Compounds > asparagine

asparagine and Cardiomyopathy, Hypertrophic

asparagine has been researched along with Cardiomyopathy, Hypertrophic in 5 studies

Research

Studies (5)

TimeframeStudies, this research(%)All Research%
pre-19900 (0.00)18.7374
1990's2 (40.00)18.2507
2000's1 (20.00)29.6817
2010's2 (40.00)24.3611
2020's0 (0.00)2.80

Authors

AuthorsStudies
Borovikov, YS; Karpicheva, OE; Redwood, CS; Rysev, NA1
Fischer, S; Geeves, MA; Janco, M; Lehman, W; Li, XE; Marston, SB; Suphamungmee, W1
Hartikainen, J; Hedman, A; Jääskeläinen, P; Kuusisto, J; Laakso, M; Laitinen, T; Peuhkurinen, K; Vanninen, E1
Bing, W; Esposito, G; Marston, SB; Purcell, IF; Redwood, CS; Watkins, H1
Akanuma, M; Harada, H; Hiroi, S; Katsushika, S; Kimura, A; Nakamura, H; Ohsuzu, F; Satoh, M1

Other Studies

5 other study(ies) available for asparagine and Cardiomyopathy, Hypertrophic

ArticleYear
The effect of the Asp175Asn and Glu180Gly TPM1 mutations on actin-myosin interaction during the ATPase cycle.
    Biochimica et biophysica acta, 2012, Volume: 1824, Issue:2

    Topics: Actins; Actomyosin; Adenosine Triphosphatases; Amino Acid Substitution; Animals; Asparagine; Aspartic Acid; Cardiomyopathy, Hypertrophic; Fluorescent Dyes; Glutamic Acid; Glycine; Humans; Muscle Contraction; Mutation; Myosin Subfragments; Naphthalenesulfonates; Peptide Fragments; Protein Structure, Secondary; Rabbits; Spectrometry, Fluorescence; Tropomyosin

2012
The flexibility of two tropomyosin mutants, D175N and E180G, that cause hypertrophic cardiomyopathy.
    Biochemical and biophysical research communications, 2012, Aug-03, Volume: 424, Issue:3

    Topics: Actins; Amino Acid Substitution; Asparagine; Aspartic Acid; Calcium; Cardiomyopathy, Hypertrophic; Glutamic Acid; Glycine; Humans; Microscopy, Electron; Models, Chemical; Molecular Dynamics Simulation; Mutation; Myosins; Protein Structure, Secondary; Tropomyosin

2012
Inducibility of life-threatening ventricular arrhythmias is related to maximum left ventricular thickness and clinical markers of sudden cardiac death in patients with hypertrophic cardiomyopathy attributable to the Asp175Asn mutation in the alpha-tropomy
    Journal of molecular and cellular cardiology, 2004, Volume: 36, Issue:1

    Topics: Adult; Angiography; Arrhythmias, Cardiac; Asparagine; Aspartic Acid; Biomarkers; Cardiomyopathy, Hypertrophic; Death, Sudden, Cardiac; Echocardiography; Exercise Test; Female; Genetic Predisposition to Disease; Heart Ventricles; Humans; Male; Mutation, Missense; Pedigree; Tropomyosin

2004
Effects of two hypertrophic cardiomyopathy mutations in alpha-tropomyosin, Asp175Asn and Glu180Gly, on Ca2+ regulation of thin filament motility.
    Biochemical and biophysical research communications, 1997, Jul-30, Volume: 236, Issue:3

    Topics: Actins; Animals; Asparagine; Aspartic Acid; Cardiomyopathy, Hypertrophic; Glutamic Acid; Glycine; Muscle, Skeletal; Mutation; Myosin Subfragments; Phalloidine; Rabbits; Rhodamines; Tropomyosin; Troponin

1997
Hypertrophic obstructive cardiomyopathy due to a novel T-to-A transition at codon 624 in the beta-myosin heavy chain (beta-MHC) gene possibly related to the sudden death.
    International journal of cardiology, 1997, Dec-19, Volume: 62, Issue:3

    Topics: Amyl Nitrite; Asparagine; Aspirin; Atrial Fibrillation; Cardiomyopathy, Hypertrophic; Codon; Death, Sudden, Cardiac; Echocardiography, Doppler; Electrocardiography; Female; Heart Septum; Humans; Middle Aged; Mitral Valve; Myosin Heavy Chains; Point Mutation; Tyrosine

1997