asparagine has been researched along with Cardiovascular Diseases in 3 studies
| Timeframe | Studies, this research(%) | All Research% |
|---|---|---|
| pre-1990 | 1 (33.33) | 18.7374 |
| 1990's | 0 (0.00) | 18.2507 |
| 2000's | 2 (66.67) | 29.6817 |
| 2010's | 0 (0.00) | 24.3611 |
| 2020's | 0 (0.00) | 2.80 |
| Authors | Studies |
|---|---|
| Langebrake, C; Reinhardt, D; Ritter, J | 1 |
| Bardin, S; Baudrie, V; Bernstein, KE; Billet, S; Clauser, E; Conchon, S; Corvol, P; Elghozi, JL; Escoubet, B; Gasc, JM; Hamard, G; Michaud, A; Muffat-Joly, M; Souil, E; Verp, S | 1 |
| Moiseev, VS | 1 |
2 review(s) available for asparagine and Cardiovascular Diseases
| Article | Year |
|---|---|
Minimising the long-term adverse effects of childhood leukaemia therapy.
Topics: Alkylating Agents; Anthracyclines; Antimetabolites; Antineoplastic Agents; Asparaginase; Asparagine; Cardiovascular Diseases; Child; Clinical Trials as Topic; Glucocorticoids; Gonadal Disorders; Humans; Leukemia; Leukemia, Myeloid, Acute; Osteonecrosis; Podophyllotoxin; Polyethylene Glycols; Precursor Cell Lymphoblastic Leukemia-Lymphoma; Tretinoin; Vinca Alkaloids | 2002 |
[Pathologic changes in the cardiovascular system during treatment with glucocorticoid hormones].
Topics: Animals; Arteriosclerosis; Arthritis, Rheumatoid; Asparagine; Biological Transport, Active; Calcium; Cardiovascular Diseases; Cats; Child; Child, Preschool; Coronary Disease; Cortisone; Dipeptides; Dogs; Enzyme Induction; Glucocorticoids; Heart; Heart Failure; Humans; Hyperlipidemias; Hypertension; Infant; Lupus Erythematosus, Systemic; Magnesium; Myocarditis; Myocardium; Nephrotic Syndrome; Potassium; Prednisolone; Rabbits; Rats; Skin Diseases; Sodium; Time Factors; Triamcinolone | 1972 |
1 other study(ies) available for asparagine and Cardiovascular Diseases
| Article | Year |
|---|---|
Gain-of-function mutant of angiotensin II receptor, type 1A, causes hypertension and cardiovascular fibrosis in mice.
Topics: Angiotensins; Animals; Asparagine; Blood Pressure; Cardiovascular Diseases; Disease Progression; Female; Fibrosis; Gene Expression Regulation; Hyperaldosteronism; Hypertension; Kidney; Male; Mice; Mice, Inbred C57BL; Mice, Transgenic; Mutation; Receptor, Angiotensin, Type 1; Renin; Signal Transduction | 2007 |