aspartic acid has been researched along with Autosomal Dominant Optic Atrophy in 2 studies
| Timeframe | Studies, this research(%) | All Research% |
|---|---|---|
| pre-1990 | 0 (0.00) | 18.7374 |
| 1990's | 0 (0.00) | 18.2507 |
| 2000's | 0 (0.00) | 29.6817 |
| 2010's | 2 (100.00) | 24.3611 |
| 2020's | 0 (0.00) | 2.80 |
| Authors | Studies |
|---|---|
| Arnauné-Pelloquin, L; Arrázola, MS; Belenguer, P; Bocca, C; Chao de la Barca, JM; Iuliano, O; Lenaers, G; Reynier, P; Simard, G; Tcherkez, G | 1 |
| Angebault, C; Bielicki, G; Boddaert, N; Brabet, P; Cazevieille, C; Chaix, B; Delettre, C; Gueguen, N; Hamel, CP; Lenaers, G; Mausset-Bonnefont, AL; Puel, JL; Renou, JP; Reynier, P; Rigau, V; Sarzi, E; Seveno, M; Wang, J | 1 |
2 other study(ies) available for aspartic acid and Autosomal Dominant Optic Atrophy
| Article | Year |
|---|---|
The Metabolomic Signature of Opa1 Deficiency in Rat Primary Cortical Neurons Shows Aspartate/Glutamate Depletion and Phospholipids Remodeling.
Topics: Animals; Aspartic Acid; Cells, Cultured; Cerebral Cortex; Disease Models, Animal; Down-Regulation; Embryo, Mammalian; Female; Glutamic Acid; GTP Phosphohydrolases; Humans; Metabolomics; Neurons; Optic Atrophy, Autosomal Dominant; Phospholipids; Primary Cell Culture; Rats; RNA, Small Interfering | 2019 |
The human OPA1delTTAG mutation induces premature age-related systemic neurodegeneration in mouse.
Topics: Acoustic Stimulation; Age Factors; Aging, Premature; Animals; Aspartic Acid; Chi-Square Distribution; Creatine; Disease Models, Animal; Disease Progression; Electron Transport Chain Complex Proteins; Electron Transport Complex IV; Electroretinography; Evoked Potentials, Auditory, Brain Stem; Evoked Potentials, Visual; Gene Expression Regulation; Glycolysis; GTP Phosphohydrolases; Humans; Lactic Acid; Locomotion; Magnetic Resonance Imaging; Magnetic Resonance Spectroscopy; Membrane Proteins; Mice; Mice, Inbred C57BL; Mice, Transgenic; Mitochondrial Diseases; Muscle, Skeletal; Nervous System; Optic Atrophy, Autosomal Dominant; Optic Nerve; Phenotype; Physical Conditioning, Animal; Psychoacoustics; Psychomotor Performance; Reaction Time; Retina; Retinal Ganglion Cells; Sequence Deletion | 2012 |