3B (DNMT3B) mutations in ICF syndrome lead to altered epigenetic modifications and aberrant expression of genes regulating development, neurogenesis and immune function.
Topics: Abnormalities, Multiple; Acetylation; Azacitidine; B-Lymphocytes; Case-Control Studies; Cell Line, Transformed; Cell Transformation, Viral; Cells, Cultured; Decitabine; DNA (Cytosine-5-)-Methyltransferases; DNA Methylation; DNA Methyltransferase 3B; Enzyme Inhibitors; Epigenesis, Genetic; Female; Gene Expression Profiling; Gene Expression Regulation, Developmental; Genes, Recessive; Histones; Humans; Hydroxamic Acids; Immunologic Deficiency Syndromes; Intellectual Disability; Male; Mutation; Neurons; Oligonucleotide Array Sequence Analysis; Promoter Regions, Genetic; Time Factors | 2008 |
Rett and ICF syndromes: methylation moves into medicine.
Topics: Abnormalities, Multiple; Animals; Azacitidine; Chromosomal Proteins, Non-Histone; Craniofacial Abnormalities; DNA (Cytosine-5-)-Methyltransferases; DNA Methylation; DNA Methyltransferase 3A; DNA Methyltransferase 3B; DNA Mutational Analysis; DNA-Binding Proteins; Embryonic and Fetal Development; Female; Gene Expression Regulation, Developmental; Gene Silencing; Genes, Lethal; Genotype; Heterochromatin; Humans; Immunologic Deficiency Syndromes; Infant, Newborn; Male; Methyl-CpG-Binding Protein 2; Mice; Mutation, Missense; Phenotype; Repressor Proteins; Rett Syndrome; Sequence Deletion; Syndrome; X Chromosome | 2000 |