artenimol has been researched along with Cancer of Skin in 3 studies
| Timeframe | Studies, this research(%) | All Research% |
|---|---|---|
| pre-1990 | 0 (0.00) | 18.7374 |
| 1990's | 0 (0.00) | 18.2507 |
| 2000's | 0 (0.00) | 29.6817 |
| 2010's | 1 (33.33) | 24.3611 |
| 2020's | 2 (66.67) | 2.80 |
| Authors | Studies |
|---|---|
| Fujimoto, M; Jin, G; Jin, L; Jin, Q; Li, F; Li, H; Lin, Z; Meng, F; Ren, X; Wei, Q; Yu, R | 1 |
| Li, Z; Liu, J; Teng, M; Wang, Y | 1 |
| Hui, HY; Liu, Y; Wu, M; Wu, N; Xiao, SX; Zhang, MF | 1 |
3 other study(ies) available for artenimol and Cancer of Skin
| Article | Year |
|---|---|
Dihydroartemisinin inhibits melanoma by regulating CTL/Treg anti-tumor immunity and STAT3-mediated apoptosis via IL-10 dependent manner.
Topics: Administration, Oral; Animals; Apoptosis; Artemisinins; Cell Line, Tumor; Drug Screening Assays, Antitumor; Female; Gene Expression Regulation, Neoplastic; Humans; Interleukin-10; Melanoma, Experimental; Mice; Mitochondria; Phosphorylation; Signal Transduction; Skin Neoplasms; STAT3 Transcription Factor; T-Lymphocytes, Cytotoxic; T-Lymphocytes, Regulatory; Tumor Escape | 2020 |
Dihydroartemisinin inhibits activation of the AIM2 inflammasome pathway and NF-κB/HIF-1α/VEGF pathway by inducing autophagy in A431 human cutaneous squamous cell carcinoma cells.
Topics: Apoptosis; Artemisinins; Autophagy; Carcinoma, Squamous Cell; Cell Line, Tumor; Cell Movement; Cell Proliferation; Drug Screening Assays, Antitumor; Humans; Hypoxia-Inducible Factor 1, alpha Subunit; Inflammasomes; NF-kappa B; Signal Transduction; Sirolimus; Skin Neoplasms; TOR Serine-Threonine Kinases; Vascular Endothelial Growth Factor A | 2021 |
Dihydroartemisinin suppresses growth of squamous cell carcinoma A431 cells by targeting the Wnt/β-catenin pathway.
Topics: Antineoplastic Agents; Apoptosis; Artemisinins; beta Catenin; Carcinoma, Squamous Cell; Caspase 3; Cell Line, Tumor; Cell Proliferation; Dose-Response Relationship, Drug; Humans; Inhibitor of Apoptosis Proteins; Signal Transduction; Skin Neoplasms; Survivin; Wnt Proteins | 2016 |