aspartic acid has been researched along with Diseases in Twins in 4 studies
Timeframe | Studies, this research(%) | All Research% |
---|---|---|
pre-1990 | 0 (0.00) | 18.7374 |
1990's | 1 (25.00) | 18.2507 |
2000's | 2 (50.00) | 29.6817 |
2010's | 1 (25.00) | 24.3611 |
2020's | 0 (0.00) | 2.80 |
Authors | Studies |
---|---|
Cleveland, SC; Frazier, TW; Gu, M; Hallmayer, JF; Hardan, AY; Hegarty, JP; Lazzeroni, LC; Phillips, JM; Raman, MM; Reiss, AL; Spielman, DM | 1 |
Imamura, A; Ito, R; Miyajima, H; Orii, KO | 1 |
Aro, A; Hu, H; Mulkern, RV; Oliveira, S; Weisskopf, MG; White, R; Wright, RO | 1 |
Kure, S; Matsubara, Y; Narisawa, K; Osamu, N; Sakata, Y; Shinka, T; Tada, K; Takayanagi, M | 1 |
4 other study(ies) available for aspartic acid and Diseases in Twins
Article | Year |
---|---|
A proton MR spectroscopy study of the thalamus in twins with autism spectrum disorder.
Topics: Adolescent; Aspartic Acid; Autism Spectrum Disorder; Child; Cohort Studies; Diseases in Twins; Female; Functional Laterality; Humans; Magnetic Resonance Imaging; Male; Neuropsychological Tests; Proton Magnetic Resonance Spectroscopy; Severity of Illness Index; Thalamus; Twins, Dizygotic; Twins, Monozygotic | 2018 |
Serial MR imaging and 1H-MR spectroscopy in monozygotic twins with Tay-Sachs disease.
Topics: Aspartic Acid; Basal Ganglia; Brain; Cerebral Cortex; Child, Preschool; Choline; Corpus Callosum; Creatine; Disease Progression; Diseases in Twins; DNA Mutational Analysis; Dominance, Cerebral; Energy Metabolism; Epilepsies, Myoclonic; Female; Gangliosidoses, GM2; Hexosaminidase A; Humans; Image Processing, Computer-Assisted; Inositol; Magnetic Resonance Imaging; Magnetic Resonance Spectroscopy; Nerve Fibers, Myelinated; Neurologic Examination; Thalamus; Twins, Monozygotic | 2008 |
Cognitive deficits and magnetic resonance spectroscopy in adult monozygotic twins with lead poisoning.
Topics: Aged; Aspartic Acid; Bone and Bones; Brain; Cell Count; Cognition Disorders; Creatine; Diseases in Twins; Humans; Lead; Lead Poisoning; Lead Poisoning, Nervous System, Adult; Magnetic Resonance Spectroscopy; Male; Neurons; Occupational Diseases; Twins, Monozygotic | 2004 |
A one-base deletion (183delC) and a missense mutation (D276H) in the T-protein gene from a Japanese family with nonketotic hyperglycinemia.
Topics: Amino Acid Metabolism, Inborn Errors; Aminomethyltransferase; Animals; Asian People; Aspartic Acid; Cattle; Codon; Diseases in Twins; DNA Mutational Analysis; Female; Germ-Line Mutation; Glycine; Humans; Hydroxymethyl and Formyl Transferases; Infant, Newborn; Japan; Liver; Male; Pedigree; Point Mutation; Sequence Deletion; Species Specificity; Structure-Activity Relationship; Twins, Monozygotic | 1998 |