atorvastatin has been researched along with Carcinoma, Lewis Lung in 2 studies
Timeframe | Studies, this research(%) | All Research% |
---|---|---|
pre-1990 | 0 (0.00) | 18.7374 |
1990's | 0 (0.00) | 18.2507 |
2000's | 1 (50.00) | 29.6817 |
2010's | 1 (50.00) | 24.3611 |
2020's | 0 (0.00) | 2.80 |
Authors | Studies |
---|---|
Batista, ML; Bedard, AH; Birbrair, A; Bueno, LL; Correa, VA; Farmer, SR; Franco, FO; Guilherme, A; Henriques, F; Knobl, P; Lopes, MA; Peres, SB; Santos, KB | 1 |
Cooke, JP; Glassford, AJ; Heeschen, C; Weis, M | 1 |
2 other study(ies) available for atorvastatin and Carcinoma, Lewis Lung
Article | Year |
---|---|
Toll-Like Receptor-4 Disruption Suppresses Adipose Tissue Remodeling and Increases Survival in Cancer Cachexia Syndrome.
Topics: 3T3-L1 Cells; Adipose Tissue; Adiposity; Animals; Atorvastatin; Cachexia; Carcinoma, Lewis Lung; Disease Models, Animal; Gene Deletion; Lipid Metabolism; Male; Mice; Mice, Inbred C57BL; Mice, Knockout; Neoplasms; Survival Analysis; Syndrome; Toll-Like Receptor 4; Tumor Cells, Cultured | 2018 |
Statins have biphasic effects on angiogenesis.
Topics: Animals; Apolipoproteins E; Apoptosis; Atorvastatin; Carcinoma, Lewis Lung; Cell Differentiation; Cell Division; Cell Movement; Cells, Cultured; Dose-Response Relationship, Drug; Endothelial Growth Factors; Endothelium, Vascular; Female; Heptanoic Acids; Humans; Hydroxymethylglutaryl-CoA Reductase Inhibitors; Hyperlipidemias; Lymphokines; Mice; Mice, Inbred C57BL; Mice, Knockout; Neovascularization, Pathologic; Neovascularization, Physiologic; Polyisoprenyl Phosphates; Pyridines; Pyrroles; Receptor Protein-Tyrosine Kinases; Receptors, Growth Factor; Receptors, Vascular Endothelial Growth Factor; Signal Transduction; Vascular Endothelial Growth Factor A; Vascular Endothelial Growth Factors | 2002 |