Page last updated: 2024-10-23

atenolol and Triple Negative Breast Neoplasms

atenolol has been researched along with Triple Negative Breast Neoplasms in 1 studies

Atenolol: A cardioselective beta-1 adrenergic blocker possessing properties and potency similar to PROPRANOLOL, but without a negative inotropic effect.
atenolol : An ethanolamine compound having a (4-carbamoylmethylphenoxy)methyl group at the 1-position and an N-isopropyl substituent.

Triple Negative Breast Neoplasms: Breast neoplasms that do not express ESTROGEN RECEPTORS; PROGESTERONE RECEPTORS; and do not overexpress the NEU RECEPTOR/HER-2 PROTO-ONCOGENE PROTEIN.

Research Excerpts

ExcerptRelevanceReference
"Metformin can induce breast cancer (BC) cell apoptosis and reduce BC local and metastatic growth in preclinical models."7.83Aspirin and atenolol enhance metformin activity against breast cancer by targeting both neoplastic and microenvironment cells. ( Albini, A; Bertolini, F; Calleri, A; Dallaglio, K; Gregato, G; Labanca, V; Mancuso, P; Noonan, DM; Orecchioni, S; Reggiani, F; Rossi, T; Talarico, G, 2016)
"Metformin can induce breast cancer (BC) cell apoptosis and reduce BC local and metastatic growth in preclinical models."3.83Aspirin and atenolol enhance metformin activity against breast cancer by targeting both neoplastic and microenvironment cells. ( Albini, A; Bertolini, F; Calleri, A; Dallaglio, K; Gregato, G; Labanca, V; Mancuso, P; Noonan, DM; Orecchioni, S; Reggiani, F; Rossi, T; Talarico, G, 2016)

Research

Studies (1)

TimeframeStudies, this research(%)All Research%
pre-19900 (0.00)18.7374
1990's0 (0.00)18.2507
2000's0 (0.00)29.6817
2010's1 (100.00)24.3611
2020's0 (0.00)2.80

Authors

AuthorsStudies
Talarico, G1
Orecchioni, S1
Dallaglio, K1
Reggiani, F1
Mancuso, P1
Calleri, A1
Gregato, G1
Labanca, V1
Rossi, T1
Noonan, DM1
Albini, A1
Bertolini, F1

Other Studies

1 other study available for atenolol and Triple Negative Breast Neoplasms

ArticleYear
Aspirin and atenolol enhance metformin activity against breast cancer by targeting both neoplastic and microenvironment cells.
    Scientific reports, 2016, Jan-05, Volume: 6

    Topics: Adipose Tissue, White; AMP-Activated Protein Kinases; Animals; Antineoplastic Agents; Apoptosis; Asp

2016