atrazine has been researched along with Congenital Hypothyroidism in 1 studies
Congenital Hypothyroidism: A condition in infancy or early childhood due to an in-utero deficiency of THYROID HORMONES that can be caused by genetic or environmental factors, such as thyroid dysgenesis or HYPOTHYROIDISM in infants of mothers treated with THIOURACIL during pregnancy. Endemic cretinism is the result of iodine deficiency. Clinical symptoms include severe MENTAL RETARDATION, impaired skeletal development, short stature, and MYXEDEMA.
| Excerpt | Relevance | Reference |
|---|---|---|
| "2 mg/kg/2x/day to induce hypoprolactinemia or to propylthiouracil (PTU) at 2 mg/kg/day to induce hypothyroidism." | 3.72 | Developmental atrazine exposure suppresses immune function in male, but not female Sprague-Dawley rats. ( Luebke, RW; Matulka, RA; Rooney, AA, 2003) |
| Timeframe | Studies, this research(%) | All Research% |
|---|---|---|
| pre-1990 | 0 (0.00) | 18.7374 |
| 1990's | 0 (0.00) | 18.2507 |
| 2000's | 1 (100.00) | 29.6817 |
| 2010's | 0 (0.00) | 24.3611 |
| 2020's | 0 (0.00) | 2.80 |
| Authors | Studies |
|---|---|
| Rooney, AA | 1 |
| Matulka, RA | 1 |
| Luebke, RW | 1 |
1 other study available for atrazine and Congenital Hypothyroidism
| Article | Year |
|---|---|
Developmental atrazine exposure suppresses immune function in male, but not female Sprague-Dawley rats.
Topics: Abnormalities, Drug-Induced; Adjuvants, Immunologic; Administration, Oral; Animals; Animals, Sucklin | 2003 |