asparagine and Growth Disorders

asparagine has been researched along with Growth Disorders in 2 studies

Research

Studies (2)

Timeframe	Studies, this research(%)	All Research%
pre-1990	0 (0.00)	18.7374
1990's	2 (100.00)	18.2507
2000's	0 (0.00)	29.6817
2010's	0 (0.00)	24.3611
2020's	0 (0.00)	2.80

Authors

Authors	Studies
Besmond, C; Blivet, MJ; Capeau, J; Caron, M; Desbois-Mouthon, C; Flori, E; Magre, J; Oreal, E; Sert-Langeron, C	1
Clarke, S; Kim, E; Lowenson, JD; MacLaren, DC; Young, SG	1

Other Studies

2 other study(ies) available for asparagine and Growth Disorders

Article	Year
Deletion of Asn281 in the alpha-subunit of the human insulin receptor causes constitutive activation of the receptor and insulin desensitization. The Journal of clinical endocrinology and metabolism, 1996, Volume: 81, Issue:2 Topics: Amino Acid Sequence; Asparagine; Base Sequence; DNA; Electrophoresis; Female; Fibroblasts; Gene Deletion; Glycogen; Growth Disorders; Humans; Infant, Newborn; Insulin; Molecular Sequence Data; Phosphorylation; Polymerase Chain Reaction; Receptor, Insulin; RNA, Messenger; Sequence Analysis, DNA	1996
Deficiency of a protein-repair enzyme results in the accumulation of altered proteins, retardation of growth, and fatal seizures in mice. Proceedings of the National Academy of Sciences of the United States of America, 1997, Jun-10, Volume: 94, Issue:12 Topics: Animals; Asparagine; Aspartic Acid; Body Weight; Death, Sudden; DNA Primers; Female; Growth Disorders; Heterozygote; Humans; Infant, Newborn; Male; Mice; Mice, Knockout; Pentylenetetrazole; Polymerase Chain Reaction; Protein D-Aspartate-L-Isoaspartate Methyltransferase; Protein Methyltransferases; Proteins; Recombinant Proteins; Restriction Mapping; Seizures	1997

Article

Year

Deletion of Asn281 in the alpha-subunit of the human insulin receptor causes constitutive activation of the receptor and insulin desensitization.

The Journal of clinical endocrinology and metabolism, 1996, Volume: 81, Issue:2

Topics: Amino Acid Sequence; Asparagine; Base Sequence; DNA; Electrophoresis; Female; Fibroblasts; Gene Deletion; Glycogen; Growth Disorders; Humans; Infant, Newborn; Insulin; Molecular Sequence Data; Phosphorylation; Polymerase Chain Reaction; Receptor, Insulin; RNA, Messenger; Sequence Analysis, DNA

1996

Deficiency of a protein-repair enzyme results in the accumulation of altered proteins, retardation of growth, and fatal seizures in mice.

Proceedings of the National Academy of Sciences of the United States of America, 1997, Jun-10, Volume: 94, Issue:12

Topics: Animals; Asparagine; Aspartic Acid; Body Weight; Death, Sudden; DNA Primers; Female; Growth Disorders; Heterozygote; Humans; Infant, Newborn; Male; Mice; Mice, Knockout; Pentylenetetrazole; Polymerase Chain Reaction; Protein D-Aspartate-L-Isoaspartate Methyltransferase; Protein Methyltransferases; Proteins; Recombinant Proteins; Restriction Mapping; Seizures

1997