astragaloside a has been researched along with Cardiac Remodeling, Ventricular in 3 studies
| Timeframe | Studies, this research(%) | All Research% |
|---|---|---|
| pre-1990 | 0 (0.00) | 18.7374 |
| 1990's | 0 (0.00) | 18.2507 |
| 2000's | 0 (0.00) | 29.6817 |
| 2010's | 3 (100.00) | 24.3611 |
| 2020's | 0 (0.00) | 2.80 |
| Authors | Studies |
|---|---|
| Tan, HY; Tang, B; Wei, XQ; Zhang, JG | 1 |
| Cao, Y; Chen, J; Chen, X; Hu, X; Li, X; Liu, H; Lu, S; Nie, J; Wu, X; Yang, Z; Zhao, X; Zhu, J | 1 |
| Bi, S; Han, D; Li, Y; Wang, C; Yang, X; Zhang, D; Zhang, Y | 1 |
3 other study(ies) available for astragaloside a and Cardiac Remodeling, Ventricular
| Article | Year |
|---|---|
Astragaloside IV inhibits ventricular remodeling and improves fatty acid utilization in rats with chronic heart failure.
Topics: Animals; Benzazepines; Chronic Disease; Chymases; Disease Models, Animal; Energy Metabolism; Fatty Acids; Heart Failure; Heart Ventricles; Humans; Myocardium; Myocytes, Cardiac; PPAR alpha; Rats; RNA, Messenger; Saponins; Triterpenes; Ventricular Remodeling | 2018 |
Genetic and pharmacological inhibition of Rheb1-mTORC1 signaling exerts cardioprotection against adverse cardiac remodeling in mice.
Topics: Animals; Apoptosis; Cardiomegaly; Cardiotonic Agents; Cells, Cultured; Drug Evaluation, Preclinical; Gene Deletion; Mechanistic Target of Rapamycin Complex 1; Mice; Mice, Inbred C57BL; Monomeric GTP-Binding Proteins; Multiprotein Complexes; Myocardial Infarction; Myocytes, Cardiac; Neuropeptides; Ras Homolog Enriched in Brain Protein; Saponins; Signal Transduction; TOR Serine-Threonine Kinases; Triterpenes; Ventricular Remodeling | 2013 |
Tetramethylpyrazine and Astragaloside IV Synergistically Ameliorate Left Ventricular Remodeling and Preserve Cardiac Function in a Rat Myocardial Infarction Model.
Topics: Animals; Drug Synergism; Drug Therapy, Combination; Drugs, Chinese Herbal; Myocardial Infarction; Pyrazines; Random Allocation; Rats; Rats, Sprague-Dawley; Saponins; Triterpenes; Vasodilator Agents; Ventricular Function, Left; Ventricular Remodeling | 2017 |